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PrimPol Is Required for Replicative Tolerance of G Quadruplexes in Vertebrate Cells

Molecular cell, 2016-01, Vol.61 (1), p.161-169

2016

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PrimPol Bypasses UV Photoproducts during Eukaryotic Chromosomal DNA Replication

Molecular cell, 2013-11, Vol.52 (4), p.566-573

2013

Volltextzugriff (PDF)

Mitochondrial DNA replication: a PrimPol perspective

Biochemical Society transactions, 2017-04, Vol.45 (2), p.513

2017

Volltextzugriff (PDF)

Molecular dissection of the domain architecture and catalytic activities of human PrimPol

Nucleic acids research, 2014-05, Vol.42 (9), p.5830

2014

Volltextzugriff (PDF)

Repriming by PrimPol is critical for DNA replication restart downstream of lesions and chain-terminating nucleosides

Cell cycle (Georgetown, Tex.), 2016-08, Vol.15 (15), p.1997-2008

2016

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Molecular basis for PrimPol recruitment to replication forks by RPA

Nature communications, 2017-05, Vol.8 (1), p.15222-15222, Article 15222

2017

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PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts

Nature communications, 2020-11, Vol.11 (1), p.5863-14, Article 5863

2020

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Human PrimPol is a highly error-prone polymerase regulated by single-stranded DNA binding proteins

Nucleic acids research, 2015-01, Vol.43 (2), p.1056

2015

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The involvement of tau in nucleolar transcription and the stress response

Acta neuropathologica communications, 2018-07, Vol.6 (1), p.70-13, Article 70

2018

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Pathological ribonuclease H1 causes R-loop depletion and aberrant DNA segregation in mitochondria

Proceedings of the National Academy of Sciences - PNAS, 2016-07, Vol.113 (30), p.E4276-E4285

2016

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The Involvement of Aβ42 and Tau in Nucleolar and Protein Synthesis Machinery Dysfunction

Frontiers in cellular neuroscience, 2018-08, Vol.12, p.220

2018

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Mammalian Mitochondrial DNA Replication Intermediates Are Essentially Duplex but Contain Extensive Tracts of RNA/DNA Hybrid

Journal of molecular biology, 2010-04, Vol.397 (5), p.1144-1155

2010

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PrimPol-deficient cells exhibit a pronounced G2 checkpoint response following UV damage

Cell cycle (Georgetown, Tex.), 2016-01, Vol.15 (7), p.908-918

2016

Volltextzugriff (PDF)

Mice expressing an error-prone DNA polymerase in mitochondria display elevated replication pausing and chromosomal breakage at fragile sites of mitochondrial DNA

Nucleic acids research, 2009-04, Vol.37 (7), p.2327

2009

Volltextzugriff (PDF)

PrimPol is required for the maintenance of efficient nuclear and mitochondrial DNA replication in human cells

Nucleic acids research, 2019-05, Vol.47 (8), p.4026

2019

Volltextzugriff (PDF)

PolDIP2 interacts with human PrimPol and enhances its DNA polymerase activities

Nucleic acids research, 2016-04, Vol.44 (7), p.3317

2016

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Human PrimPol mutation associated with high myopia has a DNA replication defect

Nucleic acids research, 2014-10, Vol.42 (19), p.12102

2014

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EXD2 governs germ stem cell homeostasis and lifespan by promoting mitoribosome integrity and translation

Nature cell biology, 2018-02, Vol.20 (2), p.162

2018

Volltextzugriff (PDF)

PLK1 regulates the PrimPol damage tolerance pathway during the cell cycle

Science advances, 2021-12, Vol.7 (49), p.eabh1004-eabh1004

2021

Volltextzugriff (PDF)

Author response: PRIMPOL mutation: functional study does not always reveal the truth

Investigative ophthalmology & visual science, 2015-02, Vol.56 (2), p.1183-1183

2015

Volltextzugriff (PDF)

We are what we do: research outputs of public health

American journal of preventive medicine, 2008-10, Vol.35 (4), p.380-385

2008

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Helping hospital patients quit: What the evidence supports and what guidelines recommend

Preventive medicine, 2008-04, Vol.46 (4), p.346-357

2008

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We Are What We Do

American journal of preventive medicine, 2008, Vol.35 (4), p.380-385

2008

Volltextzugriff (PDF)

Review: Helping hospital patients quit: What the evidence supports and what guidelines recommend

Preventive medicine, 2008-04, Vol.46 (4), p.346-357

2008

Volltextzugriff (PDF)

PrimPol-deficient cells exhibit a pronounced G2 checkpoint response following UV damage

Cell Cycle, 2016, Vol.15 (7), p.908-918

2016

Volltextzugriff (PDF)

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PrimPol Is Required for Replicative Tolerance of G Quadruplexes in Vertebrate Cells

PrimPol Bypasses UV Photoproducts during Eukaryotic Chromosomal DNA Replication

Mitochondrial DNA replication: a PrimPol perspective

Molecular dissection of the domain architecture and catalytic activities of human PrimPol

Repriming by PrimPol is critical for DNA replication restart downstream of lesions and chain-terminating nucleosides

Molecular basis for PrimPol recruitment to replication forks by RPA

PrimPol-dependent single-stranded gap formation mediates homologous recombination at bulky DNA adducts

Human PrimPol is a highly error-prone polymerase regulated by single-stranded DNA binding proteins

The involvement of tau in nucleolar transcription and the stress response

Pathological ribonuclease H1 causes R-loop depletion and aberrant DNA segregation in mitochondria

The Involvement of Aβ42 and Tau in Nucleolar and Protein Synthesis Machinery Dysfunction

Mammalian Mitochondrial DNA Replication Intermediates Are Essentially Duplex but Contain Extensive Tracts of RNA/DNA Hybrid

PrimPol-deficient cells exhibit a pronounced G2 checkpoint response following UV damage

Mice expressing an error-prone DNA polymerase in mitochondria display elevated replication pausing and chromosomal breakage at fragile sites of mitochondrial DNA

PrimPol is required for the maintenance of efficient nuclear and mitochondrial DNA replication in human cells

PolDIP2 interacts with human PrimPol and enhances its DNA polymerase activities

Human PrimPol mutation associated with high myopia has a DNA replication defect

EXD2 governs germ stem cell homeostasis and lifespan by promoting mitoribosome integrity and translation

PLK1 regulates the PrimPol damage tolerance pathway during the cell cycle

Author response: PRIMPOL mutation: functional study does not always reveal the truth

We are what we do: research outputs of public health

Helping hospital patients quit: What the evidence supports and what guidelines recommend

We Are What We Do

Review: Helping hospital patients quit: What the evidence supports and what guidelines recommend

PrimPol-deficient cells exhibit a pronounced G2 checkpoint response following UV damage

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