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A call to action to address rural mental health disparities

Journal of clinical and translational science, 2020-10, Vol.4 (5), p.463-467

2020

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Continuing trial responsibilities for implantable neural devices

Neuron (Cambridge, Mass.), 2023-10, Vol.111 (20), p.3143-3149

2023

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Advancing scientific excellence through inclusivity in the NIH BRAIN Initiative

Neuron (Cambridge, Mass.), 2021-11, Vol.109 (21), p.3361-3364

2021

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Epigenetics in the Nervous System

The Journal of neuroscience, 2008-11, Vol.28 (46), p.11753-11759

2008

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Alaska Native genomic research: perspectives from Alaska Native leaders, federal staff, and biomedical researchers

Genetics in medicine, 2020-12, Vol.22 (12), p.1935-1943

2020

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Poly(A) Tail Length-dependent Stabilization of GAP-43 mRNA by the RNA-binding Protein HuD

The Journal of biological chemistry, 2002-08, Vol.277 (31), p.27996-28002

2002

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Accelerating a paradigm shift: The Common Fund Single Cell Analysis Program

Science advances, 2018-08, Vol.4 (8), p.eaat8573-eaat8573

2018

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Corticosterone response to acute stress in a mouse model of Fragile X syndrome

Psychoneuroendocrinology, 2006-07, Vol.31 (6), p.781-785

2006

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The RNA-binding protein HuD is required for GAP-43 mRNA stability, GAP-43 gene expression, and PKC-dependent neurite outgrowth in PC12 cells

Molecular biology of the cell, 2000-09, Vol.11 (9), p.3191-3203

2000

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Autism and abnormal development of brain connectivity

The Journal of neuroscience, 2004-10, Vol.24 (42), p.9228-9231

2004

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Overexpression of HuD, but Not of Its Truncated Form HuD I+II, Promotes GAP‐43 Gene Expression and Neurite Outgrowth in PC12 Cells in the Absence of Nerve Growth Factor

Journal of neurochemistry, 2000-09, Vol.75 (3), p.1103-1114

2000

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RNA Cargoes Associating with FMRP Reveal Deficits in Cellular Functioning in Fmr1 Null Mice

Neuron (Cambridge, Mass.), 2003-02, Vol.37 (3), p.417-431

2003

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Epigenetics in the Nervous System: Figure 1

The Journal of neuroscience, 2008-11, Vol.28 (46), p.11753-11759

2008

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Autism and Abnormal Development of Brain Connectivity: Figure 1

The Journal of neuroscience, 2004-10, Vol.24 (42), p.9228-9231

2004

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Outcome measures for clinical trials in fragile X syndrome

Journal of developmental and behavioral pediatrics, 2013-09, Vol.34 (7), p.508-522

2013

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The State of the NIH BRAIN Initiative

The Journal of neuroscience, 2018-07, Vol.38 (29), p.6427-6438

2018

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Effects of Fragile X syndrome and an FMR1 knockout mouse model on forebrain neuronal cell biology

Microscopy research and technique, 2002-05, Vol.57 (3), p.156-158

2002

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Correlates across the structural, functional, and molecular phenotypes of fragile X syndrome

Mental retardation and developmental disabilities research reviews, 2004-02, Vol.10 (1), p.53-59

2004

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Community resources and technologies developed through the NIH Roadmap Epigenomics Program

Methods in molecular biology (Clifton, N.J.), 2015, Vol.1238, p.27

2015

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Erratum to “Neuroepigenomics: resources, obstacles, and opportunities” [Neuroepigenetics 1 (2015) 2-13]

Neuroepigenetics, 2015-07, Vol.3, p.7-7

2015

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Neuroepigenomics: resources, obstacles, and opportunities

Neuroepigenetics, 2015-01, Vol.1, p.2-13

2015

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A novel transcriptional element in circular DNA monomers of the duck hepatitis B virus

Journal of Virology, 1997-10, Vol.71 (10), p.7917-7922

1997

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The RNA binding protein HuD stabilizes GAP-43 mRNA by controlling the rate of deadenylation

Beckel-Mitchener, Andrea Celine

1999

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Correlates across the structural, functional, and molecular phenotypes of fragile X syndrome: Fragile X Syndrome: frontiers of understanding gene-brain-behavior relationships

Mental retardation and developmental disabilities research reviews, 2004, Vol.10 (1), p.53-59

2004

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A call to action to address rural mental health disparities

Continuing trial responsibilities for implantable neural devices

Advancing scientific excellence through inclusivity in the NIH BRAIN Initiative

Epigenetics in the Nervous System

Alaska Native genomic research: perspectives from Alaska Native leaders, federal staff, and biomedical researchers

Poly(A) Tail Length-dependent Stabilization of GAP-43 mRNA by the RNA-binding Protein HuD

Accelerating a paradigm shift: The Common Fund Single Cell Analysis Program

Corticosterone response to acute stress in a mouse model of Fragile X syndrome

The RNA-binding protein HuD is required for GAP-43 mRNA stability, GAP-43 gene expression, and PKC-dependent neurite outgrowth in PC12 cells

Autism and abnormal development of brain connectivity

Overexpression of HuD, but Not of Its Truncated Form HuD I+II, Promotes GAP‐43 Gene Expression and Neurite Outgrowth in PC12 Cells in the Absence of Nerve Growth Factor

RNA Cargoes Associating with FMRP Reveal Deficits in Cellular Functioning in Fmr1 Null Mice

Epigenetics in the Nervous System: Figure 1

Autism and Abnormal Development of Brain Connectivity: Figure 1

Outcome measures for clinical trials in fragile X syndrome

The State of the NIH BRAIN Initiative

Effects of Fragile X syndrome and an FMR1 knockout mouse model on forebrain neuronal cell biology

Correlates across the structural, functional, and molecular phenotypes of fragile X syndrome

Community resources and technologies developed through the NIH Roadmap Epigenomics Program

Erratum to “Neuroepigenomics: resources, obstacles, and opportunities” [Neuroepigenetics 1 (2015) 2-13]

Neuroepigenomics: resources, obstacles, and opportunities

A novel transcriptional element in circular DNA monomers of the duck hepatitis B virus

The RNA binding protein HuD stabilizes GAP-43 mRNA by controlling the rate of deadenylation

Correlates across the structural, functional, and molecular phenotypes of fragile X syndrome: Fragile X Syndrome: frontiers of understanding gene-brain-behavior relationships

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