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Delayed Wound Healing in Keratin 6a Knockout Mice

Molecular and Cellular Biology, 2000-07, Vol.20 (14), p.5248-5255

2000

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Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophy

Human molecular genetics, 2005-06, Vol.14 (11), p.1539-1547

2005

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The mouse keratin 6 isoforms are differentially expressed in the hair follicle, footpad, tongue and activated epidermis

Differentiation (London), 1999-10, Vol.65 (2), p.119-130

1999

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Redundant Pathways for Negative Feedback Regulation of Bile Acid Production

Developmental cell, 2002-06, Vol.2 (6), p.721-731

2002

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Targeting expression of a dominant-negative retinoic acid receptor mutant in the epidermis of transgenic mice results in loss of barrier function

Genes & development, 1995-02, Vol.9 (3), p.317-329

1995

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Expression of a Dominant-Negative Type II Transforming Growth Factor β (TGF-β ) Receptor in the Epidermis of Transgenic Mice Blocks TGF-β -Mediated Growth Inhibition

Proceedings of the National Academy of Sciences - PNAS, 1997-03, Vol.94 (6), p.2386-2391

1997

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Inhibition of skin development by overexpression of transforming growth factor beta 1 in the epidermis of transgenic mice

Proceedings of the National Academy of Sciences - PNAS, 1993-06, Vol.90 (11), p.5237-5241

1993

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The Proximal Promoter of the Mouse Loricrin Gene Contains a Functional AP-1 Element and Directs Keratinocyte-specific but Not Differentiation-specific Expression

The Journal of biological chemistry, 1995-05, Vol.270 (18), p.10792-10799

1995

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Loricrin Expression Is Coordinated with Other Epidermal Proteins and the Appearance of Lipid Lamellar Granules in Development

Journal of investigative dermatology, 1995-03, Vol.104 (3), p.405-410

1995

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Inhibition of Melanoma Growth by Adenoviral-Mediated HSV Thymidine Kinase Gene Transfer In Vivo

Journal of investigative dermatology, 1995-03, Vol.104 (3), p.313-317

1995

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Induction of epidermal hyperplasia, hyperkeratosis, and papillomas in transgenic mice by a targeted v-Ha-ras oncogene

Molecular carcinogenesis, 1993, Vol.7 (2), p.99-110

1993

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Characterization of loricrin regulation in vitro and in transgenic mice

Differentiation (London), 1999-05, Vol.64 (4), p.225-235

1999

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Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle

Differentiation (London), 1999-10, Vol.65 (2), p.97-112

1999

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Lessons from Loricrin-Deficient Mice: Compensatory Mechanisms Maintaining Skin Barrier Function in the Absence of a Major Cornified Envelope Protein

The Journal of cell biology, 2000-10, Vol.151 (2), p.389-400

2000

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Hyperplasia, hyperkeratosis and benign tumor production in transgenic mice by a targeted v-fos oncogene suggest a role for fos in epidermal differentiation and neoplasia

Oncogene, 1993-08, Vol.8 (8), p.2145-2157

1993

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Proteolysis of specific porcine zona pellucida glycoproteins by boar acrosin

Biology of reproduction, 1985-04, Vol.32 (3), p.619-630

1985

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Transgenic Mice Expressing a Mutant Form of Loricrin Reveal the Molecular Basis of the Skin Diseases, Vohwinkel Syndrome and Progressive Symmetric Erythrokeratoderma

The Journal of cell biology, 2000-10, Vol.151 (2), p.401-412

2000

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Adenoviral-Mediated Herpes Simplex Virus-Thymidine Kinase Gene Transfer in Vivo for Treatment of Experimental Human Melanoma

Journal of investigative dermatology, 1996-06, Vol.106 (6), p.1163-1168

1996

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A transgenic mouse model that recapitulates the clinical features of both neonatal and adult forms of the skin disease epidermolytic hyperkeratosis

Differentiation (London), 1996-12, Vol.61 (2), p.129-139

1996

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Use of specific monoclonal and polyclonal antibodies to define distinct antigens of the porcine zonae pellucidae

