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Characterization of the human TARDBP gene promoter

Scientific reports, 2021-05, Vol.11 (1), p.10438-13, Article 10438

2021

Volltextzugriff (PDF)

Alternative splicing and liver disease

Annals of hepatology, 2021-12, Vol.26, p.100534-100534, Article 100534

2021

Volltextzugriff (PDF)

TDP-43 regulates its mRNA levels through a negative feedback loop

The EMBO journal, 2011-01, Vol.30 (2), p.277-288

2011

Volltextzugriff (PDF)

Exon and intron definition in pre-mRNA splicing

Wiley interdisciplinary reviews. RNA, 2013-01, Vol.4 (1), p.49-60

2013

Volltextzugriff (PDF)

The role of TDP-43 in the pathogenesis of ALS and FTLD

Biochemical Society transactions, 2013-12, Vol.41 (6), p.1536

2013

Volltextzugriff (PDF)

Nuclear factor TDP-43 can affect selected microRNA levels

The FEBS journal, 2010-05, Vol.277 (10), p.2268-2281

2010

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Age-Related Alternative Splicing: Driver or Passenger in the Aging Process?

Cells (Basel, Switzerland), 2023-12, Vol.12 (24), p.2819

2023

Volltextzugriff (PDF)

Thioridazine reverts the phenotype in cellular and Drosophila models of amyotrophic lateral sclerosis by enhancing TDP-43 aggregate clearance

Neurobiology of disease, 2021-12, Vol.160, p.105515-105515, Article 105515

2021

Volltextzugriff (PDF)

TDP-43 aggregation mirrors TDP-43 knockdown, affecting the expression levels of a common set of proteins

Scientific reports, 2016-09, Vol.6 (1), p.33996-33996, Article 33996

2016

Volltextzugriff (PDF)

TDP-43 affects splicing profiles and isoform production of genes involved in the apoptotic and mitotic cellular pathways

Nucleic acids research, 2015-10, Vol.43 (18), p.8990

2015

Volltextzugriff (PDF)

Physiological tissue-specific and age-related reduction of mouse TDP-43 levels is regulated by epigenetic modifications

Disease models & mechanisms, 2022-04, Vol.15 (4)

2022

Volltextzugriff (PDF)

A Simplified and Efficient Process for Insulin Production in Pichia pastoris

PloS one, 2016-12, Vol.11 (12), p.e0167207-e0167207

2016

Volltextzugriff (PDF)

Predominance of spliceosomal complex formation over polyadenylation site selection in TDP-43 autoregulation

Nucleic acids research, 2014-03, Vol.42 (5), p.3362

2014

Volltextzugriff (PDF)

Force-feeding malignant mesothelioma stem-cell like with exosome-delivered miR-126 induces tumour cell killing

Translational oncology, 2022-06, Vol.20, p.101400-101400, Article 101400

2022

Volltextzugriff (PDF)

An Amyloid-Like Pathological Conformation of TDP-43 Is Stabilized by Hypercooperative Hydrogen Bonds

Frontiers in molecular neuroscience, 2016-11, Vol.9, p.125-125

2016

Volltextzugriff (PDF)

The splicing code

BioSystems, 2018-02, Vol.164, p.39-48

2018

Volltextzugriff (PDF)

NF1 mRNA biogenesis: Effect of the genomic milieu in splicing regulation of the NF1 exon 37 region

FEBS letters, 2006-08, Vol.580 (18), p.4449-4456

2006

Volltextzugriff (PDF)

High Throughput miRNA Screening Identifies miR-574-3p Hyperproductive Effect in CHO Cells

Biomolecules (Basel, Switzerland), 2021-07, Vol.11 (8), p.1125

2021

Volltextzugriff (PDF)

Emerging Trends in the Field of Inflammation and Proteinopathy in ALS/FTD Spectrum Disorder

Biomedicines, 2023-05, Vol.11 (6), p.1599

2023

Volltextzugriff (PDF)

Functional Analysis of Mutations in Exon 9 of NF1 Reveals the Presence of Several Elements Regulating Splicing

PloS one, 2015-10, Vol.10 (10), p.e0141735-e0141735

2015

Volltextzugriff (PDF)

TDP‐43 regulates its mRNA levels through a negative feedback loop

The EMBO journal, 2011-01, Vol.30 (2), p.277-288

2011

Volltextzugriff (PDF)

Neurodegeneration and RNA‐binding proteins

Wiley interdisciplinary reviews. RNA, 2017-03, Vol.8 (2), p.np-n/a

2017

Volltextzugriff (PDF)

Complexities of 5'splice site definition: Implications in clinical analyses

RNA biology, 2012-06, Vol.9 (6), p.911-923

2012

Volltextzugriff (PDF)

Absence of TDP-43 is difficult to digest

The EMBO journal, 2016-01, Vol.35 (2), p.115-117

2016

Volltextzugriff (PDF)

Tailored antisense oligonucleotides designed to correct aberrant splicing reveal actionable groups of mutations for rare genetic disorders

Experimental & molecular medicine, 2024-08

2024

Volltextzugriff (PDF)

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Characterization of the human TARDBP gene promoter

Alternative splicing and liver disease

TDP-43 regulates its mRNA levels through a negative feedback loop

Exon and intron definition in pre-mRNA splicing

The role of TDP-43 in the pathogenesis of ALS and FTLD

Nuclear factor TDP-43 can affect selected microRNA levels

Age-Related Alternative Splicing: Driver or Passenger in the Aging Process?

Thioridazine reverts the phenotype in cellular and Drosophila models of amyotrophic lateral sclerosis by enhancing TDP-43 aggregate clearance

TDP-43 aggregation mirrors TDP-43 knockdown, affecting the expression levels of a common set of proteins

TDP-43 affects splicing profiles and isoform production of genes involved in the apoptotic and mitotic cellular pathways

Physiological tissue-specific and age-related reduction of mouse TDP-43 levels is regulated by epigenetic modifications

A Simplified and Efficient Process for Insulin Production in Pichia pastoris

Predominance of spliceosomal complex formation over polyadenylation site selection in TDP-43 autoregulation

Force-feeding malignant mesothelioma stem-cell like with exosome-delivered miR-126 induces tumour cell killing

An Amyloid-Like Pathological Conformation of TDP-43 Is Stabilized by Hypercooperative Hydrogen Bonds

The splicing code

NF1 mRNA biogenesis: Effect of the genomic milieu in splicing regulation of the NF1 exon 37 region

High Throughput miRNA Screening Identifies miR-574-3p Hyperproductive Effect in CHO Cells

Emerging Trends in the Field of Inflammation and Proteinopathy in ALS/FTD Spectrum Disorder

Functional Analysis of Mutations in Exon 9 of NF1 Reveals the Presence of Several Elements Regulating Splicing

TDP‐43 regulates its mRNA levels through a negative feedback loop

Neurodegeneration and RNA‐binding proteins

Complexities of 5'splice site definition: Implications in clinical analyses

Absence of TDP-43 is difficult to digest

Tailored antisense oligonucleotides designed to correct aberrant splicing reveal actionable groups of mutations for rare genetic disorders

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