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Oxidative damage and metabolic dysfunction in Huntington's disease: Selective vulnerability of the basal ganglia

Annals of neurology, 1997-05, Vol.41 (5), p.646-653

1997

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Evidence for the GluR6 gene associated with younger onset age of Huntington's disease

Neurology, 1999-10, Vol.53 (6), p.1330-1330

1999

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Increased GABAA receptor binding in superficial layers of cingulate cortex in schizophrenics

The Journal of neuroscience, 1992-03, Vol.12 (3), p.924-929

1992

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Kynurenine pathway abnormalities in Parkinson's disease

Neurology, 1992-09, Vol.42 (9), p.1702-1702

1992

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Reduced neuronal size in posterior hippocampus of schizophrenic patients

Schizophrenia bulletin, 1991, Vol.17 (4), p.597-608

1991

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Selective Sparing of a Class of Striatal Neurons in Huntington's Disease

Science (American Association for the Advancement of Science), 1985-11, Vol.230 (4725), p.561-563

1985

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Decreased Neuronal and Increased Oligodendroglial Densities in Huntingtonʼs Disease Caudate Nucleus

Journal of neuropathology and experimental neurology, 1991-11, Vol.50 (6), p.729-742

1991

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De novo expansion of a (CAG) n repeat in sporadic Huntington's disease

Nature genetics, 1993-10, Vol.5 (2), p.168-173

1993

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Morphometric analysis of the prefrontal cortex in Huntington's disease

Neurology, 1991-07, Vol.41 (7), p.1117-1117

1991

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Growth-Associated Protein GAP-43 is Expressed Selectively in Associative Regions of the Adult Human Brain

Proceedings of the National Academy of Sciences - PNAS, 1988-05, Vol.85 (10), p.3638-3642

1988

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Kynurenine Pathway Measurements in Huntington's Disease Striatum: Evidence for Reduced Formation of Kynurenic Acid

Journal of neurochemistry, 1990-10, Vol.55 (4), p.1327-1339

1990

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An Improved Approach to Prepare Human Brains for Research

Journal of neuropathology and experimental neurology, 1995-01, Vol.54 (1), p.42-56

1995

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3-hydroxyanthranilate Oxygenase Activity is Increased in the Brains of Huntington Disease Victims

Proceedings of the National Academy of Sciences - PNAS, 1988-06, Vol.85 (11), p.4079-4081

1988

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Neuropathological Classification of Huntingtonʼs Disease

Journal of neuropathology and experimental neurology, 1985-11, Vol.44 (6), p.559-577

1985

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Meta-analysis of postmortem studies of Alzheimer's disease-like neuropathology in schizophrenia [published erratum appears in Am J Psychiatry 1997 Aug;154(8):1180]

The American journal of psychiatry, 1997-06, Vol.154 (6), p.861-863

1997

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Kynurenic acid concentrations are reduced in Huntington's disease cerebral cortex

Journal of the neurological sciences, 1992-03, Vol.108 (1), p.80-87

1992

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Localization of the growth-associated phosphoprotein GAP-43 (B-50, F1) in the human cerebral cortex

The Journal of neuroscience, 1989-03, Vol.9 (3), p.990-995

1989

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Dysfunction of brain kynurenic acid metabolism in Huntington's disease: focus on kynurenine aminotransferases

Journal of the neurological sciences, 1995-05, Vol.130 (1), p.39-47

1995

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Human brain dopamine receptors in children and aging adults

Synapse (New York, N.Y.), 1987, Vol.1 (5), p.399-404

1987

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Clinical and neuropathologic assessment of severity in Huntington's disease

Neurology, 1988-03, Vol.38 (3), p.341-341

1988

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Quantitative neuropathological changes in presymptomatic Huntington's disease

Annals of neurology, 2001-01, Vol.49 (1), p.29-34

2001

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Basic fibroblast growth factor in Alzheimer's disease

Biochemical and biophysical research communications, 1990-09, Vol.171 (2), p.690-696

1990

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Lysosomal proteinase antigens are prominently localized within senile plaques of Alzheimer's disease: evidence for a neuronal origin

Brain research, 1990-04, Vol.513 (2), p.181-192

1990

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Early central Atlantic Ocean seafloor spreading history

Geosphere (Boulder, Colo.), 2007-10, Vol.3 (5), p.282-298

2007

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Levels of the Growth-Associated Protein GAP-43 are Selectively Increased in Association Cortices in Schizophrenia

Proceedings of the National Academy of Sciences - PNAS, 1996-11, Vol.93 (24), p.14182-14187

1996

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Oxidative damage and metabolic dysfunction in Huntington's disease: Selective vulnerability of the basal ganglia

Evidence for the GluR6 gene associated with younger onset age of Huntington's disease

Increased GABAA receptor binding in superficial layers of cingulate cortex in schizophrenics

Kynurenine pathway abnormalities in Parkinson's disease

Reduced neuronal size in posterior hippocampus of schizophrenic patients

Selective Sparing of a Class of Striatal Neurons in Huntington's Disease

Decreased Neuronal and Increased Oligodendroglial Densities in Huntingtonʼs Disease Caudate Nucleus

De novo expansion of a (CAG) n repeat in sporadic Huntington's disease

Morphometric analysis of the prefrontal cortex in Huntington's disease

Growth-Associated Protein GAP-43 is Expressed Selectively in Associative Regions of the Adult Human Brain

Kynurenine Pathway Measurements in Huntington's Disease Striatum: Evidence for Reduced Formation of Kynurenic Acid

An Improved Approach to Prepare Human Brains for Research

3-hydroxyanthranilate Oxygenase Activity is Increased in the Brains of Huntington Disease Victims

Neuropathological Classification of Huntingtonʼs Disease

Meta-analysis of postmortem studies of Alzheimer's disease-like neuropathology in schizophrenia [published erratum appears in Am J Psychiatry 1997 Aug;154(8):1180]

Kynurenic acid concentrations are reduced in Huntington's disease cerebral cortex

Localization of the growth-associated phosphoprotein GAP-43 (B-50, F1) in the human cerebral cortex

Dysfunction of brain kynurenic acid metabolism in Huntington's disease: focus on kynurenine aminotransferases

Human brain dopamine receptors in children and aging adults

Clinical and neuropathologic assessment of severity in Huntington's disease

Quantitative neuropathological changes in presymptomatic Huntington's disease

Basic fibroblast growth factor in Alzheimer's disease

Lysosomal proteinase antigens are prominently localized within senile plaques of Alzheimer's disease: evidence for a neuronal origin

Early central Atlantic Ocean seafloor spreading history

Levels of the Growth-Associated Protein GAP-43 are Selectively Increased in Association Cortices in Schizophrenia

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