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Ano1 is a selective marker of interstitial cells of Cajal in the human and mouse gastrointestinal tract

American journal of physiology: Gastrointestinal and liver physiology, 2009-06, Vol.296 (6), p.G1370-G1381

2009

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Hyperglycemia Increases Interstitial Cells of Cajal via MAPK1 and MAPK3 Signaling to ETV1 and KIT, Leading to Rapid Gastric Emptying

Gastroenterology (New York, N.Y. 1943), 2017-08, Vol.153 (2), p.521-535.e20

2017

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A functional family-wide screening of SP/KLF proteins identifies a subset of suppressors of KRAS-mediated cell growth

Biochemical journal, 2011-04, Vol.435 (2), p.529-537

2011

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Ano1 as a regulator of proliferation

American journal of physiology: Gastrointestinal and liver physiology, 2011-12, Vol.301 (6), p.G1044-G1051

2011

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Progenitors of Interstitial Cells of Cajal in the Postnatal Murine Stomach

Gastroenterology (New York, N.Y. 1943), 2008-04, Vol.134 (4), p.1083-1093

2008

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Differential gene expression in functional classes of interstitial cells of Cajal in murine small intestine

Physiological genomics, 2007-11, Vol.31 (3), p.492-509

2007

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Membrane-to-nucleus signaling links insulin-like growth factor-1- and stem cell factor-activated pathways

PloS one, 2013-10, Vol.8 (10), p.e76822

2013

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Platelet-Derived Growth Factor Receptor-α Regulates Proliferation of Gastrointestinal Stromal Tumor Cells With Mutations in KIT by Stabilizing ETV1

Gastroenterology (New York, N.Y. 1943), 2015-08, Vol.149 (2), p.420-432.e16

2015

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Hedgehog pathway dysregulation contributes to the pathogenesis of human gastrointestinal stromal tumors via GLI-mediated activation of KIT expression

Oncotarget, 2016-11, Vol.7 (48), p.78226-78241

2016

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FAM96A is a novel pro‐apoptotic tumor suppressor in gastrointestinal stromal tumors

International journal of cancer, 2015-09, Vol.137 (6), p.1318-1329

2015

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Loss of Kitlow progenitors, reduced stem cell factor and high oxidative stress underlie gastric dysfunction in progeric mice

The Journal of physiology, 2010-08, Vol.588 (16), p.3101-3117

2010

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Wnt-induced, TRP53-mediated Cell Cycle Arrest of Precursors Underlies Interstitial Cell of Cajal Depletion During Aging

Cellular and molecular gastroenterology and hepatology, 2021-01, Vol.11 (1), p.117-145

2021

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113 Restoration of Kit Expression by Pharmacological Inhibition of Polycomb Group (PcG)-Mediated Epigenetic Repression in Interstitial Cells of Cajal (ICC) Subjected to Chronic Kit Ligand (Kitl) Withdrawal

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-27-S-27

2013

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125 Trp53 Mediates Stem Cell Depletion From Chronically Elevated Canonical Wnt Signaling

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-30-S-30

2012

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Tu1886 Autophagy and Multidrug Resistance Limit the Efficacy of Salinomycin (Sal) in Gastrointestinal Stromal Tumor (GIST) Cells and Stem Cells (SC) for Interstitial Cells of Cajal (ICC)

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-872-S-872

2013

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Tu1166 Salinomycin Enhances Imatinib Effects in Murine Interstitial Cell of Cajal (ICC) Stem Cells and Gastrointestinal Stromal Tumor (GIST) Cells and Inhibits Glycosylation of Kit Protein While Triggering Expression of Truncated Intracellular Kit

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-763-S-764

2012

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124 Effects of Crenolanib, a Highly Selective Platelet-Derived Growth Factor Receptor α/β (PDGFRA/B) Tyrosine Kinase Inhibitor, on the Proliferation of Kii-Mutant Gastrointestinal Stromal Tumor (GIST) Cells and Interstitial Cell of Cajal (ICC) Precursors

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-30-S-30

2012

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275 Adult Stem Cells for Murine Interstitial Cells of Cajal (ICC) Mediate Immunosuppression In Vitro

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-66-S-66

2012

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Persistent β-Catenin Activation Underlies Senescence of Interstitial Cell of Cajal (ICC) Stem Cells in Naturally Aging Mice

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-139-S-139

2011

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Protein Kinase Cγ Mediates Regulation of Proliferation by the Serotonin 5-Hydroxytryptamine Receptor 2B

The Journal of biological chemistry, 2009-08, Vol.284 (32), p.21177-21184

2009

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163 Hyperglycemia-Induced Hyperplasia of Interstitial Cells of Cajal (ICC) and ICC Stem Cells (ICC-SC) Is Associated With Accelerated Gastric Emptying in Obese Diabetic LeprDb/Db Mice

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-38-S-38

2013

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10 The Stem Cell-Targeting Drug Salinomycin Inhibits Proliferation of Normal, Benign Hyperplastic, and Transformed Interstitial Cells of Cajal and Their Stem Cells (ICC-SC) by Increasing the Expression of Cell Cycle Restriction Checkpoint Regulators

