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Islet Amyloid in Type 2 Diabetes, and the Toxic Oligomer Hypothesis

Endocrine reviews, 2008-05, Vol.29 (3), p.303-316

2008

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β-Cell Replication Is the Primary Mechanism Subserving the Postnatal Expansion of β-Cell Mass in Humans

Diabetes (New York, N.Y.), 2008-06, Vol.57 (6), p.1584-1594

2008

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Insulin-Degrading Enzyme Inhibition, a Novel Therapy for Type 2 Diabetes?

Cell metabolism, 2014-08, Vol.20 (2), p.201-203

2014

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IAPP toxicity activates HIF1α/PFKFB3 signaling delaying β-cell loss at the expense of β-cell function

Nature communications, 2019-06, Vol.10 (1), p.2679-17, Article 2679

2019

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Reversing type 1 diabetes with stem cell-derived islets: a step closer to the dream?

The Journal of clinical investigation, 2022-02, Vol.132 (3)

2022

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β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes

Diabetes (New York, N.Y.), 2003-01, Vol.52 (1), p.102-110

2003

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High Expression Rates of Human Islet Amyloid Polypeptide Induce Endoplasmic Reticulum Stress–Mediated β-Cell Apoptosis, a Characteristic of Humans With Type 2 but Not Type 1 Diabetes

Diabetes (New York, N.Y.), 2007-08, Vol.56 (8), p.2016-2027

2007

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Increased Risk of Type 2 Diabetes in Alzheimer Disease

Diabetes (New York, N.Y.), 2004-02, Vol.53 (2), p.474-481

2004

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Increased Hormone-Negative Endocrine Cells in the Pancreas in Type 1 Diabetes

The journal of clinical endocrinology and metabolism, 2016-09, Vol.101 (9), p.3487-3496

2016

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Rb and p130 control cell cycle gene silencing to maintain the postmitotic phenotype in cardiac myocytes

The Journal of cell biology, 2011-08, Vol.194 (3), p.407-423

2011

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Increased Proliferation of the Pancreatic Duct Gland Compartment in Type 1 Diabetes

The journal of clinical endocrinology and metabolism, 2017-01, Vol.102 (1), p.200-209

2017

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Hyperinsulinemic Hypoglycemia After Gastric Bypass Surgery Is Not Accompanied by Islet Hyperplasia or Increased β-Cell Turnover

Diabetes care, 2006-07, Vol.29 (7), p.1554-1559

2006

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Pulsatile Portal Vein Insulin Delivery Enhances Hepatic Insulin Action and Signaling

Diabetes (New York, N.Y.), 2012-09, Vol.61 (9), p.2269-2279

2012

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β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes

Diabetes (New York, N.Y.), 2006-07, Vol.55 (7), p.2106-2114

2006

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Glucagonlike Peptide 1-based drugs and pancreatitis: clarity at last, but what about pancreatic cancer?

JAMA internal medicine, 2013-04, Vol.173 (7), p.539

2013

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Toxic Human Islet Amyloid Polypeptide (h-IAPP) Oligomers Are Intracellular, and Vaccination to Induce Anti-Toxic Oligomer Antibodies Does Not Prevent h-IAPP–Induced β-Cell Apoptosis in h-IAPP Transgenic Mice

Diabetes (New York, N.Y.), 2007-05, Vol.56 (5), p.1324-1332

2007

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Type 2 diabetes as a protein misfolding disease

Trends in molecular medicine, 2015-07, Vol.21 (7), p.439-449

2015

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Pulsatile Insulin Secretion Dictates Systemic Insulin Delivery by Regulating Hepatic Insulin Extraction In Humans

Diabetes (New York, N.Y.), 2005-06, Vol.54 (6), p.1649-1656

2005

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Calcium-activated Calpain-2 Is a Mediator of Beta Cell Dysfunction and Apoptosis in Type 2 Diabetes

The Journal of biological chemistry, 2010-01, Vol.285 (1), p.339-348

2010

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Supplying Insulin while Evading Immunity

