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Gain-of-function mutant p53: history and speculation

Journal of molecular cell biology, 2019-07, Vol.11 (7), p.605-609

2019

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Context-dependent roles of MDMX (MDM4) and MDM2 in breast cancer proliferation and circulating tumor cells

Breast cancer research : BCR, 2019-01, Vol.21 (1), p.5-5, Article 5

2019

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Proteome-wide analysis of mutant p53 targets in breast cancer identifies new levels of gain-of-function that influence PARP, PCNA, and MCM4

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (11), p.E1220-E1229

2015

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Mutant p53 Disrupts Mammary Tissue Architecture via the Mevalonate Pathway

Cell, 2012-01, Vol.148 (1-2), p.244-258

2012

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p53 suppresses MHC class II presentation by intestinal epithelium to protect against radiation-induced gastrointestinal syndrome

Nature communications, 2024-01, Vol.15 (1), p.137-15, Article 137

2024

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A Single Nucleotide Polymorphism in the MDM2 Promoter Attenuates the p53 Tumor Suppressor Pathway and Accelerates Tumor Formation in Humans

Cell, 2004-11, Vol.119 (5), p.591-602

2004

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Hot Spot Mutation in TP53 (R248Q) Causes Oncogenic Gain-of-Function Phenotypes in a Breast Cancer Cell Line Derived from an African American patient

International journal of environmental research and public health, 2015-12, Vol.13 (1), p.ijerph13010022-ijerph13010022

2015

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C. elegans CEP-1/p53 and BEC-1 are involved in DNA repair

PloS one, 2014-02, Vol.9 (2), p.e88828-e88828

2014

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Impedimetric detection of mutant p53 biomarker-driven metastatic breast cancers under hyposmotic pressure

PloS one, 2014-06, Vol.9 (6), p.e99351-e99351

2014

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How Choreostorming Informs Thinking in Molecular Genetics and Cancer Biology

Leonardo (Oxford), 2021-12, Vol.54 (6), p.611-614

Bargonetti, Jill

2021

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Endogenous human MDM2-C is highly expressed in human cancers and functions as a p53-independent growth activator

PloS one, 2013-10, Vol.8 (10), p.e77643-e77643

2013

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Small molecule MMRi62 targets MDM4 for degradation and induces leukemic cell apoptosis regardless of p53 status

Frontiers in oncology, 2022-08, Vol.12, p.933446-933446

2022

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Oligomerization of Mutant p53 R273H is not Required for Gain-of-Function Chromatin Associated Activities

Frontiers in cell and developmental biology, 2021-11, Vol.9, p.772315

2021

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A Protein in the Yeast Saccharomyces cerevisiae Presents DNA Binding Homology to the p53 Checkpoint Protein and Tumor Suppressor

Biomolecules (Basel, Switzerland), 2020-03, Vol.10 (3), p.417

2020

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A Chromatin-associated and Transcriptionally Inactive p53-Mdm2 Complex Occurs in mdm2 SNP309 Homozygous Cells

The Journal of biological chemistry, 2005-07, Vol.280 (29), p.26776-26787

2005

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Identification, validation, and targeting of the mutant p53-PARP-MCM chromatin axis in triple negative breast cancer

NPJ breast cancer, 2017-01, Vol.3 (1), p.1-9, Article 1

2017

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Differential Toxicity of DNA Adducts of Mitomycin C

Journal of Nucleic Acids, 2010-01, Vol.2010 (2010), p.675-680

2010

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Mutant C. elegans p53 Together with Gain-of-Function GLP-1/Notch Decreases UVC-Damage-Induced Germline Cell Death but Increases PARP Inhibitor-Induced Germline Cell Death

Cancers, 2022-10, Vol.14 (19), p.4929

2022

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Phospholipase D Elevates the Level of MDM2 and Suppresses DNA Damage-Induced Increases in p53

Molecular and Cellular Biology, 2004-07, Vol.24 (13), p.5677-5686

2004

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Mutant IC. elegans/I p53 Together with Gain-of-Function GLP-1/Notch Decreases UVC-Damage-Induced Germline Cell Death but Increases PARP Inhibitor-Induced Germline Cell Death