Gastroenterology (New York, N.Y. 1943), 2013-05, Vol.144 (5), p.S-3-S-3

2013

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571 KitLow Interstitial Cell of Cajal Stem Cells are a Cause of Resistance to Imatinib Mesylate in Gastrointestinal Stromal Tumors

Gastroenterology (New York, N.Y. 1943), 2010-05, Vol.138 (5), p.S-80-S-80

2010

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T2050 Immunophenotypic Characterization of the Cellular Infiltrate During Murine Experimental Colitis Using Multiparameter Flow Cytometry

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-621-S-621

2010

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Chromatin-Remodeling Events Reveal a Novel Paradigm for the Epigenetic Control of Stem Cell Factor ( Kitl ) Expression in Gastrointestinal Smooth Muscles, Mesenchymal Cells and Stromal Tumors

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-120-S-120

2011

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Ano1 is a selective marker of interstitial cells of Cajal in the human and mouse gastrointestinal tract

Hyperglycemia Increases Interstitial Cells of Cajal via MAPK1 and MAPK3 Signaling to ETV1 and KIT, Leading to Rapid Gastric Emptying

A functional family-wide screening of SP/KLF proteins identifies a subset of suppressors of KRAS-mediated cell growth

Ano1 as a regulator of proliferation

Progenitors of Interstitial Cells of Cajal in the Postnatal Murine Stomach

Differential gene expression in functional classes of interstitial cells of Cajal in murine small intestine

Membrane-to-nucleus signaling links insulin-like growth factor-1- and stem cell factor-activated pathways

Platelet-Derived Growth Factor Receptor-α Regulates Proliferation of Gastrointestinal Stromal Tumor Cells With Mutations in KIT by Stabilizing ETV1

Hedgehog pathway dysregulation contributes to the pathogenesis of human gastrointestinal stromal tumors via GLI-mediated activation of KIT expression

FAM96A is a novel pro‐apoptotic tumor suppressor in gastrointestinal stromal tumors

Loss of Kitlow progenitors, reduced stem cell factor and high oxidative stress underlie gastric dysfunction in progeric mice

Wnt-induced, TRP53-mediated Cell Cycle Arrest of Precursors Underlies Interstitial Cell of Cajal Depletion During Aging

113 Restoration of Kit Expression by Pharmacological Inhibition of Polycomb Group (PcG)-Mediated Epigenetic Repression in Interstitial Cells of Cajal (ICC) Subjected to Chronic Kit Ligand (Kitl) Withdrawal

125 Trp53 Mediates Stem Cell Depletion From Chronically Elevated Canonical Wnt Signaling

Tu1886 Autophagy and Multidrug Resistance Limit the Efficacy of Salinomycin (Sal) in Gastrointestinal Stromal Tumor (GIST) Cells and Stem Cells (SC) for Interstitial Cells of Cajal (ICC)

Tu1166 Salinomycin Enhances Imatinib Effects in Murine Interstitial Cell of Cajal (ICC) Stem Cells and Gastrointestinal Stromal Tumor (GIST) Cells and Inhibits Glycosylation of Kit Protein While Triggering Expression of Truncated Intracellular Kit

124 Effects of Crenolanib, a Highly Selective Platelet-Derived Growth Factor Receptor α/β (PDGFRA/B) Tyrosine Kinase Inhibitor, on the Proliferation of Kii-Mutant Gastrointestinal Stromal Tumor (GIST) Cells and Interstitial Cell of Cajal (ICC) Precursors

275 Adult Stem Cells for Murine Interstitial Cells of Cajal (ICC) Mediate Immunosuppression In Vitro

Persistent β-Catenin Activation Underlies Senescence of Interstitial Cell of Cajal (ICC) Stem Cells in Naturally Aging Mice

Protein Kinase Cγ Mediates Regulation of Proliferation by the Serotonin 5-Hydroxytryptamine Receptor 2B

163 Hyperglycemia-Induced Hyperplasia of Interstitial Cells of Cajal (ICC) and ICC Stem Cells (ICC-SC) Is Associated With Accelerated Gastric Emptying in Obese Diabetic LeprDb/Db Mice

10 The Stem Cell-Targeting Drug Salinomycin Inhibits Proliferation of Normal, Benign Hyperplastic, and Transformed Interstitial Cells of Cajal and Their Stem Cells (ICC-SC) by Increasing the Expression of Cell Cycle Restriction Checkpoint Regulators

571 KitLow Interstitial Cell of Cajal Stem Cells are a Cause of Resistance to Imatinib Mesylate in Gastrointestinal Stromal Tumors

T2050 Immunophenotypic Characterization of the Cellular Infiltrate During Murine Experimental Colitis Using Multiparameter Flow Cytometry

Chromatin-Remodeling Events Reveal a Novel Paradigm for the Epigenetic Control of Stem Cell Factor ( Kitl ) Expression in Gastrointestinal Smooth Muscles, Mesenchymal Cells and Stromal Tumors

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