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Soluble immune complexes shift the TLR-induced cytokine production of distinct polarized human macrophage subsets towards IL-10

PloS one, 2012-04, Vol.7 (4), p.e35994

2012

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Anti-citrullinated protein antibody response after primary EBV infection in kidney transplant patients

PloS one, 2018-05, Vol.13 (5), p.e0197219-e0197219

2018

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Sustained remission with methotrexate monotherapy after 22-week induction treatment with TNF-alpha inhibitor and methotrexate in early psoriatic arthritis: an open-label extension of a randomized placebo-controlled trial

Arthritis research & therapy, 2019-09, Vol.21 (1), p.208-208, Article 208

2019

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APRIL Induces a Novel Subset of IgA + Regulatory B Cells That Suppress Inflammation via Expression of IL-10 and PD-L1

Frontiers in immunology, 2019-06, Vol.10, p.1368-1368

2019

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Fc gamma receptor-TLR cross-talk elicits pro-inflammatory cytokine production by human M2 macrophages

Nature communications, 2014-11, Vol.5 (1), p.5444-5444, Article 5444

2014

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Control of cytokine production by human fc gamma receptors: implications for pathogen defense and autoimmunity

Frontiers in immunology, 2015-02, Vol.6, p.79-79

2015

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Intimal lining layer macrophages but not synovial sublining macrophages display an IL-10 polarized-like phenotype in chronic synovitis

Arthritis research & therapy, 2012-04, Vol.14 (2), p.R74-R74, Article R74

2012

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C-Reactive Protein Controls IL-23 Production by Human Monocytes

International journal of molecular sciences, 2021-10, Vol.22 (21), p.11638

2021

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Anti-IL-17A treatment reduces serum inflammatory, angiogenic and tissue remodeling biomarkers accompanied by less synovial high endothelial venules in peripheral spondyloarthritis

Scientific reports, 2020-12, Vol.10 (1), p.21094-21094, Article 21094

2020

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The inflammatory function of human IgA

Cellular and molecular life sciences : CMLS, 2019-03, Vol.76 (6), p.1041-1055

2019

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FcαRI co-stimulation converts human intestinal CD103+ dendritic cells into pro-inflammatory cells through glycolytic reprogramming

Nature communications, 2018-02, Vol.9 (1), p.863-13, Article 863

2018

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IL-12p40/IL-23p40 Blockade With Ustekinumab Decreases the Synovial Inflammatory Infiltrate Through Modulation of Multiple Signaling Pathways Including MAPK-ERK and Wnt

Frontiers in immunology, 2021-03, Vol.12, p.611656-611656

2021

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FcγR-TLR Cross-Talk Enhances TNF Production by Human Monocyte-Derived DCs via IRF5-Dependent Gene Transcription and Glycolytic Reprogramming

Frontiers in immunology, 2019-04, Vol.10, p.739-739

2019

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Fc gamma receptor IIa suppresses type I and III interferon production by human myeloid immune cells

European journal of immunology, 2018-11, Vol.48 (11), p.1796-1809

2018

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Serum IgA Immune Complexes Promote Proinflammatory Cytokine Production by Human Macrophages, Monocytes, and Kupffer Cells through FcαRI-TLR Cross-Talk

The Journal of immunology (1950), 2017-12, Vol.199 (12), p.4124-4131

2017

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Residual disease activity and treatment adjustments in psoriatic arthritis in current clinical practice

Arthritis research & therapy, 2017-10, Vol.19 (1), p.226-8, Article 226

2017

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The Transcriptional Coactivator Bob1 Is Associated With Pathologic B Cell Responses in Autoimmune Tissue Inflammation

Arthritis & rheumatology (Hoboken, N.J.), 2017-04, Vol.69 (4), p.750-762

2017

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The novel cytokine interleukin-36α is expressed in psoriatic and rheumatoid arthritis synovium

Annals of the rheumatic diseases, 2013-09, Vol.72 (9), p.1569-1574

2013

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The IL-17A-Producing CD8+ T-Cell Population in Psoriatic Lesional Skin Comprises Mucosa-Associated Invariant T Cells and Conventional T Cells

Journal of investigative dermatology, 2014-12, Vol.134 (12), p.2898-2907

2014

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How Cytokine Networks Fuel Inflammation: Interleukin-17 and a tale of two autoimmune diseases

Nature medicine, 2013-07, Vol.19 (7), p.824-825

2013

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C-Reactive Protein Promotes Inflammation through FcγR-Induced Glycolytic Reprogramming of Human Macrophages

