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SETD2-Dependent Histone H3K36 Trimethylation Is Required for Homologous Recombination Repair and Genome Stability

Cell reports (Cambridge), 2014-06, Vol.7 (6), p.2006-2018

2014

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ATRX dysfunction induces replication defects in primary mouse cells

PloS one, 2014-03, Vol.9 (3), p.e92915-e92915

2014

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Mobile D-loops are a preferred substrate for the Bloom's syndrome helicase

Nucleic acids research, 2006-01, Vol.34 (8), p.2269

2006

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The Human RecQ Helicases, BLM and RECQ1, Display Distinct DNA Substrate Specificities

The Journal of biological chemistry, 2008-06, Vol.283 (26), p.17766-17776

2008

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RecQ helicases: suppressors of tumorigenesis and premature aging

Biochemical journal, 2003-09, Vol.374 (Pt 3), p.577-606

2003

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Analysis of the DNA Unwinding Activity of RecQ Family Helicases

Methods in Enzymology, 2006, Vol.409, p.86-100

2006

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Inflammation-induced DNA damage and damage-induced inflammation: a vicious cycle

Microbes and infection, 2014-10, Vol.16 (10), p.822-832

2014

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The Bloom's syndrome helicase (BLM) interacts physically and functionally with p12, the smallest subunit of human DNA polymerase delta

Nucleic acids research, 2008-09, Vol.36 (16), p.5166

2008

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RecQ helicases: guardian angels of the DNA replication fork

Chromosoma, 2008-06, Vol.117 (3), p.219-233

2008

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Rmi1 stimulates decatenation of double Holliday junctions during dissolution by Sgs1-Top3

Nature structural & molecular biology, 2010-11, Vol.17 (11), p.1377

2010

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A simple and efficient method for PCR amplifiable DNA extraction from ancient bones

Nucleic acids research, 2000-06, Vol.28 (12), p.E67

2000

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Physical and Functional Interaction between the Bloom's Syndrome Gene Product and the Largest Subunit of Chromatin Assembly Factor 1

Molecular and Cellular Biology, 2004-06, Vol.24 (11), p.4710-4719

2004

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A Small Molecule Inhibitor of the BLM Helicase Modulates Chromosome Stability in Human Cells

Chemistry & biology, 2013-01, Vol.20 (1), p.55-62

2013

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RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability

Genes & development, 2008-10, Vol.22 (20), p.2843-2855

2008

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BLAP75/RMI1 Promotes the BLM-Dependent Dissolution of Homologous Recombination Intermediates

Proceedings of the National Academy of Sciences - PNAS, 2006-03, Vol.103 (11), p.4068-4073

2006

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Targeting Ultrasmall Gold Nanoparticles with cRGD Peptide Increases the Uptake and Efficacy of Cytotoxic Payload

Nanomaterials (Basel, Switzerland), 2022-11, Vol.12 (22), p.4013

2022

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BLM and RMI1 alleviate RPA inhibition of TopoIIIα decatenase activity

PloS one, 2012-07, Vol.7 (7), p.e41208-e41208

2012

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Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II binding

Chemical science (Cambridge), 2017-12, Vol.8 (12), p.8183-8192

2017

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The Bloom's Syndrome Helicase Interacts Directly with the Human DNA Mismatch Repair Protein hMSH6

Biological chemistry, 2003-08, Vol.384 (8), p.1155-1164

2003

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Dissolution of double Holliday junctions by the concerted action of BLM and topoisomerase IIIalpha

Methods in molecular biology (Clifton, N.J.), 2009, Vol.582, p.91

2009

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Human Topoisomerase IIIα Is a Single-stranded DNA Decatenase That Is Stimulated by BLM and RMI1

The Journal of biological chemistry, 2010-07, Vol.285 (28), p.21426-21436

2010

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Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II bindingElectronic supplementary information (ESI) available: Peptide synthesis, HPLC, LC-MS analysis, NMR spectra, microbiological data (MIC, MBC, time kill kinetics), lipid II and geranyl pyrophosphate binding, cytotoxicity assay. See DOI: 10.1039/c7sc03241b

2017-11

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Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II binding† †Electronic supplementary information (ESI) available: Peptide synthesis, HPLC, LC-MS analysis, NMR spectra, microbiological data (MIC, MBC, time kill kinetics), lipid II and geranyl pyrophosphate binding, cytotoxicity assay. See DOI: 10.1039/c7sc03241b

Chemical science (Cambridge), 2017-10, Vol.8 (12), p.8183-8192

2017

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BLM and RMI1 Alleviate RPA Inhibition of TopoIII[alpha] Decatenase Activity

PloS one, 2012-07, Vol.7 (7), p.e41208

2012

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Dear editor

The Laryngoscope, 1999-06, Vol.109 (6), p.1011-1013

1999

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SETD2-Dependent Histone H3K36 Trimethylation Is Required for Homologous Recombination Repair and Genome Stability

ATRX dysfunction induces replication defects in primary mouse cells

Mobile D-loops are a preferred substrate for the Bloom's syndrome helicase

The Human RecQ Helicases, BLM and RECQ1, Display Distinct DNA Substrate Specificities

RecQ helicases: suppressors of tumorigenesis and premature aging

Analysis of the DNA Unwinding Activity of RecQ Family Helicases

Inflammation-induced DNA damage and damage-induced inflammation: a vicious cycle

The Bloom's syndrome helicase (BLM) interacts physically and functionally with p12, the smallest subunit of human DNA polymerase delta

RecQ helicases: guardian angels of the DNA replication fork

Rmi1 stimulates decatenation of double Holliday junctions during dissolution by Sgs1-Top3

A simple and efficient method for PCR amplifiable DNA extraction from ancient bones

Physical and Functional Interaction between the Bloom's Syndrome Gene Product and the Largest Subunit of Chromatin Assembly Factor 1

A Small Molecule Inhibitor of the BLM Helicase Modulates Chromosome Stability in Human Cells

RMI, a new OB-fold complex essential for Bloom syndrome protein to maintain genome stability

BLAP75/RMI1 Promotes the BLM-Dependent Dissolution of Homologous Recombination Intermediates

Targeting Ultrasmall Gold Nanoparticles with cRGD Peptide Increases the Uptake and Efficacy of Cytotoxic Payload

BLM and RMI1 alleviate RPA inhibition of TopoIIIα decatenase activity

Teixobactin analogues reveal enduracididine to be non-essential for highly potent antibacterial activity and lipid II binding

The Bloom's Syndrome Helicase Interacts Directly with the Human DNA Mismatch Repair Protein hMSH6

Dissolution of double Holliday junctions by the concerted action of BLM and topoisomerase IIIalpha

Human Topoisomerase IIIα Is a Single-stranded DNA Decatenase That Is Stimulated by BLM and RMI1

BLM and RMI1 Alleviate RPA Inhibition of TopoIII[alpha] Decatenase Activity

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