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Protein tyrosine phosphatases: molecular switches in metabolism and diabetes

Trends in endocrinology and metabolism, 2015-01, Vol.26 (1), p.30-39

2015

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Deficiency of the innate immune adaptor STING promotes autoreactive T cell expansion in NOD mice

Diabetologia, 2021-04, Vol.64 (4), p.878-889

2021

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Type I Interferon Signaling Is Required for Dacryoadenitis in the Nonobese Diabetic Mouse Model of Sjögren Syndrome

International journal of molecular sciences, 2018-10, Vol.19 (10), p.3259

2018

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A Role for lncRNAs in Regulating Inflammatory and Autoimmune Responses Underlying Type 1 Diabetes

Advances in experimental medicine and biology, 2022, Vol.1363, p.97-118

2022

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Granzyme A Deficiency Breaks Immune Tolerance and Promotes Autoimmune Diabetes Through a Type I Interferon-Dependent Pathway

Diabetes (New York, N.Y.), 2017-12, Vol.66 (12), p.3041-3050

2017

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Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice

PloS one, 2012-07, Vol.7 (7), p.e40357

2012

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Increased autoimmune diabetes in pIgR-deficient NOD mice is due to a "Hitchhiking" interval that refines the genetic effect of Idd5.4

PloS one, 2015-04, Vol.10 (4), p.e0121979-e0121979

2015

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GSK3 inhibition, but not epigenetic remodeling, mediates efficient derivation of germline embryonic stem cells from nonobese diabetic mice

Stem cell research, 2018-08, Vol.31, p.5-10

2018

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IL-1 Receptor Deficiency Slows Progression to Diabetes in the NOD Mouse

Diabetes (New York, N.Y.), 2004-01, Vol.53 (1), p.113-121

2004

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Altering β Cell Antigen Exposure to Exhausted CD8+ T Cells Prevents Autoimmune Diabetes in Mice

The Journal of immunology (1950), 2024-06, Vol.212 (11), p.1658-1669

2024

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Proinsulin-specific, HLA-DQ8, and HLA-DQ8-transdimer-restricted CD4+ T cells infiltrate islets in type 1 diabetes

Diabetes (New York, N.Y.), 2015-01, Vol.64 (1), p.172-182

2015

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Proapoptotic BH3-Only Bcl-2 Family Member Bik/Blk/Nbk Is Expressed in Hemopoietic and Endothelial Cells but Is Redundant for Their Programmed Death

Molecular and Cellular Biology, 2004-02, Vol.24 (4), p.1570-1581

2004

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T-Cell-Specific PTPN2 Deficiency in NOD Mice Accelerates the Development of Type 1 Diabetes and Autoimmune Comorbidities

Diabetes (New York, N.Y.), 2019-06, Vol.68 (6), p.1251-1266

2019

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Repurposed JAK1/JAK2 Inhibitor Reverses Established Autoimmune Insulitis in NOD Mice

Diabetes (New York, N.Y.), 2017-06, Vol.66 (6), p.1650-1660

2017

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Gut dysbiosis promotes islet-autoimmunity by increasing T-cell attraction in islets via CXCL10 chemokine

Journal of autoimmunity, 2023-11, Vol.140, p.103090-103090, Article 103090

2023

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Interferons limit autoantigen-specific CD8+ T-cell expansion in the non-obese diabetic mouse

Cell reports (Cambridge), 2022-04, Vol.39 (4), p.110747-110747, Article 110747

2022

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How to Make Mice Tell the Truth

Diabetes (New York, N.Y.), 2016-05, Vol.65 (5), p.1161-1163

2016

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Deficiency in Type I Interferon Signaling Prevents the Early Interferon-Induced Gene Signature in Pancreatic Islets but Not Type 1 Diabetes in NOD Mice

Diabetes (New York, N.Y.), 2014-03, Vol.63 (3), p.1032-1040

2014

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Fms-like tyrosine kinase 3 ligand administration overcomes a genetically determined dendritic cell deficiency in NOD mice and protects against diabetes development

International immunology, 2005-03, Vol.17 (3), p.307-314

2005

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Mind the gap: analysis of marker-assisted breeding strategies for inbred mouse strains

