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Role of MAPKs in development and differentiation: lessons from knockout mice

Biochimie, 2006-09, Vol.88 (9), p.1091-1098

2006

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The Extracellular Signal–Regulated Kinase Isoform ERK1 Is Specifically Required for In Vitro and In Vivo Adipogenesis

Diabetes (New York, N.Y.), 2005-02, Vol.54 (2), p.402-411

2005

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The role of MAPKs in adipocyte differentiation and obesity

Biochimie, 2005, Vol.87 (1), p.51-56

2005

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Deficiency in the extracellular signal-regulated kinase 1 (ERK1) protects leptin-deficient mice from insulin resistance without affecting obesity

Diabetologia, 2011-01, Vol.54 (1), p.180-189

2011

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A p38MAPK-p53 cascade regulates mesodermal differentiation and neurogenesis of embryonic stem cells

Cell death & disease, 2013-07, Vol.4 (7), p.e737-e737

2013

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p38MAP Kinase activity is required for human primary adipocyte differentiation

FEBS letters, 2007-12, Vol.581 (29), p.5591-5596

2007

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Oncoprotein-mediated signalling cascade stimulates c-Jun activity by phosphorylation of serines 63 and 73

Molecular and Cellular Biology, 1992-08, Vol.12 (8), p.3507-3513

1992

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Inhibition of p38MAPK Increases Adipogenesis From Embryonic to Adult Stages

Diabetes (New York, N.Y.), 2006-02, Vol.55 (2), p.281-289

2006

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D027 Role de la voie de transduction P38MAPK (mitogen-activated protein kinase) dans les differenciations endotheliales et myogeniques des cellules souches embryonnaires

Archives of cardiovascular diseases, 2009, Vol.102, p.S46-S46

2009

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D028 L’expression des gènes PAI-1, tPA et uPA est fortement régulée pendant la différenciation des cellules souches embryonnaires en myocytes et adipocytes

Archives of cardiovascular diseases, 2009, Vol.102, p.S46-S47

2009

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The Wilms' tumor gene product represses the transcription of thrombospondin 1 in response to overexpression of c-Jun

Oncogene, 1999-05, Vol.18 (20), p.3143-3151

1999

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Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain

Nature (London), 1991-05, Vol.351 (6322), p.122-127

1991

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Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Molecular and Cellular Biology, 1996-05, Vol.16 (5), p.1881-1888

1996

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Retinoic acid activation of the ERK pathway is required for embryonic stem cell commitment into the adipocyte lineage

Biochemical journal, 2002-02, Vol.361 (Pt 3), p.621-627

2002

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Protein kinase-dependent overexpression of the nuclear protein pirin in c-JUN and RAS transformed fibroblasts

Cellular and molecular life sciences : CMLS, 1999-03, Vol.55 (3), p.467-471

1999

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A new role for the oncogenic high-mobility group A2 transcription factor in myogenesis of embryonic stem cells

Oncogene, 2005-09, Vol.24 (41), p.6281-6291

2005

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The Lac repressor provides a reversible gene expression system in undifferentiated and differentiated embryonic stem cell

Cellular and molecular life sciences : CMLS, 2005-07, Vol.62 (14), p.1605-1612

2005

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Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73

Nature (London), 1991-12, Vol.354 (6353), p.494-496

1991

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SPARC and thrombospondin genes are repressed by the c‐jun oncogene in rat embryo fibroblasts

The EMBO journal, 1994-12, Vol.13 (23), p.5668-5678

1994

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The c-Jun-induced transformation process involves complex regulation of tenascin-C expression

Molecular and Cellular Biology, 1997-06, Vol.17 (6), p.3202-3209

1997

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The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells

Molecular and Cellular Biology, 1991-12, Vol.11 (12), p.6286-6295

1991

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junB promoter regulation : Ras mediated transactivation by c-Ets-1 and c-Ets-2

Oncogene, 1994-03, Vol.9 (3), p.911-921

1994

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The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site

Oncogene, 2001-11, Vol.20 (50), p.7425

2001

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Control of AP-1 activity by signal transduction cascades

Advances in second messenger and phosphoprotein research, 1993, Vol.28, p.255

1993

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The transformed phenotype in culture and tumorigenicity of fischer rat fibroblast cells (FR3T3) transformed with bovine papilloma virus type 1

Virology (New York, N.Y.), 1984-06, Vol.135 (2), p.406-416

1984

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Role of MAPKs in development and differentiation: lessons from knockout mice

The Extracellular Signal–Regulated Kinase Isoform ERK1 Is Specifically Required for In Vitro and In Vivo Adipogenesis

The role of MAPKs in adipocyte differentiation and obesity

Deficiency in the extracellular signal-regulated kinase 1 (ERK1) protects leptin-deficient mice from insulin resistance without affecting obesity

A p38MAPK-p53 cascade regulates mesodermal differentiation and neurogenesis of embryonic stem cells

p38MAP Kinase activity is required for human primary adipocyte differentiation

Oncoprotein-mediated signalling cascade stimulates c-Jun activity by phosphorylation of serines 63 and 73

Inhibition of p38MAPK Increases Adipogenesis From Embryonic to Adult Stages

D027 Role de la voie de transduction P38MAPK (mitogen-activated protein kinase) dans les differenciations endotheliales et myogeniques des cellules souches embryonnaires

D028 L’expression des gènes PAI-1, tPA et uPA est fortement régulée pendant la différenciation des cellules souches embryonnaires en myocytes et adipocytes

The Wilms' tumor gene product represses the transcription of thrombospondin 1 in response to overexpression of c-Jun

Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain

Stepwise transformation of rat embryo fibroblasts: c-Jun, JunB, or JunD can cooperate with Ras for focus formation, but a c-Jun-containing heterodimer is required for immortalization

Retinoic acid activation of the ERK pathway is required for embryonic stem cell commitment into the adipocyte lineage

Protein kinase-dependent overexpression of the nuclear protein pirin in c-JUN and RAS transformed fibroblasts

A new role for the oncogenic high-mobility group A2 transcription factor in myogenesis of embryonic stem cells

The Lac repressor provides a reversible gene expression system in undifferentiated and differentiated embryonic stem cell

Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73

SPARC and thrombospondin genes are repressed by the c‐jun oncogene in rat embryo fibroblasts

The c-Jun-induced transformation process involves complex regulation of tenascin-C expression

The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells

junB promoter regulation : Ras mediated transactivation by c-Ets-1 and c-Ets-2

The defective transforming phenotype of c-Jun Ala(63/73) is rescued by mutation of the C-terminal phosphorylation site

Control of AP-1 activity by signal transduction cascades

The transformed phenotype in culture and tumorigenicity of fischer rat fibroblast cells (FR3T3) transformed with bovine papilloma virus type 1

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