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Disruption of PC1/3 Expression in Mice Causes Dwarfism and Multiple Neuroendocrine Peptide Processing Defects

Proceedings of the National Academy of Sciences - PNAS, 2002-08, Vol.99 (16), p.10293-10298

2002

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Biological Processing of the Cocaine and Amphetamine-regulated Transcript Precursors by Prohormone Convertases, PC2 and PC1/3

The Journal of biological chemistry, 2003-04, Vol.278 (17), p.15007-15014

2003

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SOX2 expression in prostate cancer drives resistance to nuclear hormone receptor signaling inhibition through the WEE1/CDK1 signaling axis

Cancer letters, 2023-07, Vol.565, p.216209-216209, Article 216209

2023

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GSK-3 inactivation or depletion promotes β-cell replication via down regulation of the CDK inhibitor, p27 (Kip1)

Islets, 2011-01, Vol.3 (1), p.21-34

2011

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The Long-Lasting Rodenticide Brodifacoum Induces Neuropathology in Adult Male Rats

Toxicological sciences, 2017-09, Vol.159 (1), p.224-237

2017

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Colony-stimulating factor-1 receptor utilizes multiple signaling pathways to induce cyclin D2 expression

Molecular biology of the cell, 2000-11, Vol.11 (11), p.3835-3848

2000

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No mantle formation in rodent islets—The prototype of islet revisited

Diabetes research and clinical practice, 2009-09, Vol.85 (3), p.252-257

2009

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The negative cell cycle regulators, p27(Kip1), p18(Ink4c), and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β-cells and restrict their ability to proliferate

Islets, 2013-07, Vol.5 (4), p.156-169

2013

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The negative cell cycle regulators, p27 Kip1 , p18 Ink4c , and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β -cells and restrict their ability to proliferate

Islets, 2013-07, Vol.5 (4), p.156-169

2013

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The negative cell cycle regulators, p27Kip1, p18Ink4c, and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β-cells and restrict their ability to proliferate

Islets, 2013-07, Vol.5 (4), p.156-169

2013

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Processing of cocaine- and amphetamine-regulated transcript (CART) precursor proteins by prohormone convertases (PCs) and its implications

Peptides (New York, N.Y. : 1980), 2006-08, Vol.27 (8), p.1919-1925

2006

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Prohormone convertases 1/3 and 2 together orchestrate the site-specific cleavages of progastrin to release gastrin-34 and gastrin-17

Biochemical journal, 2008-10, Vol.415 (1), p.35-43

2008

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GSK-3 inactivation or depletion promotes β-cell replication via downregulation of the CDK inhibitor, p27 (Kip1)

Islets, 2011-01, Vol.3 (1), p.21-34

2011

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Processing of cocaine- and amphetamine-regulated transcript (CART) precursor proteins by prohormone convertases (PCs) and its implications: CART Peptides

Peptides (New York, N.Y. : 1980), 2006, Vol.27 (8), p.1919-1925

2006

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RNAi-mediated silencing of prohormone convertase (PC) 5/6 expression leads to impairment in processing of cocaine- and amphetamine-regulated transcript (CART) precursor

Biochemical journal, 2006-11, Vol.400 (1), p.209-215

2006

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Significance of Prohormone Convertase 2, PC2, Mediated Initial Cleavage at the Proglucagon Interdomain Site, Lys70-Arg71, to Generate Glucagon

Endocrinology (Philadelphia), 2005-02, Vol.146 (2), p.713-727

2005

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Altered proglucagon processing in an alpha-cell line derived from prohormone convertase 2 null mouse islets

The Journal of biological chemistry, 2004-07, Vol.279 (30), p.31068-31075

2004

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Furin and Prohormone Convertase 1/3 Are Major Convertases in the Processing of Mouse Pro-Growth Hormone-Releasing Hormone

Endocrinology (Philadelphia), 2004-04, Vol.145 (4), p.1961-1971

2004

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Altered Proglucagon Processing in an Î±-Cell Line Derived from Prohormone Convertase 2 Null Mouse Islets

