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Beclin 1 and autophagy are required for the tumorigenicity of breast cancer stem-like/progenitor cells

Oncogene, 2013-05, Vol.32 (18), p.2261-2272

2013

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Ceramide-mediated Macroautophagy Involves Inhibition of Protein Kinase B and Up-regulation of Beclin 1

The Journal of biological chemistry, 2004-04, Vol.279 (18), p.18384-18391

2004

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Amino Acids Interfere with the ERK1/2-dependent Control of Macroautophagy by Controlling the Activation of Raf-1 in Human Colon Cancer HT-29 Cells

The Journal of biological chemistry, 2003-05, Vol.278 (19), p.16667-16674

2003

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The Tumor Suppressor PTEN Positively Regulates Macroautophagy by Inhibiting the Phosphatidylinositol 3-Kinase/Protein Kinase B Pathway

The Journal of biological chemistry, 2001-09, Vol.276 (38), p.35243-35246

2001

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Endoplasmic reticulum‐to‐cytosol transport of free polymannose oligosaccharides in permeabilized HepG2 cells

The EMBO journal, 1995-12, Vol.14 (23), p.6034-6042

1995

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Celecoxib Induces Apoptosis by Inhibiting 3-Phosphoinositide-dependent Protein Kinase-1 Activity in the Human Colon Cancer HT-29 Cell Line

The Journal of biological chemistry, 2002-08, Vol.277 (31), p.27613-27621

2002

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BAG6/BAT3 modulates autophagy by affecting EP300/p300 intracellular localization

Autophagy, 2014-07, Vol.10 (7), p.1341-1342

2014

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A Heterotrimeric G-protein Controls Autophagic Sequestration in the Human Colon Cancer Cell Line HT-29

The Journal of biological chemistry, 1995-01, Vol.270 (1), p.13

1995

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Autophagy Delays Sulindac Sulfide-Induced Apoptosis in the Human Intestinal Colon Cancer Cell Line HT-29

Experimental cell research, 2001-08, Vol.268 (2), p.139-149

2001

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Human glucocerebrosidase: heterologous expression of active site mutants in murine null cells

Glycobiology (Oxford), 2000-11, Vol.10 (11), p.1217-1224

2000

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Differentiation-dependent autophagy controls the fate of newly synthesized N-linked glycoproteins in the colon adenocarcinoma HT-29 cell line

Biochemical journal, 1995-07, Vol.309 ( Pt 2) (2), p.521-527

1995

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The G-protein Regulator AGS3 Controls an Early Event during Macroautophagy in Human Intestinal HT-29 Cells

The Journal of biological chemistry, 2003-06, Vol.278 (23), p.20995-21002

2003

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Subcellular localization of the Galphai3 protein and G alpha interacting protein, two proteins involved in the control of macroautophagy in human colon cancer HT-29 cells

Biochemical journal, 1999-01, Vol.337 ( Pt 2) (Pt 2), p.289-295

1999

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The N-glycan processing in HT-29 cells is a function of their state of enterocytic differentiation. Evidence for an atypical traffic associated with change in polypeptide stability in undifferentiated HT-29 cells

The Journal of biological chemistry, 1991-11, Vol.266 (31), p.20849-20855

1991

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Régulation post-traductionnelle de l'expression des protéines N-glycosylées dans des cellules intestinales humaines en culture

Comptes rendus des séances de la Société de biologie et de ses filiales, 1991, Vol.185 (4), p.182-189

1991

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Localization and Processing of Glycosylphosphatidylinositol-Anchored Cathepsin D

Biochemical and biophysical research communications, 1995-06, Vol.211 (3), p.935-942

1995

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Transfer of Free Polymannose-Type Oligosaccharides from the Cytosol to Lysosomes in Cultured Human Hepatocellular Carcinoma HEPG2 Cells

The Journal of cell biology, 1997-01, Vol.136 (1), p.45-59

1997

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Isoforms of the Lutheran/basal cell adhesion molecule glycoprotein are differentially delivered in polarized epithelial cells. Mapping of the basolateral sorting signal to a cytoplasmic di-leucine motif

The Journal of biological chemistry, 1999-11, Vol.274 (45), p.31903-31908

1999

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Requirement of either the NH4Cl-sensitive or the cytochalasin D-sensitive pathway for ricin toxicity depends upon the enterocytic state of differentiation of HT-29 cells

European journal of cell biology, 1994-06, Vol.64 (1), p.15-28

1994

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hnRNP G: sequence and characterization of a glycosylated RNA-binding protein

