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Monitoring patients treated with anti-TNF-α biopharmaceuticals: assessing serum infliximab and anti-infliximab antibodies

Rheumatology (Oxford, England), 2007-12, Vol.46 (12), p.1828-1834

2007

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Letter: can addition of an immunomodulator really reverse antibody formation and loss of response in patients treated with adalimumab?

Alimentary pharmacology & therapeutics, 2017-03, Vol.45 (5), p.759-760

2017

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Monitoring patients treated with anti-TNF- biopharmaceuticals: assessing serum infliximab and anti-infliximab antibodies

Rheumatology (Oxford, England), 2007-12, Vol.46 (12), p.1828-1834

2007

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Severe infusion reactions to infliximab: aetiology, immunogenicity and risk factors in patients with inflammatory bowel disease

Alimentary pharmacology & therapeutics, 2011-07, Vol.34 (1), p.51-58

2011

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Prognostic laboratory markers of joint damage in rheumatoid arthritis

Annals of the rheumatic diseases, 2005-02, Vol.64 (2), p.196-201

2005

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Lewis and AB0 blood group-phenotypes in periodontitis, cardiovascular disease, obesity and stroke

Scientific reports, 2019-04, Vol.9 (1), p.6283-6283, Article 6283

2019

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Formation of antibodies against infliximab and adalimumab strongly correlates with functional drug levels and clinical responses in rheumatoid arthritis

Annals of the rheumatic diseases, 2009-11, Vol.68 (11), p.1739-1745

2009

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Pre‐existing IgG antibodies cross‐reacting with the Fab region of infliximab predict efficacy and safety of infliximab therapy in inflammatory bowel disease

Alimentary pharmacology & therapeutics, 2013-06, Vol.37 (12), p.1172-1183

2013

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Appearance and disappearance of neutralizing antibodies during interferon-beta therapy

Neurology, 2005-07, Vol.65 (1), p.33

2005

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Cytokines, autoantibodies and viral antibodies in premorbid and postdiagnostic sera from patients with rheumatoid arthritis: case–control study nested in a cohort of Norwegian blood donors

Annals of the rheumatic diseases, 2008-06, Vol.67 (6), p.860-866

2008

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Letter: persistence of anti-infliximab antibodies after discontinuation of infliximab in patients with IBD

Alimentary pharmacology & therapeutics, 2012-09, Vol.36 (5), p.499-500

2012

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Genetic polymorphisms of tumour necrosis factor receptor superfamily 1b and fas ligand are associated with clinical efficacy and/or acute severe infusion reactions to infliximab in Crohn's disease

Alimentary pharmacology & therapeutics, 2012-10, Vol.36 (7), p.650-659

2012

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Single nucleotide polymorphisms in genes encoding toll-like receptors 7, 8 and 9 in Danish patients with systemic lupus erythematosus

Molecular biology reports, 2014-09, Vol.41 (9), p.5755-5763

2014

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Serum concentrations of the interferon-γ-inducible chemokine IP-10/CXCl10 are augmented in both newly diagnosed Type I diabetes mellitus patients and subjects at risk of developing the disease

Diabetologia, 2002-08, Vol.45 (8), p.1107-1110

2002

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Genetic polymorphisms of dsRNA ligating pattern recognition receptors TLR3, MDA5, and RIG-I. Association with systemic lupus erythematosus and clinical phenotypes

Rheumatology international, 2014-10, Vol.34 (10), p.1401-1408

2014

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Lower limb ischaemia and reperfusion injury in healthy volunteers measured by oxidative and inflammatory biomarkers

Perfusion, 2015-01, Vol.30 (1), p.64-70

2015

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FRI0031 Naturally occurring antibodies against different IFN-alpha subtypes are observed in some SLE patients, and may impact the IFN gene signature and disease activity

Annals of the rheumatic diseases, 2013-06, Vol.71 (Suppl 3), p.320-320

2013

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Specific and Strain-Independent Effects of Dexamethasone in the Prevention and Treatment of Experimental Autoimmune Encephalomyelitis in Rodents

Scandinavian journal of immunology, 2010-11, Vol.72 (5), p.396-407

2010

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Commentary: antibodies reacting with the infliximab Fab portion – something new? Authors' reply

Alimentary pharmacology & therapeutics, 2013-09, Vol.38 (5), p.552-553

2013

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Spironolactone inhibits production of proinflammatory cytokines, including tumour necrosis factor‐α and interferon‐γ, and has potential in the treatment of arthritis

Clinical and experimental immunology, 2003-10, Vol.134 (1), p.151-158

2003

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The clinical effect of neutralizing antibodies against interferon-beta is independent of the type of interferon-beta used for patients with relapsing-remitting multiple sclerosis

Multiple sclerosis, 2009-05, Vol.15 (5), p.601-605

2009

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Are ex vivo neutralising antibodies against IFN-β always detrimental to therapeutic efficacy in multiple sclerosis?

