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Alterations in tissue oxygen consumption and extraction after trauma and hemorrhagic shock

Critical care medicine, 2000-08, Vol.28 (8), p.2837-2842

2000

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Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxation

American journal of physiology. Heart and circulatory physiology, 1994-06, Vol.266 (6), p.H2535-H2541

1994

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Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage

American journal of physiology. Regulatory, integrative and comparative physiology, 1991-01, Vol.260 (1), p.167-R171

1991

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Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsis

American journal of physiology. Regulatory, integrative and comparative physiology, 1995-11, Vol.269 (5), p.988-R994

1995

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Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation

American journal of physiology. Heart and circulatory physiology, 1993-09, Vol.265 (3), p.H973-H979

1993

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Trauma-hemorrhage and resuscitation in the mouse: effects on cardiac output and organ blood flow

American journal of physiology. Heart and circulatory physiology, 1993-04, Vol.264 (4), p.H1166-H1173

1993

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Effects of nonanticoagulant heparin on cardiovascular and hepatocellular function after hemorrhagic shock

American journal of physiology. Heart and circulatory physiology, 1996-04, Vol.270 (4), p.H1294-H1302

1996

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Preheparinization improves organ function after hemorrhage and resuscitation

American journal of physiology. Regulatory, integrative and comparative physiology, 1990-09, Vol.259 (3), p.645-R650

1990

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The complex pattern of cytokines in sepsis : association between prostaglandins, cachectin, and interleukins

Annals of surgery, 1991-08, Vol.214 (2), p.141-148

1991

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Increase in hepatic blood flow during early sepsis is due to increased portal blood flow

American journal of physiology. Regulatory, integrative and comparative physiology, 1991-12, Vol.261 (6), p.1507-R1512

1991

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Up-regulation of a novel potent vasodilatory peptide adrenomedullin during polymicrobial sepsis

Shock (Augusta, Ga.), 1998-08, Vol.10 (2), p.118-122

1998

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ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitation

American journal of physiology. Heart and circulatory physiology, 1995-04, Vol.268 (4), p.H1390-H1396

1995

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Reduction in Vascular Responsiveness to Adrenomedullin during Sepsis

The Journal of surgical research, 1999-07, Vol.85 (1), p.59-65

1999

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Differential alterations in microvascular perfusion in various organs during early and late sepsis

American journal of physiology: Gastrointestinal and liver physiology, 1992-07, Vol.263 (1 Pt 1), p.G38-G43

1992

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Tumor necrosis factor-alpha produces hepatocellular dysfunction despite normal cardiac output and hepatic microcirculation

American journal of physiology: Gastrointestinal and liver physiology, 1993-07, Vol.265 (1 Pt 1), p.G126-G132

1993

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Measurement of hepatic blood flow after severe hemorrhage: lack of restoration despite adequate resuscitation

American journal of physiology: Gastrointestinal and liver physiology, 1992-01, Vol.262 (1 Pt 1), p.G92-G98

1992

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Pentoxifylline maintains vascular endothelial cell function during hyperdynamic and hypodynamic sepsis

Surgery, 1996-08, Vol.120 (2), p.367-373

1996

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Salutary effects of ATP-MgCl2 on the depressed endothelium-dependent relaxation during hyperdynamic sepsis

Critical care medicine, 1999-05, Vol.27 (5), p.959-964

1999

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Diltiazem restores cardiac output and improves renal function after hemorrhagic shock and crystalloid resuscitation

American journal of physiology. Heart and circulatory physiology, 1992-05, Vol.262 (5), p.H1435-H1440

1992

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Pentoxifylline restores cardiac output and tissue perfusion after trauma-hemorrhage and decreases susceptibility to sepsis

Surgery, 1993-08, Vol.114 (2), p.352

1993

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Is Gut the “Motor” for Producing Hepatocellular Dysfunction after Trauma and Hemorrhagic Shock?

The Journal of surgical research, 1998-02, Vol.74 (2), p.141-148

1998

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Chemically modified heparin improves hepatocellular function, cardiac output, and microcirculation after trauma-hemorrhage and resuscitation

Surgery, 1994-08, Vol.116 (2), p.169

1994

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Liver Endothelial Cell Dysfunction Occurs Early Following Hemorrhagic Shock and Persists Despite Crystalloid Resuscitation

The Journal of surgical research, 1996-06, Vol.63 (1), p.241-247

1996

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Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitation

American journal of physiology: Gastrointestinal and liver physiology, 1992-12, Vol.263 (6 Pt 1), p.G895-G900

1992

Volltextzugriff (PDF)

ATP-MgCl2 administration normalizes macrophage cAMP and beta-adrenergic receptors after hemorrhage and resuscitation

American journal of physiology: Gastrointestinal and liver physiology, 1994-07, Vol.267 (1 Pt 1), p.G52-G58

1994

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Alterations in tissue oxygen consumption and extraction after trauma and hemorrhagic shock

Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxation

Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage

Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsis

Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation

Trauma-hemorrhage and resuscitation in the mouse: effects on cardiac output and organ blood flow

Effects of nonanticoagulant heparin on cardiovascular and hepatocellular function after hemorrhagic shock

Preheparinization improves organ function after hemorrhage and resuscitation

The complex pattern of cytokines in sepsis : association between prostaglandins, cachectin, and interleukins

Increase in hepatic blood flow during early sepsis is due to increased portal blood flow

Up-regulation of a novel potent vasodilatory peptide adrenomedullin during polymicrobial sepsis

ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitation

Reduction in Vascular Responsiveness to Adrenomedullin during Sepsis

Differential alterations in microvascular perfusion in various organs during early and late sepsis

Tumor necrosis factor-alpha produces hepatocellular dysfunction despite normal cardiac output and hepatic microcirculation

Measurement of hepatic blood flow after severe hemorrhage: lack of restoration despite adequate resuscitation

Pentoxifylline maintains vascular endothelial cell function during hyperdynamic and hypodynamic sepsis

Salutary effects of ATP-MgCl2 on the depressed endothelium-dependent relaxation during hyperdynamic sepsis

Diltiazem restores cardiac output and improves renal function after hemorrhagic shock and crystalloid resuscitation

Pentoxifylline restores cardiac output and tissue perfusion after trauma-hemorrhage and decreases susceptibility to sepsis

Is Gut the “Motor” for Producing Hepatocellular Dysfunction after Trauma and Hemorrhagic Shock?

Chemically modified heparin improves hepatocellular function, cardiac output, and microcirculation after trauma-hemorrhage and resuscitation

Liver Endothelial Cell Dysfunction Occurs Early Following Hemorrhagic Shock and Persists Despite Crystalloid Resuscitation

Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitation

ATP-MgCl2 administration normalizes macrophage cAMP and beta-adrenergic receptors after hemorrhage and resuscitation

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