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TMEM16A/ANO1 Channels Contribute to the Myogenic Response in Cerebral Arteries

Circulation research, 2012-09, Vol.111 (8), p.1027-1036

2012

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Dynamic regulation of β1 subunit trafficking controls vascular contractility

Proceedings of the National Academy of Sciences - PNAS, 2014-02, Vol.111 (6), p.2361-2366

2014

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TMEM16A channels generate Ca²⁺-activated Cl⁻ currents in cerebral artery smooth muscle cells

American journal of physiology. Heart and circulatory physiology, 2011-11, Vol.301 (5), p.H1819-H1827

2011

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Transcriptional Upregulation of α2δ-1 Elevates Arterial Smooth Muscle Cell Voltage-Dependent Ca2+ Channel Surface Expression and Cerebrovascular Constriction in Genetic Hypertension

Hypertension (Dallas, Tex. 1979), 2012-10, Vol.60 (4), p.1006-1015

2012

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Smooth Muscle Cell α2δ-1 Subunits Are Essential for Vasoregulation by CaV1.2 Channels

Circulation research, 2009-11, Vol.105 (10), p.948-955

2009

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LRRC26 Is a Functional BK Channel Auxiliary γ Subunit in Arterial Smooth Muscle Cells

Circulation research, 2014-08, Vol.115 (4), p.423-431

2014

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A Novel CaV1.2 N Terminus Expressed in Smooth Muscle Cells of Resistance Size Arteries Modifies Channel Regulation by Auxiliary Subunits

The Journal of biological chemistry, 2007-10, Vol.282 (40), p.29211-29221

2007

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The voltage‐dependent L‐type Ca2+ (CaV1.2) channel C‐terminus fragment is a bi‐modal vasodilator

The Journal of physiology, 2013-06, Vol.591 (12), p.2987-2998

2013

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Angiotensin II stimulates internalization and degradation of arterial myocyte plasma membrane BK channels to induce vasoconstriction

American Journal of Physiology: Cell Physiology, 2015-09, Vol.309 (6), p.C392-C402

2015

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Mutations in voltage-gated potassium channel KCNC3 cause degenerative and developmental central nervous system phenotypes

Nature genetics, 2006-04, Vol.38 (4), p.447-451

2006

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Intravascular pressure enhances the abundance of functional Kv1.5 channels at the surface of arterial smooth muscle cells

Science signaling, 2015-08, Vol.8 (390), p.ra83

2015

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Isoform-Selective Physical Coupling of TRPC3 Channels to IP3 Receptors in Smooth Muscle Cells Regulates Arterial Contractility

Circulation research, 2010-05, Vol.106 (10), p.1603-1612

2010

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Optical detection of rate-determining ion-modulated conformational changes of the ether-à-go-go K+ channel voltage sensor

Proceedings of the National Academy of Sciences - PNAS, 2005-12, Vol.102 (51), p.18718-18723

2005

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Rab25 influences functional Cav1.2 channel surface expression in arterial smooth muscle cells

American Journal of Physiology: Cell Physiology, 2016-06, Vol.310 (11), p.C885-C893

2016

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Atomic Proximity between S4 Segment and Pore Domain in Shaker Potassium Channels

Neuron (Cambridge, Mass.), 2003-07, Vol.39 (3), p.467-481

2003

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A novel Ca(V)1.2 N terminus expressed in smooth muscle cells of resistance size arteries modifies channel regulation by auxiliary subunits

The Journal of biological chemistry, 2007-10, Vol.282 (40), p.29211-29221

2007

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Role of hydrophobic and ionic forces in the movement of S4 of the Shaker potassium channel

Molecular membrane biology, 2012-12, Vol.29 (8), p.321-332

2012

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Binding Site in Eag Voltage Sensor Accommodates a Variety of Ions and is Accessible in Closed Channel

Biophysical journal, 2004-11, Vol.87 (5), p.3110-3121

2004

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Ca(V)1.2 channel N-terminal splice variants modulate functional surface expression in resistance size artery smooth muscle cells

