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A new phosphorylation site in cardiac L-type Ca2+ channels (Cav1.2) responsible for its cAMP-mediated modulation

American Journal of Physiology: Cell Physiology, 2014-12, Vol.307 (11), p.C999-C1009

2014

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Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca2+ channels

American Journal of Physiology: Cell Physiology, 2016-05, Vol.310 (10), p.C773-C779

2016

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Adenosine triphosphate regulates the activity of guinea pig Cav1.2 channel by direct binding to the channel in a dose-dependent manner

American Journal of Physiology: Cell Physiology, 2014-05, Vol.306 (9), p.C856-C863

2014

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Mechanisms underlying the modulation of L-type Ca2+ channel by hydrogen peroxide in guinea pig ventricular myocytes

The journal of physiological sciences, 2013-11, Vol.63 (6), p.419-426

2013

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Calmodulin Kinase II Activation Is Required for the Maintenance of Basal Activity of L-Type Ca2+ Channels in Guinea-Pig Ventricular Myocytes

Journal of Pharmacological Sciences, 2008, Vol.108(3), pp.290-300

2008

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The Distinct Roles of Calmodulin and Calmodulin Kinase II in the Reversal of Run-Down of L-Type Ca2+ Channels in Guinea-Pig Ventricular Myocytes

Journal of Pharmacological Sciences, 2009, Vol.111(4), pp.416-425

2009

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Tetrodotoxin-resistant Na+ channels in human neuroblastoma cells are encoded by new variants of Nav1.5/SCN5A

The European journal of neuroscience, 2005-08, Vol.22 (4), p.793-801

2005

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Analysis of four novel variants of Nav1.5/SCN5A cloned from the brain

Neuroscience research, 2009-08, Vol.64 (4), p.339-347

2009

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Cloning and expression of the two new variants of Nav1.5/SCN5A in rat brain

Molecular and cellular biochemistry, 2012-06, Vol.365 (1-2), p.139-148

2012

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Calmodulin reverses rundown of L-type Ca2+ channels in guinea pig ventricular myocytes

American Journal of Physiology: Cell Physiology, 2004-12, Vol.287 (6), p.C1717

2004

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Distinct Roles of CaM and Ca2+/CaM-Dependent Protein Kinase II in Ca2+-Dependent Facilitation and Inactivation of Cardiac L-Type Ca2+ Channels

The Journal of Physiological Sciences, 2007, Vol.57(3), pp.167-173

2007

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Calpastatin Domain L Is Involved in the Regulation of L-Type Ca2+ Channels in Guinea Pig Cardiac Myocytes

Biochemical and biophysical research communications, 2000-12, Vol.279 (3), p.756-761

2000

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Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca 2+ channels

American Journal of Physiology: Cell Physiology, 2016-05, Vol.310 (10), p.C773-C779

2016

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Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca^sup 2+^ channels

American Journal of Physiology: Cell Physiology, 2016-05, Vol.310 (10), p.C773

2016

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Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca super( 2+) channels

American Journal of Physiology: Cell Physiology, 2016-05, Vol.310 (10), p.C773-C773

2016

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A new phosphorylation site in cardiac L-type Ca 2+ channels (Cav1.2) responsible for its cAMP-mediated modulation

American Journal of Physiology: Cell Physiology, 2014-12, Vol.307 (11), p.C999-C1009

2014

Volltextzugriff (PDF)

A new phosphorylation site in cardiac L-type Ca^sup 2+^ channels (Cav1.2) responsible for its cAMP-mediated modulation

American Journal of Physiology: Cell Physiology, 2014-12, Vol.307 (11), p.C999

2014

Volltextzugriff (PDF)

A new phosphorylation site in cardiac L-type Ca super( 2+) channels (Cav1.2) responsible for its cAMP-mediated modulation

American Journal of Physiology: Cell Physiology, 2014-12, Vol.307 (11), p.C999-C999

2014

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A cytoplasmic factor, calpastatin and ATP together reverse run-down of Ca2+ channel activity in guinea-pig heart

