UB Paderborn / Katalog / Suche / in CDI

Post-cardiac arrest hyperoxia and mitochondrial function

Resuscitation, 2011-12, Vol.82, p.S48-S51

2011

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Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion

Journal of molecular and cellular cardiology, 2014-07, Vol.72, p.138-145

2014

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HDAC1 localizes to the mitochondria of cardiac myocytes and contributes to early cardiac reperfusion injury

Journal of molecular and cellular cardiology, 2018-01, Vol.114, p.309-319

2018

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Sivelestat Attenuates Myocardial Reperfusion Injury during Brief Low Flow Postischemic Infusion

Oxidative medicine and cellular longevity, 2013-01, Vol.2013, p.279847-9

2013

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Induction and Assessment of Ischemia-reperfusion Injury in Langendorff-perfused Rat Hearts

Journal of visualized experiments, 2015-07 (101), p.e52908-e52908

2015

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Oxygen requirement during cardiopulmonary resuscitation (CPR) to effect return of spontaneous circulation

Resuscitation, 2009-08, Vol.80 (8), p.951-955

2009

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Histone Deacetylases Exert Class-Specific Roles in Conditioning the Brain and Heart Against Acute Ischemic Injury

Frontiers in neurology, 2015, Vol.6, p.145-145

2015

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Preservation of mitochondrial function with cardiopulmonary resuscitation in prolonged cardiac arrest in rats

Journal of molecular and cellular cardiology, 2009-12, Vol.47 (6), p.789-797

2009

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Hyperoxemic reperfusion after prolonged cardiac arrest in a rat cardiopulmonary bypass resuscitation model

Resuscitation, 2013-01, Vol.84 (1), p.114-120

2013

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Cardiovascular response to epinephrine varies with increasing duration of cardiac arrest

Resuscitation, 2008-04, Vol.77 (1), p.101-110

2008

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Measurement of hydrogen peroxide and oxidant stress in a recirculating whole blood-perfused rat heart model

Resuscitation, 2011-02, Vol.82 (2), p.222-227

2011

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O2 delivery and redox state are determinants of compartment-specific reactive O2 species in myocardial reperfusion

American journal of physiology. Heart and circulatory physiology, 2007-01, Vol.292 (1), p.H109-H116

2007

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Inhibition of peroxynitrite precursors, NO and O2 , at the onset of reperfusion improves myocardial recovery

Resuscitation, 2007-09, Vol.74 (3), p.508-515

2007

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Abstract 405: Inhibition of Class I Histone Deacetylases at Reperfusion Attenuates Ischemia-reperfusion Injury and Modifies Mitochondrial Acetylation

Circulation research, 2015-07, Vol.117 (suppl_1)

2015

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Abstract 443: Class I Histone Deacetylases Mediate Cardiac I/R Injury and Modify the Mitochondrial Acetylome

Circulation research, 2016-07, Vol.119 (suppl_1)

2016

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Abstract 363: Class I HDAC Inhibition Causes Selective Activation of Pro-Survival Kinases at Reperfusion in Post-Ischemic Hearts

Circulation research, 2014-07, Vol.115 (suppl_1)

2014

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Abstract 222: Inhibition Of Class I But Not Class IIb Lysine Deacetylases Is Responsible For Cardioprotection From Ischemia-Reperfusion

Circulation research, 2013-08, Vol.113 (suppl_1)

2013

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Induction and Assessment of Ischemia-reperfusion Injury in Langendorff-perfused Rat Hearts

Journal of visualized experiments, 2015-07 (101)

2015

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O 2 delivery and redox state are determinants of compartment-specific reactive O 2 species in myocardial reperfusion

American journal of physiology. Heart and circulatory physiology, 2007-01, Vol.292 (1), p.H109-H116

2007

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Abstract 2631: Reactive Oxygen Species (ROS) Generation and Recovery Of Myocardial Function After Low Flow Reperfusion In The Whole Heart

Circulation (New York, N.Y.), 2007-10, Vol.116 (suppl_16)

2007

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Inhibition of peroxynitrite precursors, NO and O 2, at the onset of reperfusion improves myocardial recovery

Resuscitation, 2007-09, Vol.74 (3), p.508-515

2007

Volltextzugriff (PDF)

O2 delivery and redox state are determinants of compartment-specific reactive O2 species in myocardial reperfusion

American journal of physiology. Heart and circulatory physiology, 2007, Vol.61 (1)

2007

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O2 delivery and redox state are determinants of compartment-specific reactive 02 species in myocardial reperfusion

American journal of physiology. Heart and circulatory physiology, 2007-01, Vol.292 (1), p.H109

2007

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The Role of Reactive Oxygen Species in Post-Ischemic Low Flow in the Myocardium

Aune, Sverre E

2012

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Detection of Nonadherence to Antihypertensive Treatment by Measurements of Serum Drug Concentrations

Hypertension (Dallas, Tex. 1979), 2021-09, Vol.78 (3), p.617-628

2021

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Post-cardiac arrest hyperoxia and mitochondrial function

Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion

HDAC1 localizes to the mitochondria of cardiac myocytes and contributes to early cardiac reperfusion injury

Sivelestat Attenuates Myocardial Reperfusion Injury during Brief Low Flow Postischemic Infusion

Induction and Assessment of Ischemia-reperfusion Injury in Langendorff-perfused Rat Hearts

Oxygen requirement during cardiopulmonary resuscitation (CPR) to effect return of spontaneous circulation

Histone Deacetylases Exert Class-Specific Roles in Conditioning the Brain and Heart Against Acute Ischemic Injury

Preservation of mitochondrial function with cardiopulmonary resuscitation in prolonged cardiac arrest in rats

Hyperoxemic reperfusion after prolonged cardiac arrest in a rat cardiopulmonary bypass resuscitation model

Cardiovascular response to epinephrine varies with increasing duration of cardiac arrest

Measurement of hydrogen peroxide and oxidant stress in a recirculating whole blood-perfused rat heart model

O2 delivery and redox state are determinants of compartment-specific reactive O2 species in myocardial reperfusion

Inhibition of peroxynitrite precursors, NO and O2 , at the onset of reperfusion improves myocardial recovery

Abstract 405: Inhibition of Class I Histone Deacetylases at Reperfusion Attenuates Ischemia-reperfusion Injury and Modifies Mitochondrial Acetylation

Abstract 443: Class I Histone Deacetylases Mediate Cardiac I/R Injury and Modify the Mitochondrial Acetylome

Abstract 363: Class I HDAC Inhibition Causes Selective Activation of Pro-Survival Kinases at Reperfusion in Post-Ischemic Hearts

Abstract 222: Inhibition Of Class I But Not Class IIb Lysine Deacetylases Is Responsible For Cardioprotection From Ischemia-Reperfusion

Induction and Assessment of Ischemia-reperfusion Injury in Langendorff-perfused Rat Hearts

O 2 delivery and redox state are determinants of compartment-specific reactive O 2 species in myocardial reperfusion

Abstract 2631: Reactive Oxygen Species (ROS) Generation and Recovery Of Myocardial Function After Low Flow Reperfusion In The Whole Heart

Inhibition of peroxynitrite precursors, NO and O 2, at the onset of reperfusion improves myocardial recovery

O2 delivery and redox state are determinants of compartment-specific reactive O2 species in myocardial reperfusion

O2 delivery and redox state are determinants of compartment-specific reactive 02 species in myocardial reperfusion

The Role of Reactive Oxygen Species in Post-Ischemic Low Flow in the Myocardium

Detection of Nonadherence to Antihypertensive Treatment by Measurements of Serum Drug Concentrations

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