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Class III PI3K Vps34 plays an essential role in autophagy and in heart and liver function

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (6), p.2003-2008

2012

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Class IA PI3K p110β Subunit Promotes Autophagy through Rab5 Small GTPase in Response to Growth Factor Limitation

Molecular cell, 2013-04, Vol.50 (1), p.29-42

2013

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Tumor-intrinsic PIK3CA represses tumor immunogenicity in a model of pancreatic cancer

The Journal of clinical investigation, 2019-08, Vol.129 (8), p.3264-3276

2019

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Suppression of phosphoinositide 3-kinase signaling and alteration of multiple ion currents in drug-induced long QT syndrome

Science translational medicine, 2012-04, Vol.4 (131), p.131ra50

2012

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Increased Persistent Sodium Current Due to Decreased PI3K Signaling Contributes to QT Prolongation in the Diabetic Heart

Diabetes (New York, N.Y.), 2013-12, Vol.62 (12), p.4257-4265

2013

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Regulation of heart rate and the pacemaker current by phosphoinositide 3-kinase signaling

The Journal of general physiology, 2019-08, Vol.151 (8), p.1051-1058

2019

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Regulation of HCN2 Current by PI3K/Akt Signaling

Frontiers in physiology, 2020-11, Vol.11, p.587040-587040

2020

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PI3Ks maintain the structural integrity of T-tubules in cardiac myocytes

PloS one, 2011-09, Vol.6 (9), p.e24404

2011

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Decreased l-Type Ca2+ Current in Cardiac Myocytes of Type 1 Diabetic Akita Mice Due to Reduced Phosphatidylinositol 3-Kinase Signaling

Diabetes (New York, N.Y.), 2007-11, Vol.56 (11), p.2780-2789

2007

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Control of Cardiac Repolarization by Phosphoinositide 3-Kinase Signaling to Ion Channels

Circulation research, 2015-01, Vol.116 (1), p.127-137

2015

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PI 3‐kinase regulation of dopamine uptake

Journal of neurochemistry, 2002-05, Vol.81 (4), p.859-869

2002

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Inhibition of Mammalian Target of Rapamycin Signaling by 2-(Morpholin-1-yl)pyrimido[2,1-α]isoquinolin-4-one

The Journal of biological chemistry, 2007-08, Vol.282 (33), p.24463-24470

2007

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Acquired Long QT Syndrome and Phosphoinositide 3-kinase

Trends in cardiovascular medicine, 2017-10, Vol.27 (7), p.451-459

2017

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Adipose tissue insulin resistance due to loss of PI3K p110α leads to decreased energy expenditure and obesity

American journal of physiology: endocrinology and metabolism, 2014-05, Vol.306 (10), p.E1205-E1216

2014

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Ablation of PI3K p110-α Prevents High-Fat Diet-Induced Liver Steatosis

Diabetes (New York, N.Y.), 2011-05, Vol.60 (5), p.1483-1492

2011

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class IA phosphatidylinositol 3-kinase p110-β subunit is a positive regulator of autophagy

The Journal of cell biology, 2010-11, Vol.191 (4), p.827-843

2010

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Activation of Gαq in Cardiomyocytes Increases Vps34 Activity and Stimulates Autophagy

Journal of cardiovascular pharmacology, 2017-04, Vol.69 (4), p.198-211

2017

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Loss of Cardiac Phosphoinositide 3-Kinase p110α Results in Contractile Dysfunction

Circulation (New York, N.Y.), 2009-07, Vol.120 (4), p.318-325

2009

Volltextzugriff (PDF)

Restoration of Defective L-type Ca2+ Current in Cardiac Myocytes of Type 2 Diabetic db/db Mice by Akt and PKC-ι

Journal of cardiovascular pharmacology, 2011-10, Vol.58 (4), p.439-445

2011

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Gαq binds to p110α/p85α phosphoinositide 3-kinase and displaces Ras

Biochemical journal, 2006-03, Vol.394 (3), p.557-562

2006

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Activated G alpha q inhibits p110 alpha phosphatidylinositol 3-kinase and Akt

The Journal of biological chemistry, 2003-06, Vol.278 (26), p.23472-23479

2003

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Rapamycin-insensitive Regulation of 4E-BP1 in Regenerating Rat Liver

The Journal of biological chemistry, 2001-04, Vol.276 (14), p.10943-10951

2001

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Energy balancing by fat Pik3ca

Adipocyte, 2015-01, Vol.4 (1), p.70-74

2015

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Increase in Human Muscle Protein Synthesis Induced by Food Intake Is Similar When Assessed with the Constant Infusion and Flooding Techniques

The Journal of nutrition, 2006-06, Vol.136 (6), p.1504-1510

2006

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Ca(2+)- and phospholipase D-dependent and -independent pathways activate mTOR signaling

FEBS letters, 2003-08, Vol.550 (1-3), p.51-56

2003

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Class III PI3K Vps34 plays an essential role in autophagy and in heart and liver function

Class IA PI3K p110β Subunit Promotes Autophagy through Rab5 Small GTPase in Response to Growth Factor Limitation

Tumor-intrinsic PIK3CA represses tumor immunogenicity in a model of pancreatic cancer

Suppression of phosphoinositide 3-kinase signaling and alteration of multiple ion currents in drug-induced long QT syndrome

Increased Persistent Sodium Current Due to Decreased PI3K Signaling Contributes to QT Prolongation in the Diabetic Heart

Regulation of heart rate and the pacemaker current by phosphoinositide 3-kinase signaling

Regulation of HCN2 Current by PI3K/Akt Signaling

PI3Ks maintain the structural integrity of T-tubules in cardiac myocytes

Decreased l-Type Ca2+ Current in Cardiac Myocytes of Type 1 Diabetic Akita Mice Due to Reduced Phosphatidylinositol 3-Kinase Signaling

Control of Cardiac Repolarization by Phosphoinositide 3-Kinase Signaling to Ion Channels

PI 3‐kinase regulation of dopamine uptake

Inhibition of Mammalian Target of Rapamycin Signaling by 2-(Morpholin-1-yl)pyrimido[2,1-α]isoquinolin-4-one

Acquired Long QT Syndrome and Phosphoinositide 3-kinase

Adipose tissue insulin resistance due to loss of PI3K p110α leads to decreased energy expenditure and obesity

Ablation of PI3K p110-α Prevents High-Fat Diet-Induced Liver Steatosis

class IA phosphatidylinositol 3-kinase p110-β subunit is a positive regulator of autophagy

Activation of Gαq in Cardiomyocytes Increases Vps34 Activity and Stimulates Autophagy

Loss of Cardiac Phosphoinositide 3-Kinase p110α Results in Contractile Dysfunction

Restoration of Defective L-type Ca2+ Current in Cardiac Myocytes of Type 2 Diabetic db/db Mice by Akt and PKC-ι

Gαq binds to p110α/p85α phosphoinositide 3-kinase and displaces Ras

Activated G alpha q inhibits p110 alpha phosphatidylinositol 3-kinase and Akt

Rapamycin-insensitive Regulation of 4E-BP1 in Regenerating Rat Liver

Energy balancing by fat Pik3ca

Increase in Human Muscle Protein Synthesis Induced by Food Intake Is Similar When Assessed with the Constant Infusion and Flooding Techniques

Ca(2+)- and phospholipase D-dependent and -independent pathways activate mTOR signaling

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