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Interactions between cancer-associated fibroblasts and tumor cells promote MCL-1 dependency in estrogen receptor-positive breast cancers

Oncogene, 2019-04, Vol.38 (17), p.3261-3273

2019

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Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-10, Vol.5 (10), p.e1429-e1429

2014

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Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-06, Vol.5 (6), p.e1291-e1291

2014

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Significant impact of survivin on myeloma cell growth

Leukemia, 2007-05, Vol.21 (5), p.1070-1078

2007

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Impact of XIAP protein levels on the survival of myeloma cells

Haematologica (Roma), 2009-01, Vol.94 (1), p.87-93

2009

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Regulation of Cancer Cell Survival by BCL2 Family Members upon Prolonged Mitotic Arrest: Opportunities for Anticancer Therapy

Anticancer research, 2012-10, Vol.32 (10), p.4225-4233

2012

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The phenotype of normal, reactive and malignant plasma cells. Identification of "many and multiple myelomas" and of new targets for myeloma therapy

Haematologica (Roma), 2006-09, Vol.91 (9), p.1234-1240

2006

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Cell cycle and cell death associates in western France

Cell death and differentiation, 2007-05, Vol.14 (5), p.1066-1067

2007

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New insights in myeloma-induced osteolysis

Leukemia & lymphoma, 2003-09, Vol.44 (9), p.1463-1467

2003

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Lack of receptor activator of nuclear factor-κB ligand (RANKL) expression and functional production by human multiple myeloma cells

Haematologica (Roma), 2005-02, Vol.90 (2), p.275-278

2005

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Lack of receptor activator of nuclear factor-kB ligand (RANKL) expression and functional production by human multiple myeloma cells

Haematologica (Roma), 2005-02, Vol.90 (2), p.275-278

2005

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Impact of the cGAS/STING pathway on the tumour ecosystem under chemotherapeutic pressure in triple negative breast cancer

Journal of comparative pathology, 2024-04, Vol.210, p.60-60

2024

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Do Human Myeloma Cells Directly Produce the Receptor Activator of Nuclear Factor κB Ligand (RANKL) or Induce RANKL in the Bone Marrow Microenvironment?

Cancer research (Chicago, Ill.), 2004-01, Vol.64 (2), p.772-773

2004

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Survivin Protects Breast Cancer Cells from DNA Damage during Mitosis

Cancer research (Chicago, Ill.), 2009-12, Vol.69 (24_Supplement), p.1124-1124

2009

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Blockade of Notch Signaling by GSI Induces Apoptosis in Human Breast Cancer Cells through Various Molecular Mechanisms Involving Survivin and BH3-Only Members of the Bcl-2 Family

Cancer research (Chicago, Ill.), 2009-12, Vol.69 (24_Supplement), p.2160-2160

2009

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Erratum: Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-10, Vol.5 (10), p.e1429-e1429

2014

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mTORC1 Activity Contributes to the Mcl-1 Dependence of HER2 Amplified Breast Cancer Cells

Cancer research (Chicago, Ill.), 2009-12, Vol.69 (24_Supplement), p.2161-2161

2009

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STING-dependent paracriny shapes apoptotic priming of breast tumors in response to anti-mitotic treatment

Nature communications, 2020-01, Vol.11 (1), p.259-259, Article 259

2020

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Survivin contributes to DNA repair by homologous recombination in breast cancer cells

Breast cancer research and treatment, 2016-01, Vol.155 (1), p.53-63

2016

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miR-720 is a downstream target of an ADAM8-induced ERK signaling cascade that promotes the migratory and invasive phenotype of triple-negative breast cancer cells

Breast cancer research : BCR, 2016-04, Vol.18 (1), p.40-40, Article 40

2016

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BCL-X L directly modulates RAS signalling to favour cancer cell stemness

Nature communications, 2017-10, Vol.8 (1), p.1123

2017

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Novel 1,6-naphthyridin-2(1H)-ones as potential anticancer agents targeting Hsp90

European journal of medicinal chemistry, 2016-08, Vol.119, p.17-33

2016

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E2F1 interacts with BCL‐xL and regulates its subcellular localization dynamics to trigger cell death

EMBO reports, 2018-02, Vol.19 (2), p.234-243

2018

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YM155 potently triggers cell death in breast cancer cells through an autophagy-NF-kB network

Oncotarget, 2015-05, Vol.6 (15), p.13476-13486

2015

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Notch2 signaling sensitizes endothelial cells to apoptosis by negatively regulating the key protective molecule survivin

PloS one, 2009-12, Vol.4 (12), p.e8244

2009

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Interactions between cancer-associated fibroblasts and tumor cells promote MCL-1 dependency in estrogen receptor-positive breast cancers

Bcl-xL controls a switch between cell death modes during mitotic arrest

Bcl-xL controls a switch between cell death modes during mitotic arrest

Significant impact of survivin on myeloma cell growth

Impact of XIAP protein levels on the survival of myeloma cells

Regulation of Cancer Cell Survival by BCL2 Family Members upon Prolonged Mitotic Arrest: Opportunities for Anticancer Therapy

The phenotype of normal, reactive and malignant plasma cells. Identification of "many and multiple myelomas" and of new targets for myeloma therapy

Cell cycle and cell death associates in western France

New insights in myeloma-induced osteolysis

Lack of receptor activator of nuclear factor-κB ligand (RANKL) expression and functional production by human multiple myeloma cells

Lack of receptor activator of nuclear factor-kB ligand (RANKL) expression and functional production by human multiple myeloma cells

Impact of the cGAS/STING pathway on the tumour ecosystem under chemotherapeutic pressure in triple negative breast cancer

Do Human Myeloma Cells Directly Produce the Receptor Activator of Nuclear Factor κB Ligand (RANKL) or Induce RANKL in the Bone Marrow Microenvironment?

Survivin Protects Breast Cancer Cells from DNA Damage during Mitosis

Blockade of Notch Signaling by GSI Induces Apoptosis in Human Breast Cancer Cells through Various Molecular Mechanisms Involving Survivin and BH3-Only Members of the Bcl-2 Family

Erratum: Bcl-xL controls a switch between cell death modes during mitotic arrest

mTORC1 Activity Contributes to the Mcl-1 Dependence of HER2 Amplified Breast Cancer Cells

STING-dependent paracriny shapes apoptotic priming of breast tumors in response to anti-mitotic treatment

Survivin contributes to DNA repair by homologous recombination in breast cancer cells

miR-720 is a downstream target of an ADAM8-induced ERK signaling cascade that promotes the migratory and invasive phenotype of triple-negative breast cancer cells

BCL-X L directly modulates RAS signalling to favour cancer cell stemness

Novel 1,6-naphthyridin-2(1H)-ones as potential anticancer agents targeting Hsp90

E2F1 interacts with BCL‐xL and regulates its subcellular localization dynamics to trigger cell death

YM155 potently triggers cell death in breast cancer cells through an autophagy-NF-kB network

Notch2 signaling sensitizes endothelial cells to apoptosis by negatively regulating the key protective molecule survivin

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