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PINK1/Parkin Mediated Mitophagy, Ca2+ Signalling, and ER–Mitochondria Contacts in Parkinson’s Disease

International journal of molecular sciences, 2020-03, Vol.21 (5), p.1772

2020

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An expanded palette of improved SPLICS reporters detects multiple organelle contacts in vitro and in vivo

Nature communications, 2020-11, Vol.11 (1), p.6069-6069, Article 6069

2020

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ER-Mitochondria Contact Sites Reporters: Strengths and Weaknesses of the Available Approaches

International journal of molecular sciences, 2020-10, Vol.21 (21), p.8157

2020

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A SPLICS reporter reveals α-synuclein regulation of lysosome-mitochondria contacts which affects TFEB nuclear translocation

Nature communications, 2024-02, Vol.15 (1), p.1516-20

2024

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Perturbation of the host cell Ca2+ homeostasis and ER-mitochondria contact sites by the SARS-CoV-2 structural proteins E and M

Cell death & disease, 2023-04, Vol.14 (4), p.297-297, Article 297

2023

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splitGFP Technology Reveals Dose-Dependent ER-Mitochondria Interface Modulation by α-Synuclein A53T and A30P Mutants

Cells (Basel, Switzerland), 2019-09, Vol.8 (9), p.1072

2019

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The ataxia-linked E1081Q mutation affects the sub-plasma membrane Ca2+-microdomains by tuning PMCA3 activity

Cell death & disease, 2022-10, Vol.13 (10), p.855-855, Article 855

2022

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Stable Integration of Inducible SPLICS Reporters Enables Spatio-Temporal Analysis of Multiple Organelle Contact Sites upon Modulation of Cholesterol Traffic

Cells (Basel, Switzerland), 2022-05, Vol.11 (10), p.1643

2022

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Unraveling Presenilin 2 Functions in a Knockout Zebrafish Line to Shed Light into Alzheimer's Disease Pathogenesis

Cells (Basel, Switzerland), 2023-01, Vol.12 (3), p.376

2023

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Mitochondria Associated Membranes (MAMs): Architecture and physiopathological role

Cell calcium (Edinburgh), 2021-03, Vol.94, p.102343-102343, Article 102343

2021

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Ca2+ signalling: A common language for organelles crosstalk in Parkinson's disease

Cell calcium (Edinburgh), 2023-11, Vol.115, p.102783-102783, Article 102783

2023

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A SPLICS reporter reveals [Formula: see text]-synuclein regulation of lysosome-mitochondria contacts which affects TFEB nuclear translocation

Nature communications, 2024-02, Vol.15 (1), p.1516-1516, Article 1516

2024

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ER-Mitochondria Calcium Transfer, Organelle Contacts and Neurodegenerative Diseases

Calcium Signaling, 2020, Vol.1131, p.719-746

2020

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Sorcin promotes migration in cancer and regulates the EGF-dependent EGFR signaling pathways

Cellular and molecular life sciences : CMLS, 2023-08, Vol.80 (8), p.202-202, Article 202

2023

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A split-GFP tool reveals differences in the sub-mitochondrial distribution of wt and mutant alpha-synuclein

Cell death & disease, 2019-11, Vol.10 (11), p.857-16, Article 857

2019

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Sorcin is an early marker of neurodegeneration, Ca2+ dysregulation and endoplasmic reticulum stress associated to neurodegenerative diseases

Cell death & disease, 2020-10, Vol.11 (10), p.861-861, Article 861

2020

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A SPLICS reporter reveals \documentclass[12pt]{minimal} \usepackage{amsmath} \usepackage{wasysym} \usepackage{amsfonts} \usepackage{amssymb} \usepackage{amsbsy} \usepackage{mathrsfs} \usepackage{upgreek} \setlength{\oddsidemargin}{-69pt} \begin{document}$${{{{{\boldsymbol{\alpha }}}}}}$$\end{document}α-synuclein regulation of lysosome-mitochondria contacts which affects TFEB nuclear translocation