Multiple sclerosis, 2007-06, Vol.13 (5), p.616-621

2007

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In Vitro Cytokine Responses to Periodontal Pathogens: Generalized Aggressive Periodontitis is Associated with Increased IL-6 Response to Porphyromonas gingivalis

Scandinavian journal of immunology, 2010-06, Vol.71 (6), p.440-446

2010

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Validity of histopathological grading of articular cartilage from osteoarthritic knee joints

Annals of the rheumatic diseases, 1999-04, Vol.58 (4), p.208-213

1999

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Methotrexate induces poly(ADP-ribose) polymerase-dependent, caspase 3-independent apoptosis in subsets of proliferating CD4⁺ T cells

Clinical and experimental immunology, 2007-05, Vol.148 (2), p.288-295

2007

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Monitoring patients treated with anti-TNF-α biopharmaceuticals: assessing serum infliximab and anti-infliximab antibodies

Letter: can addition of an immunomodulator really reverse antibody formation and loss of response in patients treated with adalimumab?

Monitoring patients treated with anti-TNF- biopharmaceuticals: assessing serum infliximab and anti-infliximab antibodies

Severe infusion reactions to infliximab: aetiology, immunogenicity and risk factors in patients with inflammatory bowel disease

Prognostic laboratory markers of joint damage in rheumatoid arthritis

Lewis and AB0 blood group-phenotypes in periodontitis, cardiovascular disease, obesity and stroke

Formation of antibodies against infliximab and adalimumab strongly correlates with functional drug levels and clinical responses in rheumatoid arthritis

Pre‐existing IgG antibodies cross‐reacting with the Fab region of infliximab predict efficacy and safety of infliximab therapy in inflammatory bowel disease

Appearance and disappearance of neutralizing antibodies during interferon-beta therapy

Cytokines, autoantibodies and viral antibodies in premorbid and postdiagnostic sera from patients with rheumatoid arthritis: case–control study nested in a cohort of Norwegian blood donors

Letter: persistence of anti-infliximab antibodies after discontinuation of infliximab in patients with IBD

Genetic polymorphisms of tumour necrosis factor receptor superfamily 1b and fas ligand are associated with clinical efficacy and/or acute severe infusion reactions to infliximab in Crohn's disease

Single nucleotide polymorphisms in genes encoding toll-like receptors 7, 8 and 9 in Danish patients with systemic lupus erythematosus

Serum concentrations of the interferon-γ-inducible chemokine IP-10/CXCl10 are augmented in both newly diagnosed Type I diabetes mellitus patients and subjects at risk of developing the disease

Genetic polymorphisms of dsRNA ligating pattern recognition receptors TLR3, MDA5, and RIG-I. Association with systemic lupus erythematosus and clinical phenotypes

Lower limb ischaemia and reperfusion injury in healthy volunteers measured by oxidative and inflammatory biomarkers

FRI0031 Naturally occurring antibodies against different IFN-alpha subtypes are observed in some SLE patients, and may impact the IFN gene signature and disease activity

Specific and Strain-Independent Effects of Dexamethasone in the Prevention and Treatment of Experimental Autoimmune Encephalomyelitis in Rodents

Commentary: antibodies reacting with the infliximab Fab portion – something new? Authors' reply

Spironolactone inhibits production of proinflammatory cytokines, including tumour necrosis factor‐α and interferon‐γ, and has potential in the treatment of arthritis

The clinical effect of neutralizing antibodies against interferon-beta is independent of the type of interferon-beta used for patients with relapsing-remitting multiple sclerosis

Are ex vivo neutralising antibodies against IFN-β always detrimental to therapeutic efficacy in multiple sclerosis?

In Vitro Cytokine Responses to Periodontal Pathogens: Generalized Aggressive Periodontitis is Associated with Increased IL-6 Response to Porphyromonas gingivalis

Validity of histopathological grading of articular cartilage from osteoarthritic knee joints

Methotrexate induces poly(ADP-ribose) polymerase-dependent, caspase 3-independent apoptosis in subsets of proliferating CD4⁺ T cells

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