The Journal of immunology (1950), 2019-07, Vol.203 (1), p.225-235

2019

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Transmembrane TNF drives osteoproliferative joint inflammation reminiscent of human spondyloarthritis

The Journal of experimental medicine, 2020-10, Vol.217 (10)

2020

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Expansion of Interleukin‐22– and Granulocyte–Macrophage Colony‐Stimulating Factor–Expressing, but Not Interleukin‐17A–Expressing, Group 3 Innate Lymphoid Cells in the Inflamed Joints of Patients With Spondyloarthritis

Arthritis & rheumatology (Hoboken, N.J.), 2019-03, Vol.71 (3), p.392-402

2019

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IgG Subclasses Shape Cytokine Responses by Human Myeloid Immune Cells through Differential Metabolic Reprogramming

The Journal of immunology (1950), 2020-12, Vol.205 (12), p.3400-3407

2020

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Interleukin‐17A Is Produced by CD4+ but Not CD8+ T Cells in Synovial Fluid Following T Cell Receptor Activation and Regulates Different Inflammatory Mediators Compared to Tumor Necrosis Factor in a Model of Psoriatic Arthritis Synovitis

Arthritis & rheumatology (Hoboken, N.J.), 2020-08, Vol.72 (8), p.1303-1313

2020

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Soluble immune complexes shift the TLR-induced cytokine production of distinct polarized human macrophage subsets towards IL-10

Anti-citrullinated protein antibody response after primary EBV infection in kidney transplant patients

Sustained remission with methotrexate monotherapy after 22-week induction treatment with TNF-alpha inhibitor and methotrexate in early psoriatic arthritis: an open-label extension of a randomized placebo-controlled trial

APRIL Induces a Novel Subset of IgA + Regulatory B Cells That Suppress Inflammation via Expression of IL-10 and PD-L1

Fc gamma receptor-TLR cross-talk elicits pro-inflammatory cytokine production by human M2 macrophages

Control of cytokine production by human fc gamma receptors: implications for pathogen defense and autoimmunity

Intimal lining layer macrophages but not synovial sublining macrophages display an IL-10 polarized-like phenotype in chronic synovitis

C-Reactive Protein Controls IL-23 Production by Human Monocytes

Anti-IL-17A treatment reduces serum inflammatory, angiogenic and tissue remodeling biomarkers accompanied by less synovial high endothelial venules in peripheral spondyloarthritis

The inflammatory function of human IgA

FcαRI co-stimulation converts human intestinal CD103+ dendritic cells into pro-inflammatory cells through glycolytic reprogramming

IL-12p40/IL-23p40 Blockade With Ustekinumab Decreases the Synovial Inflammatory Infiltrate Through Modulation of Multiple Signaling Pathways Including MAPK-ERK and Wnt

FcγR-TLR Cross-Talk Enhances TNF Production by Human Monocyte-Derived DCs via IRF5-Dependent Gene Transcription and Glycolytic Reprogramming

Fc gamma receptor IIa suppresses type I and III interferon production by human myeloid immune cells

Serum IgA Immune Complexes Promote Proinflammatory Cytokine Production by Human Macrophages, Monocytes, and Kupffer Cells through FcαRI-TLR Cross-Talk

Residual disease activity and treatment adjustments in psoriatic arthritis in current clinical practice

The Transcriptional Coactivator Bob1 Is Associated With Pathologic B Cell Responses in Autoimmune Tissue Inflammation

The novel cytokine interleukin-36α is expressed in psoriatic and rheumatoid arthritis synovium

The IL-17A-Producing CD8+ T-Cell Population in Psoriatic Lesional Skin Comprises Mucosa-Associated Invariant T Cells and Conventional T Cells

How Cytokine Networks Fuel Inflammation: Interleukin-17 and a tale of two autoimmune diseases

C-Reactive Protein Promotes Inflammation through FcγR-Induced Glycolytic Reprogramming of Human Macrophages

Transmembrane TNF drives osteoproliferative joint inflammation reminiscent of human spondyloarthritis

Expansion of Interleukin‐22– and Granulocyte–Macrophage Colony‐Stimulating Factor–Expressing, but Not Interleukin‐17A–Expressing, Group 3 Innate Lymphoid Cells in the Inflamed Joints of Patients With Spondyloarthritis

IgG Subclasses Shape Cytokine Responses by Human Myeloid Immune Cells through Differential Metabolic Reprogramming

Interleukin‐17A Is Produced by CD4+ but Not CD8+ T Cells in Synovial Fluid Following T Cell Receptor Activation and Regulates Different Inflammatory Mediators Compared to Tumor Necrosis Factor in a Model of Psoriatic Arthritis Synovitis

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