Cancers, 2022-10, Vol.14 (19)

2022

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Mapping DNA Adducts of Mitomycin C and Decarbamoyl Mitomycin C in Cell Lines Using Liquid Chromatography/Electrospray Tandem Mass Spectrometry

Chemical research in toxicology, 2008-12, Vol.21 (12), p.2370-2378

2008

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Wild-type p53 activates transcription in vitro

Nature (London), 1992-07, Vol.358 (6381), p.83-86

1992

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Contemplations on MDMX (MDM4) driving triple negative breast cancer circulating tumor cells and metastasis

Oncotarget, 2019-08, Vol.10 (49), p.5007-5010

2019

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Abstract 2410: The mutant p53 C-terminal domain assists in DNA interactions and cell cycle promotion

Cancer research (Chicago, Ill.), 2021-07, Vol.81 (13_Supplement), p.2410-2410

2021

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The C-terminus of Gain-of-Function Mutant p53 R273H Is Required for Association with PARP1 and Poly-ADP-Ribose

Molecular cancer research, 2022-12, Vol.20 (12), p.1799-1810

2022

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Gain-of-function mutant p53: history and speculation

Context-dependent roles of MDMX (MDM4) and MDM2 in breast cancer proliferation and circulating tumor cells

Proteome-wide analysis of mutant p53 targets in breast cancer identifies new levels of gain-of-function that influence PARP, PCNA, and MCM4

Mutant p53 Disrupts Mammary Tissue Architecture via the Mevalonate Pathway

p53 suppresses MHC class II presentation by intestinal epithelium to protect against radiation-induced gastrointestinal syndrome

A Single Nucleotide Polymorphism in the MDM2 Promoter Attenuates the p53 Tumor Suppressor Pathway and Accelerates Tumor Formation in Humans

Hot Spot Mutation in TP53 (R248Q) Causes Oncogenic Gain-of-Function Phenotypes in a Breast Cancer Cell Line Derived from an African American patient

C. elegans CEP-1/p53 and BEC-1 are involved in DNA repair

Impedimetric detection of mutant p53 biomarker-driven metastatic breast cancers under hyposmotic pressure

How Choreostorming Informs Thinking in Molecular Genetics and Cancer Biology

Endogenous human MDM2-C is highly expressed in human cancers and functions as a p53-independent growth activator

Small molecule MMRi62 targets MDM4 for degradation and induces leukemic cell apoptosis regardless of p53 status

Oligomerization of Mutant p53 R273H is not Required for Gain-of-Function Chromatin Associated Activities

A Protein in the Yeast Saccharomyces cerevisiae Presents DNA Binding Homology to the p53 Checkpoint Protein and Tumor Suppressor

A Chromatin-associated and Transcriptionally Inactive p53-Mdm2 Complex Occurs in mdm2 SNP309 Homozygous Cells

Identification, validation, and targeting of the mutant p53-PARP-MCM chromatin axis in triple negative breast cancer

Differential Toxicity of DNA Adducts of Mitomycin C

Mutant C. elegans p53 Together with Gain-of-Function GLP-1/Notch Decreases UVC-Damage-Induced Germline Cell Death but Increases PARP Inhibitor-Induced Germline Cell Death

Phospholipase D Elevates the Level of MDM2 and Suppresses DNA Damage-Induced Increases in p53

Mutant IC. elegans/I p53 Together with Gain-of-Function GLP-1/Notch Decreases UVC-Damage-Induced Germline Cell Death but Increases PARP Inhibitor-Induced Germline Cell Death

Mapping DNA Adducts of Mitomycin C and Decarbamoyl Mitomycin C in Cell Lines Using Liquid Chromatography/Electrospray Tandem Mass Spectrometry

Wild-type p53 activates transcription in vitro

Contemplations on MDMX (MDM4) driving triple negative breast cancer circulating tumor cells and metastasis

Abstract 2410: The mutant p53 C-terminal domain assists in DNA interactions and cell cycle promotion

The C-terminus of Gain-of-Function Mutant p53 R273H Is Required for Association with PARP1 and Poly-ADP-Ribose

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