The Journal of biological chemistry, 2004-07, Vol.279 (30), p.31068

2004

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HPV16 E6 Oncoprotein Stimulates the Transforming Growth Factor-β1 Promoter in Fibroblasts through a Specific GC-Rich Sequence

Virology (New York, N.Y.), 1997-02, Vol.228 (2), p.190-199

1997

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Cell cycle-independent induction of D1 and D2 cyclin expression, but not cyclin–Cdk complex formation or Rb phosphorylation, by IFNγ in macrophages

Biochimica et biophysica acta. Molecular cell research, 2000-06, Vol.1497 (1), p.135-147

2000

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Biological processing of the cocaine and amphetamine-regulated transcript precursors by prohormone convertases, PC2 and PC1/3

The Journal of biological chemistry, 2003-06, Vol.278 (26), p.24242

2003

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Gamma interferon induces expression of Mad1 gene in macrophage, which inhibits colony-stimulating factor-1-dependent mitogenesis

Journal of cellular biochemistry, 1999-02, Vol.72 (2), p.232-241

1999

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Disruption of PC1/3 Expression in Mice Causes Dwarfism and Multiple Neuroendocrine Peptide Processing Defects

Biological Processing of the Cocaine and Amphetamine-regulated Transcript Precursors by Prohormone Convertases, PC2 and PC1/3

SOX2 expression in prostate cancer drives resistance to nuclear hormone receptor signaling inhibition through the WEE1/CDK1 signaling axis

GSK-3 inactivation or depletion promotes β-cell replication via down regulation of the CDK inhibitor, p27 (Kip1)

The Long-Lasting Rodenticide Brodifacoum Induces Neuropathology in Adult Male Rats

Colony-stimulating factor-1 receptor utilizes multiple signaling pathways to induce cyclin D2 expression

No mantle formation in rodent islets—The prototype of islet revisited

The negative cell cycle regulators, p27(Kip1), p18(Ink4c), and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β-cells and restrict their ability to proliferate

The negative cell cycle regulators, p27 Kip1 , p18 Ink4c , and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β -cells and restrict their ability to proliferate

The negative cell cycle regulators, p27Kip1, p18Ink4c, and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β-cells and restrict their ability to proliferate

Processing of cocaine- and amphetamine-regulated transcript (CART) precursor proteins by prohormone convertases (PCs) and its implications

Prohormone convertases 1/3 and 2 together orchestrate the site-specific cleavages of progastrin to release gastrin-34 and gastrin-17

GSK-3 inactivation or depletion promotes β-cell replication via downregulation of the CDK inhibitor, p27 (Kip1)

Processing of cocaine- and amphetamine-regulated transcript (CART) precursor proteins by prohormone convertases (PCs) and its implications: CART Peptides

RNAi-mediated silencing of prohormone convertase (PC) 5/6 expression leads to impairment in processing of cocaine- and amphetamine-regulated transcript (CART) precursor

Significance of Prohormone Convertase 2, PC2, Mediated Initial Cleavage at the Proglucagon Interdomain Site, Lys70-Arg71, to Generate Glucagon

Altered proglucagon processing in an alpha-cell line derived from prohormone convertase 2 null mouse islets

Furin and Prohormone Convertase 1/3 Are Major Convertases in the Processing of Mouse Pro-Growth Hormone-Releasing Hormone

Altered Proglucagon Processing in an Î±-Cell Line Derived from Prohormone Convertase 2 Null Mouse Islets

HPV16 E6 Oncoprotein Stimulates the Transforming Growth Factor-β1 Promoter in Fibroblasts through a Specific GC-Rich Sequence

Cell cycle-independent induction of D1 and D2 cyclin expression, but not cyclin–Cdk complex formation or Rb phosphorylation, by IFNγ in macrophages

Biological processing of the cocaine and amphetamine-regulated transcript precursors by prohormone convertases, PC2 and PC1/3

Gamma interferon induces expression of Mad1 gene in macrophage, which inhibits colony-stimulating factor-1-dependent mitogenesis

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