The Journal of biological chemistry, 2011-04, Vol.286 (17), p.15058-15066

2011

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Rab25 stimulates functional Ca V 1.2 channel surface expression in arterial smooth muscle cells

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Rab25 influences functional Ca sub( v)1.2 channel surface expression in arterial smooth muscle cells

American Journal of Physiology: Cell Physiology, 2016-06, Vol.310 (11), p.C885-C885

2016

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Rab25 influences functional Ca v 1.2 channel surface expression in arterial smooth muscle cells

American Journal of Physiology: Cell Physiology, 2016-06, Vol.310 (11), p.C885-C893

2016

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Rab25 influences functional Ca^sub v^1.2 channel surface expression in arterial smooth muscle cells

American Journal of Physiology: Cell Physiology, 2016-06, Vol.310 (11), p.C885

2016

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Intravascular pressure enhances the abundance of functional K v 1.5 channels at the surface of arterial smooth muscle cells

Science signaling, 2015-08, Vol.8 (390)

2015

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CaV1.2 Channel N-terminal Splice Variants Modulate Functional Surface Expression in Resistance Size Artery Smooth Muscle Cells

The Journal of biological chemistry, 2011-04, Vol.286 (17), p.15058-15066

2011

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TMEM16A/ANO1 Channels Contribute to the Myogenic Response in Cerebral Arteries

Dynamic regulation of β1 subunit trafficking controls vascular contractility

TMEM16A channels generate Ca²⁺-activated Cl⁻ currents in cerebral artery smooth muscle cells

Transcriptional Upregulation of α2δ-1 Elevates Arterial Smooth Muscle Cell Voltage-Dependent Ca2+ Channel Surface Expression and Cerebrovascular Constriction in Genetic Hypertension

Smooth Muscle Cell α2δ-1 Subunits Are Essential for Vasoregulation by CaV1.2 Channels

LRRC26 Is a Functional BK Channel Auxiliary γ Subunit in Arterial Smooth Muscle Cells

A Novel CaV1.2 N Terminus Expressed in Smooth Muscle Cells of Resistance Size Arteries Modifies Channel Regulation by Auxiliary Subunits

The voltage‐dependent L‐type Ca2+ (CaV1.2) channel C‐terminus fragment is a bi‐modal vasodilator

Angiotensin II stimulates internalization and degradation of arterial myocyte plasma membrane BK channels to induce vasoconstriction

Mutations in voltage-gated potassium channel KCNC3 cause degenerative and developmental central nervous system phenotypes

Intravascular pressure enhances the abundance of functional Kv1.5 channels at the surface of arterial smooth muscle cells

Isoform-Selective Physical Coupling of TRPC3 Channels to IP3 Receptors in Smooth Muscle Cells Regulates Arterial Contractility

Optical detection of rate-determining ion-modulated conformational changes of the ether-à-go-go K+ channel voltage sensor

Rab25 influences functional Cav1.2 channel surface expression in arterial smooth muscle cells

Atomic Proximity between S4 Segment and Pore Domain in Shaker Potassium Channels

A novel Ca(V)1.2 N terminus expressed in smooth muscle cells of resistance size arteries modifies channel regulation by auxiliary subunits

Role of hydrophobic and ionic forces in the movement of S4 of the Shaker potassium channel

Binding Site in Eag Voltage Sensor Accommodates a Variety of Ions and is Accessible in Closed Channel

Ca(V)1.2 channel N-terminal splice variants modulate functional surface expression in resistance size artery smooth muscle cells

Rab25 stimulates functional Ca V 1.2 channel surface expression in arterial smooth muscle cells

Rab25 influences functional Ca sub( v)1.2 channel surface expression in arterial smooth muscle cells

Rab25 influences functional Ca v 1.2 channel surface expression in arterial smooth muscle cells

Rab25 influences functional Ca^sub v^1.2 channel surface expression in arterial smooth muscle cells

Intravascular pressure enhances the abundance of functional K v 1.5 channels at the surface of arterial smooth muscle cells

CaV1.2 Channel N-terminal Splice Variants Modulate Functional Surface Expression in Resistance Size Artery Smooth Muscle Cells

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