Mammalian genome, 2006-04, Vol.17 (4), p.273-287

2006

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Two genetic loci independently confer susceptibility to autoimmune gastritis

International immunology, 2007-09, Vol.19 (9), p.1135-1144

2007

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BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells

Diabetes (New York, N.Y.), 2015-09, Vol.64 (9), p.3229-3238

2015

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Disruption of Serinc1, which facilitates serine-derived lipid synthesis, fails to alter macrophage function, lymphocyte proliferation or autoimmune disease susceptibility

Molecular immunology, 2017-02, Vol.82, p.19-33

2017

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Autoimmune-Disease-Prone NOD Mice Help To Reveal a New Genetic Locus for Reducing Pulmonary Disease Caused by Mycoplasma pulmonis

Infection and immunity, 2018-03, Vol.86 (3)

2018

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A Susceptibility Allele From a Non-Diabetes-Prone Mouse Strain Accelerates Diabetes in NOD Congenic Mice

Diabetes (New York, N.Y.), 2003-01, Vol.52 (1), p.218-222

2003

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Protein tyrosine phosphatases: molecular switches in metabolism and diabetes

Deficiency of the innate immune adaptor STING promotes autoreactive T cell expansion in NOD mice

Type I Interferon Signaling Is Required for Dacryoadenitis in the Nonobese Diabetic Mouse Model of Sjögren Syndrome

A Role for lncRNAs in Regulating Inflammatory and Autoimmune Responses Underlying Type 1 Diabetes

Granzyme A Deficiency Breaks Immune Tolerance and Promotes Autoimmune Diabetes Through a Type I Interferon-Dependent Pathway

Granzyme B is dispensable in the development of diabetes in non-obese diabetic mice

Increased autoimmune diabetes in pIgR-deficient NOD mice is due to a "Hitchhiking" interval that refines the genetic effect of Idd5.4

GSK3 inhibition, but not epigenetic remodeling, mediates efficient derivation of germline embryonic stem cells from nonobese diabetic mice

IL-1 Receptor Deficiency Slows Progression to Diabetes in the NOD Mouse

Altering β Cell Antigen Exposure to Exhausted CD8+ T Cells Prevents Autoimmune Diabetes in Mice

Proinsulin-specific, HLA-DQ8, and HLA-DQ8-transdimer-restricted CD4+ T cells infiltrate islets in type 1 diabetes

Proapoptotic BH3-Only Bcl-2 Family Member Bik/Blk/Nbk Is Expressed in Hemopoietic and Endothelial Cells but Is Redundant for Their Programmed Death

T-Cell-Specific PTPN2 Deficiency in NOD Mice Accelerates the Development of Type 1 Diabetes and Autoimmune Comorbidities

Repurposed JAK1/JAK2 Inhibitor Reverses Established Autoimmune Insulitis in NOD Mice

Gut dysbiosis promotes islet-autoimmunity by increasing T-cell attraction in islets via CXCL10 chemokine

Interferons limit autoantigen-specific CD8+ T-cell expansion in the non-obese diabetic mouse

How to Make Mice Tell the Truth

Deficiency in Type I Interferon Signaling Prevents the Early Interferon-Induced Gene Signature in Pancreatic Islets but Not Type 1 Diabetes in NOD Mice

Fms-like tyrosine kinase 3 ligand administration overcomes a genetically determined dendritic cell deficiency in NOD mice and protects against diabetes development

Mind the gap: analysis of marker-assisted breeding strategies for inbred mouse strains

Two genetic loci independently confer susceptibility to autoimmune gastritis

BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells

Disruption of Serinc1, which facilitates serine-derived lipid synthesis, fails to alter macrophage function, lymphocyte proliferation or autoimmune disease susceptibility

Autoimmune-Disease-Prone NOD Mice Help To Reveal a New Genetic Locus for Reducing Pulmonary Disease Caused by Mycoplasma pulmonis

A Susceptibility Allele From a Non-Diabetes-Prone Mouse Strain Accelerates Diabetes in NOD Congenic Mice

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