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Regulation of fibrosis in muscular dystrophy

Matrix biology, 2018-08, Vol.68-69, p.602-615

2018

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Actions and interactions of IGF-I and MMPs during muscle regeneration

Seminars in cell & developmental biology, 2021-11, Vol.119, p.11-22

2021

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SMASH - semi-automatic muscle analysis using segmentation of histology: a MATLAB application

Skeletal muscle, 2014-11, Vol.4 (1), p.21-21

2014

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A Zebrafish Embryo Culture System Defines Factors that Promote Vertebrate Myogenesis across Species

Cell, 2013-11, Vol.155 (4), p.909-921

2013

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Role of IGF-I signaling in muscle bone interactions

Bone (New York, N.Y.), 2015-11, Vol.80, p.79-88

2015

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Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse

PloS one, 2012, Vol.7 (1), p.e30063-e30063

2012

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Deleting nebulin's C-terminus reveals its importance to sarcomeric structure and function and is sufficient to invoke nemaline myopathy

Human molecular genetics, 2019-05, Vol.28 (10), p.1709-1725

2019

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Phosphorylation of AMPKα at Ser485/491 Is Dependent on Muscle Contraction and Not Muscle-Specific IGF-I Overexpression

International journal of molecular sciences, 2023-07, Vol.24 (15), p.11950

2023

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Collagen content does not alter the passive mechanical properties of fibrotic skeletal muscle in mdx mice

American Journal of Physiology: Cell Physiology, 2014-05, Vol.306 (10), p.C889-C898

2014

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Activin Receptor Type IIB Inhibition Improves Muscle Phenotype and Function in a Mouse Model of Spinal Muscular Atrophy

PloS one, 2016-11, Vol.11 (11), p.e0166803-e0166803

2016

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Osteopontin ablation ameliorates muscular dystrophy by shifting macrophages to a pro-regenerative phenotype

The Journal of cell biology, 2016-04, Vol.213 (2), p.275-288

2016

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Caspase-12 ablation preserves muscle function in the mdx mouse

Human molecular genetics, 2014-10, Vol.23 (20), p.5325-5341

2014

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Increased collagen cross-linking is a signature of dystrophin-deficient muscle

Muscle & nerve, 2016-06, Vol.54 (1), p.71-78

2016

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Chloroplast transformation for bioencapsulation and oral delivery using the immunoglobulin G fragment crystallizable (Fc) domain

Scientific reports, 2023-11, Vol.13 (1), p.18916-18916, Article 18916

2023

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Antagonistic control of myofiber size and muscle protein quality control by the ubiquitin ligase UBR4 during aging

Nature communications, 2021-03, Vol.12 (1), p.1418-1418, Article 1418

2021

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Insulin-like growth factor-I E-peptide activity is dependent on the IGF-I receptor

PloS one, 2012-09, Vol.7 (9), p.e45588-e45588

2012

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Optimizing IGF-I for skeletal muscle therapeutics

Growth hormone & IGF research, 2014-10, Vol.24 (5), p.157-163

2014

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Regulation of Muscle Mass by Follistatin and Activins

Molecular endocrinology (Baltimore, Md.), 2010-10, Vol.24 (10), p.1998-2008

2010

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The D2.mdx mouse as a preclinical model of the skeletal muscle pathology associated with Duchenne muscular dystrophy

Scientific reports, 2020-08, Vol.10 (1), p.14070-14070, Article 14070

2020

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Pharmacologic approaches to prevent skeletal muscle atrophy after spinal cord injury

Current opinion in pharmacology, 2021-10, Vol.60, p.193-199

2021

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The ABCs of IGF-I isoforms: impact on muscle hypertrophy and implications for repair

Applied physiology, nutrition, and metabolism, 2006-12, Vol.31 (6), p.791

Barton, Elisabeth R

2006

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A Key Role for the Ubiquitin Ligase UBR4 in Myofiber Hypertrophy in Drosophila and Mice

Cell reports (Cambridge), 2019-07, Vol.28 (5), p.1268-1281.e6

2019

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Loss of Calpain 3 dysregulates store‐operated calcium entry and its exercise response in mice

The FASEB journal, 2024-07, Vol.38 (14), p.e23825-n/a

2024

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Genetic and pharmacologic inhibition of mitochondrial-dependent necrosis attenuates muscular dystrophy

Nature medicine, 2008-04, Vol.14 (4), p.442-447

2008

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The insulin-like growth factor (IGF)-I E-peptides are required for isoform-specific gene expression and muscle hypertrophy after local IGF-I production

Journal of applied physiology (1985), 2010-05, Vol.108 (5), p.1069-1076

2010

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Regulation of fibrosis in muscular dystrophy

Actions and interactions of IGF-I and MMPs during muscle regeneration

SMASH - semi-automatic muscle analysis using segmentation of histology: a MATLAB application

A Zebrafish Embryo Culture System Defines Factors that Promote Vertebrate Myogenesis across Species

Role of IGF-I signaling in muscle bone interactions

Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse

Deleting nebulin's C-terminus reveals its importance to sarcomeric structure and function and is sufficient to invoke nemaline myopathy

Phosphorylation of AMPKα at Ser485/491 Is Dependent on Muscle Contraction and Not Muscle-Specific IGF-I Overexpression

Collagen content does not alter the passive mechanical properties of fibrotic skeletal muscle in mdx mice

Activin Receptor Type IIB Inhibition Improves Muscle Phenotype and Function in a Mouse Model of Spinal Muscular Atrophy

Osteopontin ablation ameliorates muscular dystrophy by shifting macrophages to a pro-regenerative phenotype

Caspase-12 ablation preserves muscle function in the mdx mouse

Increased collagen cross-linking is a signature of dystrophin-deficient muscle

Chloroplast transformation for bioencapsulation and oral delivery using the immunoglobulin G fragment crystallizable (Fc) domain

Antagonistic control of myofiber size and muscle protein quality control by the ubiquitin ligase UBR4 during aging

Insulin-like growth factor-I E-peptide activity is dependent on the IGF-I receptor

Optimizing IGF-I for skeletal muscle therapeutics

Regulation of Muscle Mass by Follistatin and Activins

The D2.mdx mouse as a preclinical model of the skeletal muscle pathology associated with Duchenne muscular dystrophy

Pharmacologic approaches to prevent skeletal muscle atrophy after spinal cord injury

The ABCs of IGF-I isoforms: impact on muscle hypertrophy and implications for repair

A Key Role for the Ubiquitin Ligase UBR4 in Myofiber Hypertrophy in Drosophila and Mice

Loss of Calpain 3 dysregulates store‐operated calcium entry and its exercise response in mice

Genetic and pharmacologic inhibition of mitochondrial-dependent necrosis attenuates muscular dystrophy

The insulin-like growth factor (IGF)-I E-peptides are required for isoform-specific gene expression and muscle hypertrophy after local IGF-I production

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