Nucleic acids research, 1993-09, Vol.21 (18), p.4210-4217

1993

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A mucus-secreting human colonic cancer cell line. Purification and partial characterization of the secreted mucins

Biochemical journal, 1989-03, Vol.258 (3), p.793-799

1989

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Processing of asparagine-linked oligosaccharides is an early biochemical marker of the enterocytic differentiation of HT-29 cells

Journal of cellular biochemistry, 1989-09, Vol.41 (1), p.13-23

1989

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Glucose persistence on high-mannose oligosaccharides selectively inhibits the macroautophagic sequestration of N-linked glycoproteins

Biochemical journal, 2000-02, Vol.345 Pt 3 (3), p.459-466

2000

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LA N-GLYCOSYLATION:MARQUEUR PRECOCE DE LA DIFFERENCIATION ENTEROCYTAIRE DES CELLULES HT-29

Reproduction, nutrition, development, 1989, Vol.29 (Suppl), p.26-26

1989

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Evidence for a dual control of macroautophagic sequestration and intracellular trafficking of N-linked glycoproteins by the trimeric G(i3) protein in HT-29 cells

Biochemical and biophysical research communications, 1997-06, Vol.235 (1), p.166-170

1997

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Beclin 1 and autophagy are required for the tumorigenicity of breast cancer stem-like/progenitor cells

Ceramide-mediated Macroautophagy Involves Inhibition of Protein Kinase B and Up-regulation of Beclin 1

Amino Acids Interfere with the ERK1/2-dependent Control of Macroautophagy by Controlling the Activation of Raf-1 in Human Colon Cancer HT-29 Cells

The Tumor Suppressor PTEN Positively Regulates Macroautophagy by Inhibiting the Phosphatidylinositol 3-Kinase/Protein Kinase B Pathway

Endoplasmic reticulum‐to‐cytosol transport of free polymannose oligosaccharides in permeabilized HepG2 cells

Celecoxib Induces Apoptosis by Inhibiting 3-Phosphoinositide-dependent Protein Kinase-1 Activity in the Human Colon Cancer HT-29 Cell Line

BAG6/BAT3 modulates autophagy by affecting EP300/p300 intracellular localization

A Heterotrimeric G-protein Controls Autophagic Sequestration in the Human Colon Cancer Cell Line HT-29

Autophagy Delays Sulindac Sulfide-Induced Apoptosis in the Human Intestinal Colon Cancer Cell Line HT-29

Human glucocerebrosidase: heterologous expression of active site mutants in murine null cells

Differentiation-dependent autophagy controls the fate of newly synthesized N-linked glycoproteins in the colon adenocarcinoma HT-29 cell line

The G-protein Regulator AGS3 Controls an Early Event during Macroautophagy in Human Intestinal HT-29 Cells

Subcellular localization of the Galphai3 protein and G alpha interacting protein, two proteins involved in the control of macroautophagy in human colon cancer HT-29 cells

The N-glycan processing in HT-29 cells is a function of their state of enterocytic differentiation. Evidence for an atypical traffic associated with change in polypeptide stability in undifferentiated HT-29 cells

Régulation post-traductionnelle de l'expression des protéines N-glycosylées dans des cellules intestinales humaines en culture

Localization and Processing of Glycosylphosphatidylinositol-Anchored Cathepsin D

Transfer of Free Polymannose-Type Oligosaccharides from the Cytosol to Lysosomes in Cultured Human Hepatocellular Carcinoma HEPG2 Cells

Isoforms of the Lutheran/basal cell adhesion molecule glycoprotein are differentially delivered in polarized epithelial cells. Mapping of the basolateral sorting signal to a cytoplasmic di-leucine motif

Requirement of either the NH4Cl-sensitive or the cytochalasin D-sensitive pathway for ricin toxicity depends upon the enterocytic state of differentiation of HT-29 cells

hnRNP G: sequence and characterization of a glycosylated RNA-binding protein

A mucus-secreting human colonic cancer cell line. Purification and partial characterization of the secreted mucins

Processing of asparagine-linked oligosaccharides is an early biochemical marker of the enterocytic differentiation of HT-29 cells

Glucose persistence on high-mannose oligosaccharides selectively inhibits the macroautophagic sequestration of N-linked glycoproteins

LA N-GLYCOSYLATION:MARQUEUR PRECOCE DE LA DIFFERENCIATION ENTEROCYTAIRE DES CELLULES HT-29

Evidence for a dual control of macroautophagic sequestration and intracellular trafficking of N-linked glycoproteins by the trimeric G(i3) protein in HT-29 cells

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