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Presynaptic facilitation of glutamate release in the basolateral amygdala: A mechanism for the anxiogenic and seizurogenic function of GluK1 receptors

Neuroscience, 2012-09, Vol.221, p.157-169

2012

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Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis

Neuroscience, 2009-09, Vol.163 (1), p.415-429

2009

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Soman induces ictogenesis in the amygdala and interictal activity in the hippocampus that are blocked by a GluR5 kainate receptor antagonist in vitro

Neuroscience, 2009-03, Vol.159 (1), p.380-389

2009

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α7-Containing nicotinic acetylcholine receptors on interneurons of the basolateral amygdala and their role in the regulation of the network excitability

Journal of the neurological sciences, 2013-10, Vol.333, p.e54-e54

2013

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Lamotrigine reduces spontaneous and evoked GABAA receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders

Neuropharmacology, 2002-03, Vol.42 (4), p.522-529

2002

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Lamotrigine reduces spontaneous and evoked GABA A receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders : Lamotrigine and inhibition in the amygdala

Neuropharmacology, 2002, Vol.42 (4), p.522-529

2002

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Presynaptically acting toxins as pharmacological tools to study neurotransmitter release at the neuromuscular junction

Toxicon (Oxford), 1996, Vol.34 (1), p.14-15

1996

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Nanomolar concentrations of Pb2+ increase spontaneous and block evoked transmitter release in hippocampal neurons

Toxicology letters, 1998-07, Vol.95, p.129

1998

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Nanomolar concentrations of Pb 2+ increase spontaneous and block evoked transmitter release in hippocampal neurons

Toxicology letters, 1998, Vol.95, p.129-129

1998

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Effects of tacrine, velnacrine (HP029), suronacrine (HP128), and 3,4‐diaminopyridine on skeletal neuromuscular transmission in vitro

British journal of pharmacology, 1991-04, Vol.102 (4), p.909-915

1991

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Modification of ionic currents underlying action potentials in mouse nerve terminals by the thioloxidizing agent diamide

Neuropharmacology, 1995, Vol.34 (11), p.1529-1533

1995

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Reversal by cysteine of the cadmium‐induced block of skeletal neuromuscular transmission in vitro

British journal of pharmacology, 1992-09, Vol.107 (1), p.95-100

1992

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PREJUNCTIONAL ACTION OF NEOSTIGMINE ON MOUSE NEUROMUSCULAR PREPARATIONS

British Journal of Anaesthesia, 1993-04, Vol.70 (4), p.405-410

1993

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Apparent block of K+ currents in mouse motor nerve terminals by tetrodotoxin, μ‐conotoxin and reduced external sodium

British journal of pharmacology, 1992-05, Vol.106 (1), p.91-94

1992

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Interactions between suxamethonium and non-depolarizing neuromuscular blocking drugs

British journal of anaesthesia : BJA, 1994-02, Vol.72 (2), p.198-204

1994

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The pharmacological effects of cadmium on skeletal neuromuscular transmission

General pharmacology, 1994-12, Vol.25 (8), p.1729-1739

1994

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Bancroftian filariasis in two urban areas of Recife, Brazil: pre-control observations on infection and disease

Transactions of the Royal Society of Tropical Medicine and Hygiene, 1995-07, Vol.89 (4), p.373-377

1995

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Presynaptic facilitation of glutamate release in the basolateral amygdala: A mechanism for the anxiogenic and seizurogenic function of GluK1 receptors

Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis

Soman induces ictogenesis in the amygdala and interictal activity in the hippocampus that are blocked by a GluR5 kainate receptor antagonist in vitro

α7-Containing nicotinic acetylcholine receptors on interneurons of the basolateral amygdala and their role in the regulation of the network excitability

Lamotrigine reduces spontaneous and evoked GABAA receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders

Lamotrigine reduces spontaneous and evoked GABA A receptor-mediated synaptic transmission in the basolateral amygdala: implications for its effects in seizure and affective disorders : Lamotrigine and inhibition in the amygdala

Presynaptically acting toxins as pharmacological tools to study neurotransmitter release at the neuromuscular junction

Nanomolar concentrations of Pb2+ increase spontaneous and block evoked transmitter release in hippocampal neurons

Nanomolar concentrations of Pb 2+ increase spontaneous and block evoked transmitter release in hippocampal neurons

Effects of tacrine, velnacrine (HP029), suronacrine (HP128), and 3,4‐diaminopyridine on skeletal neuromuscular transmission in vitro

Modification of ionic currents underlying action potentials in mouse nerve terminals by the thioloxidizing agent diamide

Reversal by cysteine of the cadmium‐induced block of skeletal neuromuscular transmission in vitro

PREJUNCTIONAL ACTION OF NEOSTIGMINE ON MOUSE NEUROMUSCULAR PREPARATIONS

Apparent block of K+ currents in mouse motor nerve terminals by tetrodotoxin, μ‐conotoxin and reduced external sodium

Interactions between suxamethonium and non-depolarizing neuromuscular blocking drugs

The pharmacological effects of cadmium on skeletal neuromuscular transmission

Bancroftian filariasis in two urban areas of Recife, Brazil: pre-control observations on infection and disease

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