Nature communications, 2024-02, Vol.15

2024

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PINK1/Parkin Mediated Mitophagy, Ca 2+ Signalling, and ER-Mitochondria Contacts in Parkinson's Disease

International journal of molecular sciences, 2020-03, Vol.21 (5)

2020

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Apoptotic signals at the endoplasmic reticulum-mitochondria interface

Advances in protein chemistry and structural biology, 2021, Vol.126, p.307-343

2021

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Perturbation of the host cell Ca 2+ homeostasis and ER-mitochondria contact sites by the SARS-CoV-2 structural proteins E and M

Cell death & disease, 2023-04, Vol.14 (4), p.297

2023

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The ataxia-linked E1081Q mutation affects the sub-plasma membrane Ca 2+ -microdomains by tuning PMCA3 activity

Cell death & disease, 2022-10, Vol.13 (10), p.855

2022

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Sorcin is an early marker of neurodegeneration, Ca 2+ dysregulation and endoplasmic reticulum stress associated to neurodegenerative diseases

Cell death & disease, 2020-10, Vol.11 (10), p.861

2020

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PINK1/Parkin Mediated Mitophagy, Ca2+ Signalling, and ER–Mitochondria Contacts in Parkinson’s Disease

An expanded palette of improved SPLICS reporters detects multiple organelle contacts in vitro and in vivo

ER-Mitochondria Contact Sites Reporters: Strengths and Weaknesses of the Available Approaches

A SPLICS reporter reveals α-synuclein regulation of lysosome-mitochondria contacts which affects TFEB nuclear translocation

Perturbation of the host cell Ca2+ homeostasis and ER-mitochondria contact sites by the SARS-CoV-2 structural proteins E and M

splitGFP Technology Reveals Dose-Dependent ER-Mitochondria Interface Modulation by α-Synuclein A53T and A30P Mutants

The ataxia-linked E1081Q mutation affects the sub-plasma membrane Ca2+-microdomains by tuning PMCA3 activity

Stable Integration of Inducible SPLICS Reporters Enables Spatio-Temporal Analysis of Multiple Organelle Contact Sites upon Modulation of Cholesterol Traffic

Unraveling Presenilin 2 Functions in a Knockout Zebrafish Line to Shed Light into Alzheimer's Disease Pathogenesis

Mitochondria Associated Membranes (MAMs): Architecture and physiopathological role

Ca2+ signalling: A common language for organelles crosstalk in Parkinson's disease

A SPLICS reporter reveals [Formula: see text]-synuclein regulation of lysosome-mitochondria contacts which affects TFEB nuclear translocation

ER-Mitochondria Calcium Transfer, Organelle Contacts and Neurodegenerative Diseases

Sorcin promotes migration in cancer and regulates the EGF-dependent EGFR signaling pathways

A split-GFP tool reveals differences in the sub-mitochondrial distribution of wt and mutant alpha-synuclein

Sorcin is an early marker of neurodegeneration, Ca2+ dysregulation and endoplasmic reticulum stress associated to neurodegenerative diseases

ATP2B2 de novo variants as a cause of variable neurodevelopmental disorders that feature dystonia, ataxia, intellectual disability, behavioral symptoms, and seizures

PINK1/Parkin Mediated Mitophagy, Ca 2+ Signalling, and ER-Mitochondria Contacts in Parkinson's Disease

Apoptotic signals at the endoplasmic reticulum-mitochondria interface

Perturbation of the host cell Ca 2+ homeostasis and ER-mitochondria contact sites by the SARS-CoV-2 structural proteins E and M

The ataxia-linked E1081Q mutation affects the sub-plasma membrane Ca 2+ -microdomains by tuning PMCA3 activity

Sorcin is an early marker of neurodegeneration, Ca 2+ dysregulation and endoplasmic reticulum stress associated to neurodegenerative diseases

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