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Arp2/3 Is Critical for Lamellipodia and Response to Extracellular Matrix Cues but Is Dispensable for Chemotaxis

Cell, 2012-03, Vol.148 (5), p.973-987

2012

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Self-sorting of nonmuscle myosins IIA and IIB polarizes the cytoskeleton and modulates cell motility

The Journal of cell biology, 2017-09, Vol.216 (9), p.2877-2889

2017

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Tumor Cell-Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression

Cancer discovery, 2016-05, Vol.6 (5), p.516-531

2016

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F-actin bundles direct the initiation and orientation of lamellipodia through adhesion-based signaling

The Journal of cell biology, 2015-02, Vol.208 (4), p.443-455

2015

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Mesenchymal Chemotaxis Requires Selective Inactivation of Myosin II at the Leading Edge via a Noncanonical PLCγ/PKCα Pathway

Developmental cell, 2014-12, Vol.31 (6), p.747-760

2014

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Arp2/3 Complex Is Required for Macrophage Integrin Functions but Is Dispensable for FcR Phagocytosis and In Vivo Motility

Developmental cell, 2017-09, Vol.42 (5), p.498-513.e6

2017

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Cells lay their own tracks - optogenetic Cdc42 activation stimulates fibronectin deposition supporting directed migration

Journal of cell science, 2017-09, Vol.130 (18), p.2971-2983

2017

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Lamellipodia are crucial for haptotactic sensing and response

Journal of cell science, 2016-06, Vol.129 (12), p.2329-2342

2016

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GMFβ controls branched actin content and lamellipodial retraction in fibroblasts

The Journal of cell biology, 2015-06, Vol.209 (6), p.803-812

2015

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LKB1 loss in melanoma disrupts directional migration toward extracellular matrix cues

The Journal of cell biology, 2014-10, Vol.207 (2), p.299-315

2014

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Loss of Arp2/3 induces an NF-κB-dependent, nonautonomous effect on chemotactic signaling

The Journal of cell biology, 2013-12, Vol.203 (6), p.907-916

2013

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Quantitative analysis of B-lymphocyte migration directed by CXCL13

Integrative biology (Cambridge), 2016-08, Vol.8 (8), p.894-903

2016

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Emergent spatiotemporal dynamics of the actomyosin network in the presence of chemical gradients

Integrative biology (Cambridge), 2019-06, Vol.11 (6), p.280-292

2019

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Two-Dimensional Manipulation and Orientation of Actin−Myosin Systems with Dielectrophoresis

Nano letters, 2003-04, Vol.3 (4), p.431-437

2003

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LKB1 loss in melanoma disrupts directional migration toward extracellular matrix cues

The Journal of experimental medicine, 2014-11, Vol.211 (12), p.21112-21112OIA68

2014

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GMF[Beta] controls branched actin content and lamellipodial retraction in fibroblasts

The Journal of cell biology, 2015-06, Vol.209 (6), p.803

2015

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Quantitative analysis of B-lymphocyte migration directed by CXCL13Electronic supplementary information (ESI) available. See DOI: 10.1039/c6ib00128a

2016-08

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Mesenchymal chemotaxis requires selective inactivation of Myosin II at the leading edge via a non-canonical PLCγ/PKCα pathway

Developmental cell, 2014-12, Vol.31 (6), p.747-760

2014

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Abstract 1988: MenaINV interaction with α5β1 promotes tumor cell invasion in response to gradients of growth factors and fibronectin

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.1988-1988

2014

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Loss of Arp2/3 induces an NF-[kappa]B-dependent, nonautonomous effect on chemotactic signaling

The Journal of cell biology, 2013-12, Vol.203 (6), p.907

2013

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Lamellipodia are critical for haptotactic sensing and response

Journal of cell science, 2016-01

2016

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Abstract C41: MenaINV dysregulation of receptor tyrosine kinase signaling

Cancer research (Chicago, Ill.), 2013-02, Vol.73 (3_Supplement), p.C41-C41

2013

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Abstract C14: Loss of haptotaxis facilitates invasion in LKB1-deficient melanoma

Cancer research (Chicago, Ill.), 2013-02, Vol.73 (3_Supplement), p.C14-C14

2013

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Arp2/3 complex is critical for lamellipodia and organization of cell-matrix adhesion but dispensable for fibroblast chemotaxis

Cell, 2012-03, Vol.148 (5), p.973-987

2012

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Forces in cytoskeletal systems

Asokan, Sreeja B

2006

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Cell Line
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Arp2/3 Is Critical for Lamellipodia and Response to Extracellular Matrix Cues but Is Dispensable for Chemotaxis

Self-sorting of nonmuscle myosins IIA and IIB polarizes the cytoskeleton and modulates cell motility

Tumor Cell-Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression

F-actin bundles direct the initiation and orientation of lamellipodia through adhesion-based signaling

Mesenchymal Chemotaxis Requires Selective Inactivation of Myosin II at the Leading Edge via a Noncanonical PLCγ/PKCα Pathway

Arp2/3 Complex Is Required for Macrophage Integrin Functions but Is Dispensable for FcR Phagocytosis and In Vivo Motility

Cells lay their own tracks - optogenetic Cdc42 activation stimulates fibronectin deposition supporting directed migration

Lamellipodia are crucial for haptotactic sensing and response

GMFβ controls branched actin content and lamellipodial retraction in fibroblasts

LKB1 loss in melanoma disrupts directional migration toward extracellular matrix cues

Loss of Arp2/3 induces an NF-κB-dependent, nonautonomous effect on chemotactic signaling

Quantitative analysis of B-lymphocyte migration directed by CXCL13

Emergent spatiotemporal dynamics of the actomyosin network in the presence of chemical gradients

Two-Dimensional Manipulation and Orientation of Actin−Myosin Systems with Dielectrophoresis

LKB1 loss in melanoma disrupts directional migration toward extracellular matrix cues

GMF[Beta] controls branched actin content and lamellipodial retraction in fibroblasts

Quantitative analysis of B-lymphocyte migration directed by CXCL13Electronic supplementary information (ESI) available. See DOI: 10.1039/c6ib00128a

Mesenchymal chemotaxis requires selective inactivation of Myosin II at the leading edge via a non-canonical PLCγ/PKCα pathway

Abstract 1988: MenaINV interaction with α5β1 promotes tumor cell invasion in response to gradients of growth factors and fibronectin

Loss of Arp2/3 induces an NF-[kappa]B-dependent, nonautonomous effect on chemotactic signaling

Lamellipodia are critical for haptotactic sensing and response

Abstract C41: MenaINV dysregulation of receptor tyrosine kinase signaling

Abstract C14: Loss of haptotaxis facilitates invasion in LKB1-deficient melanoma

Arp2/3 complex is critical for lamellipodia and organization of cell-matrix adhesion but dispensable for fibroblast chemotaxis

Forces in cytoskeletal systems

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