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Optical Control of Muscle Function by Transplantation of Stem Cell–Derived Motor Neurons in Mice

Science (American Association for the Advancement of Science), 2014-04, Vol.344 (6179), p.94-97

2014

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An optogenetic cell therapy to restore control of target muscles in an aggressive mouse model of amyotrophic lateral sclerosis

eLife, 2024-01, Vol.12

2024

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Nidogens are therapeutic targets for the prevention of tetanus

Science (American Association for the Advancement of Science), 2014-11, Vol.346 (6213), p.1118-1123

2014

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The cholesterol depleting agent, (2-Hydroxypropyl)-ß-cyclodextrin, does not affect disease progression in SOD1G93A mice

Amyotrophic lateral sclerosis and frontotemporal degeneration, 2023-07, Vol.ahead-of-print (ahead-of-print), p.1-7

2023

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Restoring motor function using optogenetics and neural engraftment

Current opinion in biotechnology, 2016-08, Vol.40, p.75-81

2016

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Activated CREB Is Sufficient to Overcome Inhibitors in Myelin and Promote Spinal Axon Regeneration In Vivo

Neuron (Cambridge, Mass.), 2004-11, Vol.44 (4), p.609-621

2004

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Mutant glycyl-tRNA synthetase (Gars) ameliorates SOD1(G93A) motor neuron degeneration phenotype but has little affect on Loa dynein heavy chain mutant mice

PloS one, 2009-07, Vol.4 (7), p.e6218-e6218

2009

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The Cytokine Interleukin-6 Is Sufficient But Not Necessary to Mimic the Peripheral Conditioning Lesion Effect on Axonal Growth

The Journal of neuroscience, 2006-05, Vol.26 (20), p.5565-5573

2006

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Amyloid precursor protein (APP) contributes to pathology in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis

Human molecular genetics, 2012-09, Vol.21 (17), p.3871-3882

2012

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Increased Synthesis of Spermidine as a Result of Upregulation of Arginase I Promotes Axonal Regeneration in Culture and In Vivo

The Journal of neuroscience, 2009-07, Vol.29 (30), p.9545-9552

2009

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Ephrin‐B2 elicits differential growth cone collapse and axon retraction in retinal ganglion cells from distinct retinal regions

Developmental neurobiology (Hoboken, N.J.), 2010-09, Vol.70 (11), p.781-794

2010

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The cholesterol depleting agent, (2-Hydroxypropyl)-ß-cyclodextrin, does not affect disease progression in SOD1 G93A mice

Amyotrophic lateral sclerosis and frontotemporal degeneration, 2023-10, Vol.24 (7-8), p.756-762

2023

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The Inhibition Site on Myelin-Associated Glycoprotein Is within Ig-Domain 5 and Is Distinct from the Sialic Acid Binding Site

The Journal of neuroscience, 2007-08, Vol.27 (34), p.9146-9154

2007

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The cholesterol depleting agent, (2-Hydroxypropyl)-ß-cyclodextrin, does not affect disease progression in SOD1 G93A mice

Amyotrophic lateral sclerosis and frontotemporal degeneration, 2023-07, p.1

2023

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Amyloid precursor protein (APP) contributes to pathology in the SOD1G93A mouse model of amyotrophic lateral sclerosis

Human molecular genetics, 2012-09, Vol.21 (17), p.3871-3882

2012

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An optogenetic cell therapy to restore control of target muscles in an aggressive mouse model of amyotrophic lateral sclerosis

eLife, 2024-01, Vol.12

2024

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Mutant Glycyl-tRNA Synthetase (Gars) Ameliorates SOD1G93A Motor Neuron Degeneration Phenotype but Has Little Affect on Loa Dynein Heavy Chain Mutant Mice

PloS one, 2009-07, Vol.4 (7), p.e6218

2009

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A Large-Scale Chemical Screen for Regulators of the Arginase 1 Promoter Identifies the Soy Isoflavone Daidzeinas a Clinically Approved Small Molecule That Can Promote Neuronal Protection or Regeneration via a cAMP-Independent Pathway

The Journal of neuroscience, 2010-01, Vol.30 (2), p.739-748

2010

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An Integrated Bidirectional Multi-Channel Opto-Electro Arbitrary Waveform Stimulator for Treating Motor Neurone Disease

2021 IEEE International Symposium on Circuits and Systems (ISCAS), 2021, p.1-4

2021

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Amyloid precursor protein (APP) contributes to pathology in the SOD1 super(G93A) mouse model of amyotrophic lateral sclerosis

