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Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice

The Journal of clinical investigation, 2012-01, Vol.122 (1), p.280-290

2012

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Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

The Journal of biological chemistry, 2006-04, Vol.281 (14), p.9152-9162

2006

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Mechanisms of acquired long QT syndrome in patients with propionic academia

Heart rhythm, 2016-06, Vol.13 (6), p.1335-1345

2016

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Oxytocin exerts harmful cardiac repolarization prolonging effects in drug-induced LQTS

International journal of cardiology. Heart & vasculature, 2022-06, Vol.40, p.101001-101001, Article 101001

2022

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Enhancement of Cardiac Function and Suppression of Heart Failure Progression By Inhibition of Protein Phosphatase 1

Circulation research, 2005-04, Vol.96 (7), p.756-766

2005

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Molecular and Functional Characterization of a Novel Cardiac-Specific Human Tropomyosin Isoform

Circulation (New York, N.Y.), 2010-01, Vol.121 (3), p.410-418

2010

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Calcineurin Increases Cardiac Transient Outward K+ Currents via Transcriptional Up-regulation of Kv4.2 Channel Subunits

The Journal of biological chemistry, 2006-12, Vol.281 (50), p.38498-38506

2006

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Increased expression of the auxiliary beta(2)-subunit of ventricular L-type Ca(2)+ channels leads to single-channel activity characteristic of heart failure

PloS one, 2007-03, Vol.2 (3), p.e292-e292

2007

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Dilated cardiomyopathy with increased SR Ca2+ loading preceded by a hypercontractile state and diastolic failure in the alpha(1C)TG mouse

PloS one, 2009, Vol.4 (1), p.e4133-e4133

2009

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Histidine-rich Ca binding protein: a regulator of sarcoplasmic reticulum calcium sequestration and cardiac function

Journal of molecular and cellular cardiology, 2006-05, Vol.40 (5), p.653-665

2006

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Tetrodotoxin-sensitive Ca2+ Currents, but No T-type Currents in Normal, Hypertrophied, and Failing Mouse Cardiomyocytes

Journal of cardiovascular pharmacology, 2016-12, Vol.68 (6), p.452-464

2016

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Beneficial normalization of cardiac repolarization by carnitine in transgenic short QT syndrome type 1 rabbit models

Cardiovascular research, 2024-07

2024

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Calcineurin‐dependent cardiomyopathy is activated by TRPC in the adult mouse heart

The FASEB journal, 2006-08, Vol.20 (10), p.1660-1670

2006

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The L-type calcium channel in the heart: the beat goes on

The Journal of clinical investigation, 2005-12, Vol.115 (12), p.3306-3317

2005

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The IP3 Receptor Regulates Cardiac Hypertrophy in Response to Select Stimuli

Circulation research, 2010-09, Vol.107 (5), p.659-666

2010

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Postpartum hormones oxytocin and prolactin cause pro-arrhythmic prolongation of cardiac repolarization in long QT syndrome type 2

Europace (London, England), 2019-07, Vol.21 (7), p.1126-1138

2019

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Architecture of Ca2+ Channel Pore-lining Segments Revealed by Covalent Modification of Substituted Cysteines

The Journal of biological chemistry, 2000-11, Vol.275 (44), p.34493-34500

2000

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Caveolae in Rabbit Ventricular Myocytes: Distribution and Dynamic Diminution after Cell Isolation

Biophysical journal, 2017-09, Vol.113 (5), p.1047-1059

2017

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Retraction: Gαq-mediated activation of GRK2 by mechanical stretch in cardiac myocytes. THE ROLE OF PROTEIN KINASE C

The Journal of biological chemistry, 2016-07, Vol.291 (27), p.14390-14390

2016

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PKC-α regulates cardiac contractility and propensity toward heart failure

Nature medicine, 2004-03, Vol.10 (3), p.248-254

2004

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Calcium Cycling, Historic Overview and Perspectives. Role for Autonomic Nervous System Regulation

Journal of Molecular and Cellular Cardiology, 2002-08, Vol.34 (8), p.885-896

2002

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Increased Expression of the Auxiliary β2-subunit of Ventricular L-type Ca2+ Channels Leads to Single-Channel Activity Characteristic of Heart Failure

PloS one, 2007-03, Vol.2 (3), p.e292

2007

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Targeted disruption of the voltage-dependent calcium channel α2/δ-1-subunit

American journal of physiology. Heart and circulatory physiology, 2009-07, Vol.297 (1), p.H117-H124

2009

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A Region in IVS5 of the Human Cardiac L-type Calcium Channel Is Required for the Use-dependent Block by Phenylalkylamines and Benzothiazepines

The Journal of biological chemistry, 1999-04, Vol.274 (14), p.9409-9420

1999

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Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

Mechanisms of acquired long QT syndrome in patients with propionic academia

Oxytocin exerts harmful cardiac repolarization prolonging effects in drug-induced LQTS

Enhancement of Cardiac Function and Suppression of Heart Failure Progression By Inhibition of Protein Phosphatase 1

Molecular and Functional Characterization of a Novel Cardiac-Specific Human Tropomyosin Isoform

Calcineurin Increases Cardiac Transient Outward K+ Currents via Transcriptional Up-regulation of Kv4.2 Channel Subunits

Increased expression of the auxiliary beta(2)-subunit of ventricular L-type Ca(2)+ channels leads to single-channel activity characteristic of heart failure

Dilated cardiomyopathy with increased SR Ca2+ loading preceded by a hypercontractile state and diastolic failure in the alpha(1C)TG mouse

Histidine-rich Ca binding protein: a regulator of sarcoplasmic reticulum calcium sequestration and cardiac function

Tetrodotoxin-sensitive Ca2+ Currents, but No T-type Currents in Normal, Hypertrophied, and Failing Mouse Cardiomyocytes

Beneficial normalization of cardiac repolarization by carnitine in transgenic short QT syndrome type 1 rabbit models

Calcineurin‐dependent cardiomyopathy is activated by TRPC in the adult mouse heart

The L-type calcium channel in the heart: the beat goes on

The IP3 Receptor Regulates Cardiac Hypertrophy in Response to Select Stimuli

Transgenic short-QT syndrome 1 rabbits mimic the human disease phenotype with QT/action potential duration shortening in the atria and ventricles and increased ventricular tachycardia/ventricular fibrillation inducibility

Postpartum hormones oxytocin and prolactin cause pro-arrhythmic prolongation of cardiac repolarization in long QT syndrome type 2

Architecture of Ca2+ Channel Pore-lining Segments Revealed by Covalent Modification of Substituted Cysteines

Caveolae in Rabbit Ventricular Myocytes: Distribution and Dynamic Diminution after Cell Isolation

Retraction: Gαq-mediated activation of GRK2 by mechanical stretch in cardiac myocytes. THE ROLE OF PROTEIN KINASE C

PKC-α regulates cardiac contractility and propensity toward heart failure

Calcium Cycling, Historic Overview and Perspectives. Role for Autonomic Nervous System Regulation

Increased Expression of the Auxiliary β2-subunit of Ventricular L-type Ca2+ Channels Leads to Single-Channel Activity Characteristic of Heart Failure

Targeted disruption of the voltage-dependent calcium channel α2/δ-1-subunit

A Region in IVS5 of the Human Cardiac L-type Calcium Channel Is Required for the Use-dependent Block by Phenylalkylamines and Benzothiazepines

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