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EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy

Nature communications, 2016-11, Vol.7 (1), p.13685-13685, Article 13685

2016

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Ezh2 Mutations Found in the Weaver Overgrowth Syndrome Cause a Partial Loss of H3K27 Histone Methyltransferase Activity

The journal of clinical endocrinology and metabolism, 2018-04, Vol.103 (4), p.1470-1478

2018

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Loss-of-function variant in SPIN4 causes an X-linked overgrowth syndrome

JCI insight, 2023-05, Vol.8 (9)

2023

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Gradients in bone morphogenetic protein-related gene expression across the growth plate

Journal of endocrinology, 2007-04, Vol.193 (1), p.75-84

2007

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An Extensive Genetic Program Occurring during Postnatal Growth in Multiple Tissues

Endocrinology (Philadelphia), 2009-04, Vol.150 (4), p.1791-1800

2009

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The Role of the Resting Zone in Growth Plate Chondrogenesis

Endocrinology (Philadelphia), 2002-05, Vol.143 (5), p.1851-1857

2002

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Persistent Sox9 expression in hypertrophic chondrocytes suppresses transdifferentiation into osteoblasts

Bone (New York, N.Y.), 2019-08, Vol.125, p.169-177

2019

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Cartilage-Targeted IGF-1 Treatment to Promote Longitudinal Bone Growth

Molecular therapy, 2019-03, Vol.27 (3), p.673-680

2019

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Depletion of resting zone chondrocytes during growth plate senescence

Journal of endocrinology, 2006-04, Vol.189 (1), p.27-36

2006

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An imprinted gene network that controls mammalian somatic growth is down-regulated during postnatal growth deceleration in multiple organs

American journal of physiology. Regulatory, integrative and comparative physiology, 2008-07, Vol.295 (1), p.R189-R196

2008

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Evidence That Estrogen Hastens Epiphyseal Fusion and Cessation of Longitudinal Bone Growth by Irreversibly Depleting the Number of Resting Zone Progenitor Cells in Female Rabbits

Endocrinology (Philadelphia), 2014-08, Vol.155 (8), p.2892-2899

2014

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Effect of Antiandrogen, Aromatase Inhibitor, and Gonadotropin-releasing Hormone Analog on Adult Height in Familial Male Precocious Puberty

The Journal of pediatrics, 2017-11, Vol.190, p.229-235

2017

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Determinants of Growth during Gonadotropin-Releasing Hormone Analog Therapy for Precocious Puberty

The journal of clinical endocrinology and metabolism, 2004-01, Vol.89 (1), p.103-107

2004

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Wnt gene expression in the post-natal growth plate: Regulation with chondrocyte differentiation

Bone (New York, N.Y.), 2007-05, Vol.40 (5), p.1361-1369

2007

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DLG2 variants in patients with pubertal disorders

Genetics in medicine, 2020-08, Vol.22 (8), p.1329-1337

2020

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Growth plate senescence is associated with loss of DNA methylation

Journal of endocrinology, 2005-07, Vol.186 (1), p.241-249

2005

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Coordinated postnatal down-regulation of multiple growth-promoting genes: evidence for a genetic program limiting organ growth

The FASEB journal, 2010-08, Vol.24 (8), p.3083-3092

2010

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Effects of Estrogen on Growth Plate Senescence and Epiphyseal Fusion

Proceedings of the National Academy of Sciences - PNAS, 2001-06, Vol.98 (12), p.6871-6876

2001

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A set of imprinted genes required for normal body growth also promotes growth of rhabdomyosarcoma cells

Pediatric research, 2012-01, Vol.71 (1), p.32-38

2012

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Retinoic Acid Is a Potent Regulator of Growth Plate Chondrogenesis

Endocrinology (Philadelphia), 2000-01, Vol.141 (1), p.346-353

2000

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Catch-Up Growth after Hypothyroidism Is Caused by Delayed Growth Plate Senescence

Endocrinology (Philadelphia), 2008-04, Vol.149 (4), p.1820-1828

2008

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Changes in gene expression associated with aging commonly originate during juvenile growth

Mechanisms of ageing and development, 2010-10, Vol.131 (10), p.641-649

2010

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Regulation of fetal rat bone growth by C-type natriuretic peptide and cGMP

Pediatric research, 2000-02, Vol.47 (2), p.189-193

2000

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Increased Final Height in Precocious Puberty after Long-Term Treatment with LHRH Agonists: The National Institutes of Health Experience

The journal of clinical endocrinology and metabolism, 2001-10, Vol.86 (10), p.4711-4716

2001

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Catch-up growth is associated with delayed senescence of the growth plate in rabbits

Pediatric research, 2001-11, Vol.50 (5), p.618-623

2001

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EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy

Ezh2 Mutations Found in the Weaver Overgrowth Syndrome Cause a Partial Loss of H3K27 Histone Methyltransferase Activity

Loss-of-function variant in SPIN4 causes an X-linked overgrowth syndrome

Gradients in bone morphogenetic protein-related gene expression across the growth plate

An Extensive Genetic Program Occurring during Postnatal Growth in Multiple Tissues

The Role of the Resting Zone in Growth Plate Chondrogenesis

Persistent Sox9 expression in hypertrophic chondrocytes suppresses transdifferentiation into osteoblasts

Cartilage-Targeted IGF-1 Treatment to Promote Longitudinal Bone Growth

Depletion of resting zone chondrocytes during growth plate senescence

An imprinted gene network that controls mammalian somatic growth is down-regulated during postnatal growth deceleration in multiple organs

Evidence That Estrogen Hastens Epiphyseal Fusion and Cessation of Longitudinal Bone Growth by Irreversibly Depleting the Number of Resting Zone Progenitor Cells in Female Rabbits

Effect of Antiandrogen, Aromatase Inhibitor, and Gonadotropin-releasing Hormone Analog on Adult Height in Familial Male Precocious Puberty

Determinants of Growth during Gonadotropin-Releasing Hormone Analog Therapy for Precocious Puberty

Wnt gene expression in the post-natal growth plate: Regulation with chondrocyte differentiation

DLG2 variants in patients with pubertal disorders

Growth plate senescence is associated with loss of DNA methylation

Coordinated postnatal down-regulation of multiple growth-promoting genes: evidence for a genetic program limiting organ growth

Effects of Estrogen on Growth Plate Senescence and Epiphyseal Fusion

A set of imprinted genes required for normal body growth also promotes growth of rhabdomyosarcoma cells

Retinoic Acid Is a Potent Regulator of Growth Plate Chondrogenesis

Catch-Up Growth after Hypothyroidism Is Caused by Delayed Growth Plate Senescence

Changes in gene expression associated with aging commonly originate during juvenile growth

Regulation of fetal rat bone growth by C-type natriuretic peptide and cGMP

Increased Final Height in Precocious Puberty after Long-Term Treatment with LHRH Agonists: The National Institutes of Health Experience

Catch-up growth is associated with delayed senescence of the growth plate in rabbits

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