The New England journal of medicine, 2021-03, Vol.384 (10), p.967-969

2021

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Pancreatitis, Pancreatic, and Thyroid Cancer With Glucagon-Like Peptide-1–Based Therapies

Gastroenterology (New York, N.Y. 1943), 2011-07, Vol.141 (1), p.150-156

2011

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Human Islet Amyloid Polypeptide Oligomers Disrupt Cell Coupling, Induce Apoptosis, and Impair Insulin Secretion in Isolated Human Islets

Diabetes (New York, N.Y.), 2007-01, Vol.56 (1), p.65-71

2007

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Publisher Correction: IAPP toxicity activates HIF1α/PFKFB3 signaling delaying β-cell loss at the expense of β-cell function

Nature communications, 2019-07, Vol.10 (1), p.3507-1, Article 3507

2019

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β-Cell Mass and Turnover in Humans: Effects of obesity and aging

Diabetes care, 2013-01, Vol.36 (1), p.111-117

2013

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A critical analysis of the clinical use of incretin-based therapies: Are the GLP-1 therapies safe?

Diabetes care, 2013-07, Vol.36 (7), p.2118-2125

2013

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Islet Amyloid in Type 2 Diabetes, and the Toxic Oligomer Hypothesis

β-Cell Replication Is the Primary Mechanism Subserving the Postnatal Expansion of β-Cell Mass in Humans

Insulin-Degrading Enzyme Inhibition, a Novel Therapy for Type 2 Diabetes?

IAPP toxicity activates HIF1α/PFKFB3 signaling delaying β-cell loss at the expense of β-cell function

Reversing type 1 diabetes with stem cell-derived islets: a step closer to the dream?

β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes

High Expression Rates of Human Islet Amyloid Polypeptide Induce Endoplasmic Reticulum Stress–Mediated β-Cell Apoptosis, a Characteristic of Humans With Type 2 but Not Type 1 Diabetes

Increased Risk of Type 2 Diabetes in Alzheimer Disease

Increased Hormone-Negative Endocrine Cells in the Pancreas in Type 1 Diabetes

Rb and p130 control cell cycle gene silencing to maintain the postmitotic phenotype in cardiac myocytes

Increased Proliferation of the Pancreatic Duct Gland Compartment in Type 1 Diabetes

Hyperinsulinemic Hypoglycemia After Gastric Bypass Surgery Is Not Accompanied by Islet Hyperplasia or Increased β-Cell Turnover

Pulsatile Portal Vein Insulin Delivery Enhances Hepatic Insulin Action and Signaling

β-Cell Deficit Due to Increased Apoptosis in the Human Islet Amyloid Polypeptide Transgenic (HIP) Rat Recapitulates the Metabolic Defects Present in Type 2 Diabetes

Glucagonlike Peptide 1-based drugs and pancreatitis: clarity at last, but what about pancreatic cancer?

Toxic Human Islet Amyloid Polypeptide (h-IAPP) Oligomers Are Intracellular, and Vaccination to Induce Anti-Toxic Oligomer Antibodies Does Not Prevent h-IAPP–Induced β-Cell Apoptosis in h-IAPP Transgenic Mice

Type 2 diabetes as a protein misfolding disease

Pulsatile Insulin Secretion Dictates Systemic Insulin Delivery by Regulating Hepatic Insulin Extraction In Humans

Calcium-activated Calpain-2 Is a Mediator of Beta Cell Dysfunction and Apoptosis in Type 2 Diabetes

Supplying Insulin while Evading Immunity

Pancreatitis, Pancreatic, and Thyroid Cancer With Glucagon-Like Peptide-1–Based Therapies

Human Islet Amyloid Polypeptide Oligomers Disrupt Cell Coupling, Induce Apoptosis, and Impair Insulin Secretion in Isolated Human Islets

Publisher Correction: IAPP toxicity activates HIF1α/PFKFB3 signaling delaying β-cell loss at the expense of β-cell function

β-Cell Mass and Turnover in Humans: Effects of obesity and aging

A critical analysis of the clinical use of incretin-based therapies: Are the GLP-1 therapies safe?

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