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Rare mutations in Pfmdr1 gene of Plasmodium falciparum detected in clinical isolates from patients treated with anti-malarial drug in Nigeria

Malaria journal, 2019-09, Vol.18 (1), p.319-319, Article 319

2019

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Spectrum of musculoskeletal disorders in Nigerians with types 2 diabetes mellitus: prevalence and predictors

Reumatismo, 2020-01, Vol.71 (4), p.209-217

2020

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The COVID-19 pandemic in sub-Saharan Africa: The significance of presumed immune sufficiency

African journal of laboratory medicine, 2023, Vol.12 (1), p.1-10

2023

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Preponderance of toxigenic Escherichia coli in stool pathogens correlates with toxin detection in accessible drinking-water sources

Epidemiology and infection, 2015-02, Vol.143 (3), p.494-504

2015

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Intrathymic and intrasplenic oxidative stress mediates thymocyte and splenocyte damage in acutely exercised mice

Journal of applied physiology (1985), 1999-06, Vol.86 (6), p.1823-1827

1999

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The Roles of Unfolded Protein Response Pathways in Chlamydia Pathogenesis

The Journal of infectious diseases, 2017-02, Vol.215 (3), p.456-465

2017

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The molecular mechanism of induction of unfolded protein response by Chlamydia

Biochemical and biophysical research communications, 2019-01, Vol.508 (2), p.421-429

2019

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Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling events

Biological chemistry, 2004-01, Vol.385 (1), p.67-74

2004

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Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophages

Immunobiology (1979), 2009-02, Vol.214 (2), p.143-152

2009

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Chlamydia pneumoniae infected macrophages exhibit enhanced plasma membrane fluidity and show increased adherence to endothelial cells

Molecular and cellular biochemistry, 2005-01, Vol.269 (1-2), p.69-84

2005

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Chlamydia pneumoniae survival in macrophages is regulated by free Ca2+ dependent reactive nitrogen and oxygen species

The Journal of infection, 2003-02, Vol.46 (2), p.120-128

2003

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Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cells

Immunobiology (1979), 2011-12, Vol.216 (12), p.1248-1255

2011

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Effective macrophage redox defense against Chlamydia pneumoniae depends on L-type Ca2+ channel activation

Medical microbiology and immunology, 2003-05, Vol.192 (2), p.99-106

2003

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Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophages

Comparative immunology, microbiology and infectious diseases, 2010-12, Vol.33 (6), p.513-528

2010

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Generation of reactive oxygen species and formation of membrane lipid peroxides in cells infected with Chlamydia trachomatis

International journal of infectious diseases, 2000, Vol.4 (1), p.46-50

2000

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Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genes

Medical microbiology and immunology, 2005, Vol.194 (1-2), p.91-103

2005

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Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibility

Immunobiology (1979), 2003, Vol.207 (4), p.237-245

2003

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Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: Evidence of the effect of redox balance on host–pathogen relationship

Immunobiology (1979), 2006-01, Vol.211 (5), p.325-339

2006

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17 beta-estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophages

Journal of reproductive immunology, 2003-06, Vol.59 (1), p.17-28

2003

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Association of Chlamydia trachomatis antibodies with genital contact disease in women in Benin City, Nigeria

Tropical medicine & international health, 1997-04, Vol.2 (4), p.389-392

1997

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Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelae

International journal of medical sciences, 2007-09, Vol.4 (4), p.223-231

2007

Volltextzugriff (PDF)

Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca super(2+) dependent manner in human macrophages

Comparative immunology, microbiology and infectious diseases, 2010-12, Vol.33 (6), p.513-528

2010

Volltextzugriff (PDF)

Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca 2+ dependent manner in human macrophages

Comparative immunology, microbiology and infectious diseases, 2010, Vol.33 (6), p.513-528

2010

Volltextzugriff (PDF)

Free intracellular Ca 2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophages

Immunobiology (1979), 2009, Vol.214 (2), p.143-152

2009

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17β-Estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophages

Journal of reproductive immunology, 2003-06, Vol.59 (1), p.17-28

2003

Volltextzugriff (PDF)

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Rare mutations in Pfmdr1 gene of Plasmodium falciparum detected in clinical isolates from patients treated with anti-malarial drug in Nigeria

Spectrum of musculoskeletal disorders in Nigerians with types 2 diabetes mellitus: prevalence and predictors

The COVID-19 pandemic in sub-Saharan Africa: The significance of presumed immune sufficiency

Preponderance of toxigenic Escherichia coli in stool pathogens correlates with toxin detection in accessible drinking-water sources

Intrathymic and intrasplenic oxidative stress mediates thymocyte and splenocyte damage in acutely exercised mice

The Roles of Unfolded Protein Response Pathways in Chlamydia Pathogenesis

The molecular mechanism of induction of unfolded protein response by Chlamydia

Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling events

Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophages

Chlamydia pneumoniae infected macrophages exhibit enhanced plasma membrane fluidity and show increased adherence to endothelial cells

Chlamydia pneumoniae survival in macrophages is regulated by free Ca2+ dependent reactive nitrogen and oxygen species

Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cells

Effective macrophage redox defense against Chlamydia pneumoniae depends on L-type Ca2+ channel activation

Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophages

Generation of reactive oxygen species and formation of membrane lipid peroxides in cells infected with Chlamydia trachomatis

Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genes

Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibility

Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: Evidence of the effect of redox balance on host–pathogen relationship

17 beta-estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophages

Association of Chlamydia trachomatis antibodies with genital contact disease in women in Benin City, Nigeria

Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelae

Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca super(2+) dependent manner in human macrophages

Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca 2+ dependent manner in human macrophages

Free intracellular Ca 2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophages

17β-Estradiol induces L-type Ca2+ channel activation and regulates redox function in macrophages

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