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Of channels and pumps: different ways to boost the aldosterone?

Acta Physiologica, 2017-07, Vol.220 (3), p.332-360

Bandulik, S.

2017

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Pathogenesis of Adrenal Aldosterone-Producing Adenomas Carrying Mutations of the Na+/K+-ATPase

Endocrinology (Philadelphia), 2015-12, Vol.156 (12), p.4582-4591

2015

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Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca2+-ATPase ATP2B3

Endocrinology (Philadelphia), 2016-06, Vol.157 (6), p.2489-2499

2016

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Pharmacology and Pathophysiology of Mutated KCNJ5 Found in Adrenal Aldosterone-Producing Adenomas

Endocrinology (Philadelphia), 2014-04, Vol.155 (4), p.1353-1362

2014

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Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca(2+)-ATPase ATP2B3

Endocrinology (Philadelphia), 2016-06, Vol.157 (6), p.2489-2499

2016

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TASK1 and TASK3 potassium channels: determinants of aldosterone secretion and adrenocortical zonation

Hormone and metabolic research, 2010-06, Vol.42 (6), p.450

2010

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6C.03: A CASE OF SEVERE HYPERALDOSTERONISM CAUSED BY A DE NOVO KCNJ5 MUTATION

Journal of hypertension, 2015-06, Vol.33 Suppl 1 - ESH 2015 Abstract Book (Supplement 1), p.e79-e80

2015

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357 Correction of the salt-losing phenotype in cystic fibrosis after elexacaftor-tezacaftor-ivacaftor treatment

Journal of cystic fibrosis, 2023-10, Vol.22, p.S180-S180

2023

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WS04.03 Elexacaftor/tezacaftor/ivacaftor (ETI) treatment corrects the salt-losing phenotype in people with cystic fibrosis (pwCF)

Journal of cystic fibrosis, 2023-06, Vol.22, p.S8-S8

2023

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Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca super(2+)-ATPase ATP2B3

Endocrinology (Philadelphia), 2016-06, Vol.157 (6), p.2489-2499

2016

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TASK1 and TASK3 Potassium Channels: Determinants of Aldosterone Secretion and Adrenocortical Zonation

Hormone and metabolic research, 2010-06, Vol.42 (6), p.450-457

2010

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Pathophysiology of Na+/K+-atpases in aldosterone secretion

Experimental and clinical endocrinology & diabetes, 2015, Vol.122 (3)

2015

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Cellular pathology of the mutated plasma membrane Ca2+ atpase ATP2B3 found in adrenal adenomas

Experimental and clinical endocrinology & diabetes, 2015, Vol.122 (3)

2015

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Ezetimib influences the expression of raft‐associated antigens in human monocytes

Cytometry. Part A, 2006-03, Vol.69A (3), p.206-208

2006

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Sekundäre Hypertonie durch primären Hyperaldosteronismus: Warum macht die Nebenniere, was sie will?

Der Nephrologe, 2018-11, Vol.13 (6), p.421-424

Bandulik, S.

2018

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Rare but Relevant Kidney Disorders

Clinical journal of the American Society of Nephrology, 2009-11, Vol.4 (11), p.1701-1704

2009

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W16-P-078 Aminopeptidase N (CD13) acts as aco-receptor for the enterocytic micellar cholesterol absorption through ‘deep tubular invaginations’

Atherosclerosis. Supplements, 2005-04, Vol.6 (1), p.120-121

2005

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W12-P-048 Ezetimib modulates the in vitrodifferentiation and membrane microdomain function in human macrophages

Atherosclerosis. Supplements, 2005-04, Vol.6 (1), p.73-73

2005

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EAST-Syndrom: Ein neues Krankheitsbild mit renalem Salzverlust

Der Nephrologe, 2011-11, Vol.6 (6), p.529-536

2011

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Cellular Pathophysiology of Mutant Voltage-Dependent Ca2+ Channel CACNA1H in Primary Aldosteronism

Endocrinology (Philadelphia), 2020-10, Vol.161 (10)

2020

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The ex vivo perfused mouse adrenal gland—a new model to study aldosterone secretion

Pflügers Archiv, 2024-06, Vol.476 (6), p.911-922

2024

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Collecting system–specific deletion of Kcnj10 predisposes for thiazide- and low-potassium diet–induced hypokalemia

Kidney international, 2020-06, Vol.97 (6), p.1208-1218

2020

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Two-pore domain potassium channels in the adrenal cortex

Pflügers Archiv, 2015-05, Vol.467 (5), p.1027-1042

2015

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KCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel function

Proceedings of the National Academy of Sciences - PNAS, 2010-08, Vol.107 (32), p.14490-14495

2010

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A Case of Severe Hyperaldosteronism Caused by a De Novo Mutation Affecting a Critical Salt Bridge Kir3.4 Residue

The journal of clinical endocrinology and metabolism, 2015-01, Vol.100 (1), p.E114-E118

2015

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Of channels and pumps: different ways to boost the aldosterone?

Pathogenesis of Adrenal Aldosterone-Producing Adenomas Carrying Mutations of the Na+/K+-ATPase

Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca2+-ATPase ATP2B3

Pharmacology and Pathophysiology of Mutated KCNJ5 Found in Adrenal Aldosterone-Producing Adenomas

Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca(2+)-ATPase ATP2B3

TASK1 and TASK3 potassium channels: determinants of aldosterone secretion and adrenocortical zonation

6C.03: A CASE OF SEVERE HYPERALDOSTERONISM CAUSED BY A DE NOVO KCNJ5 MUTATION

357 Correction of the salt-losing phenotype in cystic fibrosis after elexacaftor-tezacaftor-ivacaftor treatment

WS04.03 Elexacaftor/tezacaftor/ivacaftor (ETI) treatment corrects the salt-losing phenotype in people with cystic fibrosis (pwCF)

Cellular Pathophysiology of an Adrenal Adenoma-Associated Mutant of the Plasma Membrane Ca super(2+)-ATPase ATP2B3

TASK1 and TASK3 Potassium Channels: Determinants of Aldosterone Secretion and Adrenocortical Zonation

Pathophysiology of Na+/K+-atpases in aldosterone secretion

Cellular pathology of the mutated plasma membrane Ca2+ atpase ATP2B3 found in adrenal adenomas

Ezetimib influences the expression of raft‐associated antigens in human monocytes

Sekundäre Hypertonie durch primären Hyperaldosteronismus: Warum macht die Nebenniere, was sie will?

Rare but Relevant Kidney Disorders

W16-P-078 Aminopeptidase N (CD13) acts as aco-receptor for the enterocytic micellar cholesterol absorption through ‘deep tubular invaginations’

W12-P-048 Ezetimib modulates the in vitrodifferentiation and membrane microdomain function in human macrophages

EAST-Syndrom: Ein neues Krankheitsbild mit renalem Salzverlust

Cellular Pathophysiology of Mutant Voltage-Dependent Ca2+ Channel CACNA1H in Primary Aldosteronism

The ex vivo perfused mouse adrenal gland—a new model to study aldosterone secretion

Collecting system–specific deletion of Kcnj10 predisposes for thiazide- and low-potassium diet–induced hypokalemia

Two-pore domain potassium channels in the adrenal cortex

KCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel function

A Case of Severe Hyperaldosteronism Caused by a De Novo Mutation Affecting a Critical Salt Bridge Kir3.4 Residue

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