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AMP-Activated Protein Kinase Induces a p53-Dependent Metabolic Checkpoint

Molecular cell, 2005-04, Vol.18 (3), p.283-293

2005

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Akt stimulates aerobic glycolysis in cancer cells

Cancer research (Chicago, Ill.), 2004-06, Vol.64 (11), p.3892-3899

2004

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The glucose dependence of Akt-transformed cells can be reversed by pharmacologic activation of fatty acid β-oxidation

Oncogene, 2005-06, Vol.24 (26), p.4165-4173

2005

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Bnip3L is induced by p53 under hypoxia, and its knockdown promotes tumor growth

Cancer cell, 2004-12, Vol.6 (6), p.597-609

2004

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Systemic treatment with the antidiabetic drug metformin selectively impairs p53-deficient tumor cell growth

Cancer research (Chicago, Ill.), 2007-07, Vol.67 (14), p.6745-6752

2007

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Reduced expression of α5β1 integrin prevents spreading-dependent cell proliferation

Journal of cell science, 1999-12, Vol.112 (24), p.4663-4672

1999

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Distinct Effects of Mitogens and the Actin Cytoskeleton on CREB and Pocket Protein Phosphorylation Control the Extent and Timing of Cyclin A Promoter Activity

Molecular and Cellular Biology, 2001-11, Vol.21 (22), p.7607-7616

2001

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New molecular concepts and targets in acute myeloid leukemia

Current opinion in hematology, 2008-03, Vol.15 (2), p.82-87

2008

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Transcriptional Profiling of Polycythemia Vera Identifies Gene Expression Patterns Both Dependent and Independent from the Action of JAK2V617F

Clinical cancer research, 2010-09, Vol.16 (17), p.4339-4352

2010

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Specific MicroRNA Deregulated in Myleoproliferative Neoplasm Are Regulated by JAK2V617F and May Contribute to Aberrant Hematopoiesis

Blood, 2009-11, Vol.114 (22), p.965-965

2009

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NFIB: A Gene Consistently Upregulated in MPD and Frequently Co-Amplified with JAK2V617F Augments Growth of Erythropoietin Dependent Cells

Blood, 2008-11, Vol.112 (11), p.3727-3727

2008

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Integrative Analysis of Genomic Aberration and Gene Expression Suggests That MPD with Loss of Heterozygosity or Gain of Chromosome 9 Represents a Distinct Entity

Blood, 2008-11, Vol.112 (11), p.3735-3735

2008

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The role of AMPK in modulating the metabolism and survival of cancer cells

Buzzai, Monica

2007

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The glucose dependence of Akt-transformed cells can be reversed by pharmacologic activation of fatty acid -oxidation

Oncogene, 2005-06, Vol.24 (26), p.4165-4173

2005

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An AMPK-p53 Connection Mediates a Checkpoint to Inhibit Cell Proliferation in Response to Low Glucose

Cancer biology & therapy, 2005-06, Vol.4 (6), p.618-618

2005

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cDNA Characterization and Chromosome Mapping of the Human GAS2 Gene

Genomics (San Diego, Calif.), 1998-03, Vol.48 (2), p.265-269

1998

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AMP-Activated Protein Kinase Induces a p53-Dependent Metabolic Checkpoint

Akt stimulates aerobic glycolysis in cancer cells

The glucose dependence of Akt-transformed cells can be reversed by pharmacologic activation of fatty acid β-oxidation

Bnip3L is induced by p53 under hypoxia, and its knockdown promotes tumor growth

Systemic treatment with the antidiabetic drug metformin selectively impairs p53-deficient tumor cell growth

Reduced expression of α5β1 integrin prevents spreading-dependent cell proliferation

Distinct Effects of Mitogens and the Actin Cytoskeleton on CREB and Pocket Protein Phosphorylation Control the Extent and Timing of Cyclin A Promoter Activity

New molecular concepts and targets in acute myeloid leukemia

Transcriptional Profiling of Polycythemia Vera Identifies Gene Expression Patterns Both Dependent and Independent from the Action of JAK2V617F

Specific MicroRNA Deregulated in Myleoproliferative Neoplasm Are Regulated by JAK2V617F and May Contribute to Aberrant Hematopoiesis

NFIB: A Gene Consistently Upregulated in MPD and Frequently Co-Amplified with JAK2V617F Augments Growth of Erythropoietin Dependent Cells

Integrative Analysis of Genomic Aberration and Gene Expression Suggests That MPD with Loss of Heterozygosity or Gain of Chromosome 9 Represents a Distinct Entity

The role of AMPK in modulating the metabolism and survival of cancer cells

The glucose dependence of Akt-transformed cells can be reversed by pharmacologic activation of fatty acid -oxidation

An AMPK-p53 Connection Mediates a Checkpoint to Inhibit Cell Proliferation in Response to Low Glucose

cDNA Characterization and Chromosome Mapping of the Human GAS2 Gene

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