Human molecular genetics, 2012-09, Vol.21 (17), p.3871-3882

2012

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Correction: Mutant Glycyl-tRNA Synthetase (Gars) Ameliorates SOD1G93A Motor Neuron Degeneration Phenotype but Has Little Affect on Loa Dynein Heavy Chain Mutant Mice

PloS one, 2009-10, Vol.4 (10)

2009

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Mutant Glycyl-tRNA Synthetase Ameliorates SOD1.sup.G93A Motor Neuron Degeneration Phenotype but Has Little Affect on Loa Dynein Heavy Chain Mutant Mice

PloS one, 2009-07, Vol.4 (7), p.e6218

2009

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Mutant Glycyl-tRNA Synthetase

PloS one, 2009-07, Vol.4 (7), p.e6218

2009

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Tetanus toxin entry. Nidogens are therapeutic targets for the prevention of tetanus

Science (American Association for the Advancement of Science), 2014-11, Vol.346 (6213), p.1118

2014

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Investigating the Contribution of Amyloid Precursor Protein (APP) to Pathology in a Mouse Model of Amyotrophic Lateral Sclerosis (ALS)

Bryson, J Barney N. B

2011

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Optical Control of Muscle Function by Transplantation of Stem Cell–Derived Motor Neurons in Mice

An optogenetic cell therapy to restore control of target muscles in an aggressive mouse model of amyotrophic lateral sclerosis

Nidogens are therapeutic targets for the prevention of tetanus

The cholesterol depleting agent, (2-Hydroxypropyl)-ß-cyclodextrin, does not affect disease progression in SOD1G93A mice

Restoring motor function using optogenetics and neural engraftment

Activated CREB Is Sufficient to Overcome Inhibitors in Myelin and Promote Spinal Axon Regeneration In Vivo

Mutant glycyl-tRNA synthetase (Gars) ameliorates SOD1(G93A) motor neuron degeneration phenotype but has little affect on Loa dynein heavy chain mutant mice

The Cytokine Interleukin-6 Is Sufficient But Not Necessary to Mimic the Peripheral Conditioning Lesion Effect on Axonal Growth

Amyloid precursor protein (APP) contributes to pathology in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis

Increased Synthesis of Spermidine as a Result of Upregulation of Arginase I Promotes Axonal Regeneration in Culture and In Vivo

Ephrin‐B2 elicits differential growth cone collapse and axon retraction in retinal ganglion cells from distinct retinal regions

The cholesterol depleting agent, (2-Hydroxypropyl)-ß-cyclodextrin, does not affect disease progression in SOD1 G93A mice

The Inhibition Site on Myelin-Associated Glycoprotein Is within Ig-Domain 5 and Is Distinct from the Sialic Acid Binding Site

The cholesterol depleting agent, (2-Hydroxypropyl)-ß-cyclodextrin, does not affect disease progression in SOD1 G93A mice

Amyloid precursor protein (APP) contributes to pathology in the SOD1G93A mouse model of amyotrophic lateral sclerosis

An optogenetic cell therapy to restore control of target muscles in an aggressive mouse model of amyotrophic lateral sclerosis

Mutant Glycyl-tRNA Synthetase (Gars) Ameliorates SOD1G93A Motor Neuron Degeneration Phenotype but Has Little Affect on Loa Dynein Heavy Chain Mutant Mice

A Large-Scale Chemical Screen for Regulators of the Arginase 1 Promoter Identifies the Soy Isoflavone Daidzeinas a Clinically Approved Small Molecule That Can Promote Neuronal Protection or Regeneration via a cAMP-Independent Pathway

An Integrated Bidirectional Multi-Channel Opto-Electro Arbitrary Waveform Stimulator for Treating Motor Neurone Disease

Amyloid precursor protein (APP) contributes to pathology in the SOD1 super(G93A) mouse model of amyotrophic lateral sclerosis

Correction: Mutant Glycyl-tRNA Synthetase (Gars) Ameliorates SOD1G93A Motor Neuron Degeneration Phenotype but Has Little Affect on Loa Dynein Heavy Chain Mutant Mice

Mutant Glycyl-tRNA Synthetase Ameliorates SOD1.sup.G93A Motor Neuron Degeneration Phenotype but Has Little Affect on Loa Dynein Heavy Chain Mutant Mice

Mutant Glycyl-tRNA Synthetase

Tetanus toxin entry. Nidogens are therapeutic targets for the prevention of tetanus

Investigating the Contribution of Amyloid Precursor Protein (APP) to Pathology in a Mouse Model of Amyotrophic Lateral Sclerosis (ALS)

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