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Serum Amyloid A Is a Ligand for Scavenger Receptor Class B Type I and Inhibits High Density Lipoprotein Binding and Selective Lipid Uptake

The Journal of biological chemistry, 2005-01, Vol.280 (4), p.2954-2961

2005

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Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

Journal of lipid research, 2018-02, Vol.59 (2), p.339-347

2018

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Serum amyloid A binds to fibrin(ogen), promoting fibrin amyloid formation

Scientific reports, 2019-02, Vol.9 (1), p.3102-3102, Article 3102

2019

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Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

Journal of lipid research, 2020-03, Vol.61 (3), p.328-337

2020

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HDL remodeling during the acute phase response

Arteriosclerosis, thrombosis, and vascular biology, 2009-02, Vol.29 (2), p.261-267

2009

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Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

Journal of lipid research, 2016-06, Vol.57 (6), p.969-979

2016

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Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI

Journal of lipid research, 2012-03, Vol.53 (3), p.446-455

2012

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Serum Amyloid A Promotes Cholesterol Efflux Mediated by Scavenger Receptor B-I

The Journal of biological chemistry, 2005-10, Vol.280 (43), p.35890-35895

2005

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Impact of serum amyloid A on high density lipoprotein composition and levels

Journal of lipid research, 2010-11, Vol.51 (11), p.3117-3125

2010

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High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

The Journal of biological chemistry, 2018-08, Vol.293 (34), p.13257-13269

2018

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Serum amyloid A impairs the antiinflammatory properties of HDL

The Journal of clinical investigation, 2016-01, Vol.126 (1), p.266-281

2016

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Serum Amyloid A Is an Exchangeable Apolipoprotein

Arteriosclerosis, thrombosis, and vascular biology, 2018-08, Vol.38 (8), p.1890-1900

2018

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High-Density Lipoprotein Suppresses the Type I Interferon Response, a Family of Potent Antiviral Immunoregulators, in Macrophages Challenged With Lipopolysaccharide

Circulation (New York, N.Y.), 2010-11, Vol.122 (19), p.1919-1927

2010

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Deficiency of Endogenous Acute-Phase Serum Amyloid A Protects apoE-/- Mice From Angiotensin II-Induced Abdominal Aortic Aneurysm Formation

Arteriosclerosis, thrombosis, and vascular biology, 2015-05, Vol.35 (5), p.1156-1165

2015

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Low-density lipoprotein from apolipoprotein E-deficient mice induces macrophage lipid accumulation in a CD36 and scavenger receptor class A-dependent manner

Arteriosclerosis, thrombosis, and vascular biology, 2005-01, Vol.25 (1), p.168-173

2005

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Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice

Arteriosclerosis, thrombosis, and vascular biology, 2014-02, Vol.34 (2), p.255-261

2014

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Serum amyloid A impairs the antiinflammatory properties of HDL

The Journal of clinical investigation, 2016-02, Vol.126 (2), p.796-796

2016

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The Impairment of Macrophage-to-Feces Reverse Cholesterol Transport during Inflammation Does Not Depend on Serum Amyloid A

Journal of Lipids, 2013-01, Vol.2013 (2013), p.1-11

2013

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SAA does not induce cytokine production in physiological conditions

Cytokine (Philadelphia, Pa.), 2013-02, Vol.61 (2), p.506-512

2013

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Serum amyloid A facilitates the binding of high-density lipoprotein from mice injected with lipopolysaccharide to vascular proteoglycans

Arteriosclerosis, thrombosis, and vascular biology, 2011-06, Vol.31 (6), p.1326-1332

2011

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Macrosialin increases during normal brain aging are attenuated by caloric restriction

Neuroscience letters, 2005-12, Vol.390 (2), p.76-80

2005

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Lipoproteins are substrates for human secretory group IIA phospholipase A2: preferential hydrolysis of acute phase HDL

Journal of lipid research, 1998-11, Vol.39 (11), p.2150-2160

1998

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Remodeling of HDL remnants generated by scavenger receptor class B type I

Journal of lipid research, 2004-09, Vol.45 (9), p.1666-1673

2004

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Lack of a direct role for macrosialin in oxidized LDL metabolism

Journal of lipid research, 2003-04, Vol.44 (4), p.674-685

2003

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Scavenger receptor SR-BI in macrophage lipid metabolism

Atherosclerosis, 2011-07, Vol.217 (1), p.106-112

2011

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Serum Amyloid A Is a Ligand for Scavenger Receptor Class B Type I and Inhibits High Density Lipoprotein Binding and Selective Lipid Uptake

Serum amyloid A3 is a high density lipoprotein-associated acute-phase protein

Serum amyloid A binds to fibrin(ogen), promoting fibrin amyloid formation

Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

HDL remodeling during the acute phase response

Impact of individual acute phase serum amyloid A isoforms on HDL metabolism in mice[S]

Nascent HDL formation in hepatocytes and role of ABCA1, ABCG1, and SR-BI

Serum Amyloid A Promotes Cholesterol Efflux Mediated by Scavenger Receptor B-I

Impact of serum amyloid A on high density lipoprotein composition and levels

High-density lipoprotein inhibits serum amyloid A–mediated reactive oxygen species generation and NLRP3 inflammasome activation

Serum amyloid A impairs the antiinflammatory properties of HDL

Serum Amyloid A Is an Exchangeable Apolipoprotein

High-Density Lipoprotein Suppresses the Type I Interferon Response, a Family of Potent Antiviral Immunoregulators, in Macrophages Challenged With Lipopolysaccharide

Deficiency of Endogenous Acute-Phase Serum Amyloid A Protects apoE-/- Mice From Angiotensin II-Induced Abdominal Aortic Aneurysm Formation

Low-density lipoprotein from apolipoprotein E-deficient mice induces macrophage lipid accumulation in a CD36 and scavenger receptor class A-dependent manner

Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice

Serum amyloid A impairs the antiinflammatory properties of HDL

The Impairment of Macrophage-to-Feces Reverse Cholesterol Transport during Inflammation Does Not Depend on Serum Amyloid A

SAA does not induce cytokine production in physiological conditions

Serum amyloid A facilitates the binding of high-density lipoprotein from mice injected with lipopolysaccharide to vascular proteoglycans

Macrosialin increases during normal brain aging are attenuated by caloric restriction

Lipoproteins are substrates for human secretory group IIA phospholipase A2: preferential hydrolysis of acute phase HDL

Remodeling of HDL remnants generated by scavenger receptor class B type I

Lack of a direct role for macrosialin in oxidized LDL metabolism

Scavenger receptor SR-BI in macrophage lipid metabolism

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