Biology of reproduction, 1987-06, Vol.36 (5), p.1275-1287

1987

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Quasi-Normal Cornified Cell Envelopes in Loricrin Knockout Mice Imply the Existence of a Loricrin Backup System

Journal of investigative dermatology, 2002-01, Vol.118 (1), p.102-109

2002

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Formation of the rabbit zona pellucida and its relationship to ovarian follicular development

Developmental biology, 1984-11, Vol.106 (1), p.1,11-9,14

1984

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Evidence for a role of the major glycoprotein in the structural maintenance of the pig zona pellucida

Journal of reproduction & fertility, 1987-11, Vol.81 (2), p.363-376

1987

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Human keratin-1.bcl-2 transgenic mice aberrantly express keratin 6, exhibit reduced sensitivity to keratinocyte cell death induction, and are susceptible to skin tumor formation

Oncogene, 1998-02, Vol.16 (7), p.853-863

1998

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Inhibition of Skin Development by Overexpression of Transforming Growth Factor β1in the Epidermis of Transgenic Mice

Proceedings of the National Academy of Sciences - PNAS, 1993-06, Vol.90 (11), p.5237-5241

1993

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Delayed Wound Healing in Keratin 6a Knockout Mice

Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophy

The mouse keratin 6 isoforms are differentially expressed in the hair follicle, footpad, tongue and activated epidermis

Redundant Pathways for Negative Feedback Regulation of Bile Acid Production

Targeting expression of a dominant-negative retinoic acid receptor mutant in the epidermis of transgenic mice results in loss of barrier function

Expression of a Dominant-Negative Type II Transforming Growth Factor β (TGF-β ) Receptor in the Epidermis of Transgenic Mice Blocks TGF-β -Mediated Growth Inhibition

Inhibition of skin development by overexpression of transforming growth factor beta 1 in the epidermis of transgenic mice

The Proximal Promoter of the Mouse Loricrin Gene Contains a Functional AP-1 Element and Directs Keratinocyte-specific but Not Differentiation-specific Expression

Loricrin Expression Is Coordinated with Other Epidermal Proteins and the Appearance of Lipid Lamellar Granules in Development

Inhibition of Melanoma Growth by Adenoviral-Mediated HSV Thymidine Kinase Gene Transfer In Vivo

Induction of epidermal hyperplasia, hyperkeratosis, and papillomas in transgenic mice by a targeted v-Ha-ras oncogene

Characterization of loricrin regulation in vitro and in transgenic mice

Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle

Lessons from Loricrin-Deficient Mice: Compensatory Mechanisms Maintaining Skin Barrier Function in the Absence of a Major Cornified Envelope Protein

Hyperplasia, hyperkeratosis and benign tumor production in transgenic mice by a targeted v-fos oncogene suggest a role for fos in epidermal differentiation and neoplasia

Proteolysis of specific porcine zona pellucida glycoproteins by boar acrosin

Transgenic Mice Expressing a Mutant Form of Loricrin Reveal the Molecular Basis of the Skin Diseases, Vohwinkel Syndrome and Progressive Symmetric Erythrokeratoderma

Adenoviral-Mediated Herpes Simplex Virus-Thymidine Kinase Gene Transfer in Vivo for Treatment of Experimental Human Melanoma

A transgenic mouse model that recapitulates the clinical features of both neonatal and adult forms of the skin disease epidermolytic hyperkeratosis

Use of specific monoclonal and polyclonal antibodies to define distinct antigens of the porcine zonae pellucidae

Quasi-Normal Cornified Cell Envelopes in Loricrin Knockout Mice Imply the Existence of a Loricrin Backup System

Formation of the rabbit zona pellucida and its relationship to ovarian follicular development

Evidence for a role of the major glycoprotein in the structural maintenance of the pig zona pellucida

Human keratin-1.bcl-2 transgenic mice aberrantly express keratin 6, exhibit reduced sensitivity to keratinocyte cell death induction, and are susceptible to skin tumor formation

Inhibition of Skin Development by Overexpression of Transforming Growth Factor β1in the Epidermis of Transgenic Mice

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