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Psoriasis: A STAT3-Centric View

International journal of molecular sciences, 2018-01, Vol.19 (1), p.171

2018

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Nucleus, Mitochondrion, or Reticulum? STAT3 à La Carte

International journal of molecular sciences, 2018-09, Vol.19 (9), p.2820

2018

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The dark-side of the outside: how extracellular heat shock proteins promote cancer

Cellular and molecular life sciences : CMLS, 2021-05, Vol.78 (9), p.4069-4083

2021

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Adenoviral vaccine targeting multiple neoantigens as strategy to eradicate large tumors combined with checkpoint blockade

Nature communications, 2019-06, Vol.10 (1), p.2688-12, Article 2688

2019

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Toll-like receptor 2 promotes breast cancer progression and resistance to chemotherapy

Oncoimmunology, 2022-12, Vol.11 (1), p.2086752-2086752

2022

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The role of tumor microenvironment in drug resistance: emerging technologies to unravel breast cancer heterogeneity

Frontiers in oncology, 2023-05, Vol.13, p.1170264-1170264

2023

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The immediate early genes Fos and Egr1 become STAT1 transcriptional targets in the absence of STAT3

FEBS letters, 2011-08, Vol.585 (15), p.2455-2460

2011

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Shedding Light on NF-κB Functions in Cellular Organelles

Frontiers in cell and developmental biology, 2022-05, Vol.10, p.841646-841646

2022

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Vector Aided Microenvironment programming (VAMP): reprogramming the TME with MVA virus expressing IL-12 for effective antitumor activity

Journal for immunotherapy of cancer, 2023-04, Vol.11 (4), p.e006718

2023

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STAT3 in cancer: A double edged sword

Cytokine (Philadelphia, Pa.), 2017-10, Vol.98, p.42-50

2017

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Liver-Specific siRNA-Mediated Stat3 or C3 Knockdown Improves the Outcome of Experimental Autoimmune Myocarditis

Molecular therapy. Methods & clinical development, 2020-09, Vol.18, p.62-72

2020

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STAT3 localizes to the ER, acting as a gatekeeper for ER-mitochondrion Ca2+ fluxes and apoptotic responses

Cell death and differentiation, 2019-05, Vol.26 (5), p.932-942

2019

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The Microrna-143/145 Cluster in Tumors: A Matter of Where and When

Cancers, 2020-03, Vol.12 (3), p.708

2020

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STAT3 induces breast cancer growth via ANGPTL4, MMP13 and STC1 secretion by cancer associated fibroblasts

Oncogene, 2022-03, Vol.41 (10), p.1456-1467

2022

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Targeting the Extracellular HSP90 Co-Chaperone Morgana Inhibits Cancer Cell Migration and Promotes Anticancer Immunity

Cancer research (Chicago, Ill.), 2021-09, Vol.81 (18), p.4794-4807

2021

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MicroRNAs-143 and -145 induce epithelial to mesenchymal transition and modulate the expression of junction proteins

Cell death and differentiation, 2017-10, Vol.24 (10), p.1750-1760

2017

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STAT1 and STAT3 in tumorigenesis: A matter of balance

JAK-STAT, 2012-04, Vol.1 (2), p.65-72

2012

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An ILK/STAT3 pathway controls glioblastoma stem cell plasticity

Developmental cell, 2024-09

2024

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SP1 and STAT3 Functionally Synergize to Induce the RhoU Small GTPase and a Subclass of Non-canonical WNT Responsive Genes Correlating with Poor Prognosis in Breast Cancer

Cancers, 2019-01, Vol.11 (1), p.101

2019

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ETS-related gene (ERG) undermines genome stability in mouse prostate progenitors via Gsk3β dependent Nkx3.1 degradation

Cancer letters, 2022-05, Vol.534, p.215612-215612, Article 215612

2022

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Tumor Burden Dictates the Neoantigen Features Required to Generate an Effective Cancer Vaccine

Cancer immunology research, 2024-04, Vol.12 (4), p.440-452

2024

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Constitutive STAT3 activation in epidermal keratinocytes enhances cell clonogenicity and favours spontaneous immortalization by opposing differentiation and senescence checkpoints

Experimental dermatology, 2015-01, Vol.24 (1), p.29-34

2015

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STAT3 localizes to the ER, acting as a gatekeeper for ER-mitochondrion Ca 2+ fluxes and apoptotic responses

Cell death and differentiation, 2019-05, Vol.26 (5), p.932

2019

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Psoriasis: A STAT3-Centric View

Nucleus, Mitochondrion, or Reticulum? STAT3 à La Carte

The dark-side of the outside: how extracellular heat shock proteins promote cancer

Adenoviral vaccine targeting multiple neoantigens as strategy to eradicate large tumors combined with checkpoint blockade

Toll-like receptor 2 promotes breast cancer progression and resistance to chemotherapy

The role of tumor microenvironment in drug resistance: emerging technologies to unravel breast cancer heterogeneity

The immediate early genes Fos and Egr1 become STAT1 transcriptional targets in the absence of STAT3

Shedding Light on NF-κB Functions in Cellular Organelles

Vector Aided Microenvironment programming (VAMP): reprogramming the TME with MVA virus expressing IL-12 for effective antitumor activity

STAT3 in cancer: A double edged sword

Liver-Specific siRNA-Mediated Stat3 or C3 Knockdown Improves the Outcome of Experimental Autoimmune Myocarditis

STAT3 localizes to the ER, acting as a gatekeeper for ER-mitochondrion Ca2+ fluxes and apoptotic responses

The Microrna-143/145 Cluster in Tumors: A Matter of Where and When

STAT3 induces breast cancer growth via ANGPTL4, MMP13 and STC1 secretion by cancer associated fibroblasts

Targeting the Extracellular HSP90 Co-Chaperone Morgana Inhibits Cancer Cell Migration and Promotes Anticancer Immunity

MicroRNAs-143 and -145 induce epithelial to mesenchymal transition and modulate the expression of junction proteins

STAT1 and STAT3 in tumorigenesis: A matter of balance

An ILK/STAT3 pathway controls glioblastoma stem cell plasticity

SP1 and STAT3 Functionally Synergize to Induce the RhoU Small GTPase and a Subclass of Non-canonical WNT Responsive Genes Correlating with Poor Prognosis in Breast Cancer

ETS-related gene (ERG) undermines genome stability in mouse prostate progenitors via Gsk3β dependent Nkx3.1 degradation

Tumor Burden Dictates the Neoantigen Features Required to Generate an Effective Cancer Vaccine

Constitutive STAT3 activation in epidermal keratinocytes enhances cell clonogenicity and favours spontaneous immortalization by opposing differentiation and senescence checkpoints

RICTOR/mTORC2 downregulation in BRAFV600E melanoma cells promotes resistance to BRAF/MEK inhibition

RICTOR/mTORC2 downregulation in BRAF V600E melanoma cells promotes resistance to BRAF/MEK inhibition

STAT3 localizes to the ER, acting as a gatekeeper for ER-mitochondrion Ca 2+ fluxes and apoptotic responses

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