The Journal of physiology, 1999-02, Vol.514 (3), p.687-699

1999

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S2D-22-4 Regulation of L-type (Cav1.2) Ca2+ channels by calmodulin and ATP

Journal of Pharmacological Sciences, 2014-02, Vol.124 (suppl), p.45-45

2014

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Cloning and Expression of the Ca2+ Channel α1c and β2a Subunits from Guinea Pig Heart

Journal of biochemistry (Tokyo), 1999-04, Vol.125 (4), p.750-759

1999

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Calmodulin reverses rundown of L-type Ca(2+) channels in guinea pig ventricular myocytes

American Journal of Physiology: Cell Physiology, 2004-12, Vol.287 (6), p.C1717

2004

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Run-down of the cardiac L-type Ca 2+ channel: partial restoration of channel activity in cell-free patches by calpastatin

Pflügers Archiv, 1998-01, Vol.435 (3), p.344-349

1998

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Run-Down of L-Type Ca2+Channels Occurs on the α1Subunit

Biochemical and biophysical research communications, 1998-06, Vol.247 (3), p.844-850

1998

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Run-down of the cardiac Ca 2+ channel: characterization and restoration of channel activity by cytoplasmic factors

Pflügers Archiv, 1997-02, Vol.433 (5), p.547-556

1997

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A new phosphorylation site in cardiac L-type Ca2+ channels (Cav1.2) responsible for its cAMP-mediated modulation

Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca2+ channels

Adenosine triphosphate regulates the activity of guinea pig Cav1.2 channel by direct binding to the channel in a dose-dependent manner

Mechanisms underlying the modulation of L-type Ca2+ channel by hydrogen peroxide in guinea pig ventricular myocytes

Calmodulin Kinase II Activation Is Required for the Maintenance of Basal Activity of L-Type Ca2+ Channels in Guinea-Pig Ventricular Myocytes

The Distinct Roles of Calmodulin and Calmodulin Kinase II in the Reversal of Run-Down of L-Type Ca2+ Channels in Guinea-Pig Ventricular Myocytes

Tetrodotoxin-resistant Na+ channels in human neuroblastoma cells are encoded by new variants of Nav1.5/SCN5A

Analysis of four novel variants of Nav1.5/SCN5A cloned from the brain

Cloning and expression of the two new variants of Nav1.5/SCN5A in rat brain

Calmodulin reverses rundown of L-type Ca2+ channels in guinea pig ventricular myocytes

Distinct Roles of CaM and Ca2+/CaM-Dependent Protein Kinase II in Ca2+-Dependent Facilitation and Inactivation of Cardiac L-Type Ca2+ Channels

Calpastatin Domain L Is Involved in the Regulation of L-Type Ca2+ Channels in Guinea Pig Cardiac Myocytes

Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca 2+ channels

Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca^sup 2+^ channels

Role of protein phosphatases in the run down of guinea pig cardiac Cav1.2 Ca super( 2+) channels

A new phosphorylation site in cardiac L-type Ca 2+ channels (Cav1.2) responsible for its cAMP-mediated modulation

A new phosphorylation site in cardiac L-type Ca^sup 2+^ channels (Cav1.2) responsible for its cAMP-mediated modulation

A new phosphorylation site in cardiac L-type Ca super( 2+) channels (Cav1.2) responsible for its cAMP-mediated modulation

A cytoplasmic factor, calpastatin and ATP together reverse run-down of Ca2+ channel activity in guinea-pig heart

S2D-22-4 Regulation of L-type (Cav1.2) Ca2+ channels by calmodulin and ATP

Cloning and Expression of the Ca2+ Channel α1c and β2a Subunits from Guinea Pig Heart

Calmodulin reverses rundown of L-type Ca(2+) channels in guinea pig ventricular myocytes

Run-down of the cardiac L-type Ca 2+ channel: partial restoration of channel activity in cell-free patches by calpastatin

Run-Down of L-Type Ca2+Channels Occurs on the α1Subunit

Run-down of the cardiac Ca 2+ channel: characterization and restoration of channel activity by cytoplasmic factors

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