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Inflammation-induced DNA damage and damage-induced inflammation: a vicious cycle

Microbes and infection, 2014-10, Vol.16 (10), p.822-832

2014

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Rmi1 stimulates decatenation of double Holliday junctions during dissolution by Sgs1-Top3

Nature structural & molecular biology, 2010-11, Vol.17 (11), p.1377-1382

2010

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RecQ helicases: guardian angels of the DNA replication fork

Chromosoma, 2008-06, Vol.117 (3), p.219-233

2008

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RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability

Genes & development, 2008-10, Vol.22 (20), p.2843-2855

2008

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BLAP75/RMI1 Promotes the BLM-Dependent Dissolution of Homologous Recombination Intermediates

Proceedings of the National Academy of Sciences - PNAS, 2006-03, Vol.103 (11), p.4068-4073

2006

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SETD2-Dependent Histone H3K36 Trimethylation Is Required for Homologous Recombination Repair and Genome Stability

Cell reports (Cambridge), 2014-06, Vol.7 (6), p.2006-2018

2014

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Targeting Ultrasmall Gold Nanoparticles with cRGD Peptide Increases the Uptake and Efficacy of Cytotoxic Payload

Nanomaterials (Basel, Switzerland), 2022-11, Vol.12 (22), p.4013

2022

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BLM and RMI1 alleviate RPA inhibition of TopoIIIα decatenase activity

PloS one, 2012-07, Vol.7 (7), p.e41208-e41208

2012

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ATRX dysfunction induces replication defects in primary mouse cells

PloS one, 2014-03, Vol.9 (3), p.e92915-e92915

2014

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The Human RecQ Helicases, BLM and RECQ1, Display Distinct DNA Substrate Specificities

The Journal of biological chemistry, 2008-06, Vol.283 (26), p.17766-17776

2008

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Mobile D-loops are a preferred substrate for the Bloom's syndrome helicase

Nucleic acids research, 2006-01, Vol.34 (8), p.2269-2279

2006

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Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II binding

Chemical science (Cambridge), 2017-12, Vol.8 (12), p.8183-8192

2017

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RecQ helicases: suppressors of tumorigenesis and premature aging

Biochemical journal, 2003-09, Vol.374 (Pt 3), p.577-606

2003

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Human Topoisomerase IIIα Is a Single-stranded DNA Decatenase That Is Stimulated by BLM and RMI1

The Journal of biological chemistry, 2010-07, Vol.285 (28), p.21426-21436

2010

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The Bloom's syndrome helicase (BLM) interacts physically and functionally with p12, the smallest subunit of human DNA polymerase delta

Nucleic acids research, 2008-09, Vol.36 (16), p.5166-5179

2008

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Analysis of the DNA Unwinding Activity of RecQ Family Helicases

Methods in Enzymology, 2006, Vol.409, p.86-100

2006

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A simple and efficient method for PCR amplifiable DNA extraction from ancient bones

Nucleic acids research, 2000-06, Vol.28 (12), p.E67-67

2000

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A Small Molecule Inhibitor of the BLM Helicase Modulates Chromosome Stability in Human Cells

Chemistry & biology, 2013-01, Vol.20 (1), p.55-62

2013

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Physical and Functional Interaction between the Bloom's Syndrome Gene Product and the Largest Subunit of Chromatin Assembly Factor 1

Molecular and Cellular Biology, 2004-06, Vol.24 (11), p.4710-4719

2004

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Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II bindingElectronic supplementary information (ESI) available: Peptide synthesis, HPLC, LC-MS analysis, NMR spectra, microbiological data (MIC, MBC, time kill kinetics), lipid II and geranyl pyrophosphate binding, cytotoxicity assay. See DOI: 10.1039/c7sc03241b

2017-11

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Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II binding† †Electronic supplementary information (ESI) available: Peptide synthesis, HPLC, LC-MS analysis, NMR spectra, microbiological data (MIC, MBC, time kill kinetics), lipid II and geranyl pyrophosphate binding, cytotoxicity assay. See DOI: 10.1039/c7sc03241b

Chemical science (Cambridge), 2017-10, Vol.8 (12), p.8183-8192

2017

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BLM and RMI1 Alleviate RPA Inhibition of TopoIII[alpha] Decatenase Activity

PloS one, 2012-07, Vol.7 (7), p.e41208

2012

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Human Topoisomerase IIIa Is a Single-stranded DNA Decatenase That Is Stimulated by BLM and RMI1

The Journal of biological chemistry, 2010-07, Vol.285 (28), p.21426-21436

2010

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The Bloom's Syndrome Helicase Interacts Directly with the Human DNA Mismatch Repair Protein hMSH6

Biological chemistry, 2003-08, Vol.384 (8), p.1155-1164

2003

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Dissolution of double Holliday junctions by the concerted action of BLM and topoisomerase IIIalpha

Methods in molecular biology (Clifton, N.J.), 2009, Vol.582, p.91

2009

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Inflammation-induced DNA damage and damage-induced inflammation: a vicious cycle

Rmi1 stimulates decatenation of double Holliday junctions during dissolution by Sgs1-Top3

RecQ helicases: guardian angels of the DNA replication fork

RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability

BLAP75/RMI1 Promotes the BLM-Dependent Dissolution of Homologous Recombination Intermediates

SETD2-Dependent Histone H3K36 Trimethylation Is Required for Homologous Recombination Repair and Genome Stability

Targeting Ultrasmall Gold Nanoparticles with cRGD Peptide Increases the Uptake and Efficacy of Cytotoxic Payload

BLM and RMI1 alleviate RPA inhibition of TopoIIIα decatenase activity

ATRX dysfunction induces replication defects in primary mouse cells

The Human RecQ Helicases, BLM and RECQ1, Display Distinct DNA Substrate Specificities

Mobile D-loops are a preferred substrate for the Bloom's syndrome helicase

Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II binding

RecQ helicases: suppressors of tumorigenesis and premature aging

Human Topoisomerase IIIα Is a Single-stranded DNA Decatenase That Is Stimulated by BLM and RMI1

The Bloom's syndrome helicase (BLM) interacts physically and functionally with p12, the smallest subunit of human DNA polymerase delta

Analysis of the DNA Unwinding Activity of RecQ Family Helicases

A simple and efficient method for PCR amplifiable DNA extraction from ancient bones

A Small Molecule Inhibitor of the BLM Helicase Modulates Chromosome Stability in Human Cells

Physical and Functional Interaction between the Bloom's Syndrome Gene Product and the Largest Subunit of Chromatin Assembly Factor 1

BLM and RMI1 Alleviate RPA Inhibition of TopoIII[alpha] Decatenase Activity

Human Topoisomerase IIIa Is a Single-stranded DNA Decatenase That Is Stimulated by BLM and RMI1

The Bloom's Syndrome Helicase Interacts Directly with the Human DNA Mismatch Repair Protein hMSH6

Dissolution of double Holliday junctions by the concerted action of BLM and topoisomerase IIIalpha

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