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Correction to: Translation of curative therapy concepts with T cell and cytokine antibody combinations for type 1 diabetes reversal in the IDDM rat

Journal of molecular medicine (Berlin, Germany), 2021-12, Vol.99 (12), p.1831-1831

2021

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Translation of curative therapy concepts with T cell and cytokine antibody combinations for type 1 diabetes reversal in the IDDM rat

Journal of molecular medicine (Berlin, Germany), 2020-08, Vol.98 (8), p.1125-1137

2020

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A variable CD3⁺ T-cell frequency in peripheral blood lymphocytes associated with type 1 diabetes mellitus development in the LEW.1AR1-iddm rat

PloS one, 2013-05, Vol.8 (5), p.e64305-e64305

2013

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Diabetes Prevention by Immunomodulatory FTY720 Treatment in the LEW.1AR1-iddm Rat Despite Immune Cell Activation

Endocrinology (Philadelphia), 2010-08, Vol.151 (8), p.3555-3565

2010

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Rat Models of Human Type 1 Diabetes

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2128, p.69

2020

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Changes in immune cell frequencies in primary and secondary lymphatic organs of LEW.1AR1-iddm rats, a model of human type 1 diabetes compared to other MHC congenic LEW inbred strains

Immunologic research, 2018-08, Vol.66 (4), p.462-470

2018

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Islet infiltration, cytokine expression and beta cell death in the NOD mouse, BB rat, Komeda rat, LEW.1AR1-iddm rat and humans with type 1 diabetes

Diabetologia, 2014-03, Vol.57 (3), p.512-521

2014

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TNF-α Antibody Therapy in Combination With the T-Cell-Specific Antibody Anti-TCR Reverses the Diabetic Metabolic State in the LEW.1AR1-iddm Rat

Diabetes (New York, N.Y.), 2015-08, Vol.64 (8), p.2880-2891

2015

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TNF-[Alpha] Antibody Therapy in Combination With the T-Cell-Specific Antibody Anti-TCR Reverses the Diabetic Metabolic State in the LEW.1AR1-iddm Rat

Diabetes (New York, N.Y.), 2015-08, Vol.64 (8), p.2880

2015

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A novel Dock8 gene mutation confers diabetogenic susceptibility in the LEW.1AR1/Ztm-iddm rat, an animal model of human type 1 diabetes

Diabetologia, 2015-12, Vol.58 (12), p.2800-2809

2015

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Results, meta-analysis and a first evaluation of UNOxR, the urinary nitrate-to-nitrite molar ratio, as a measure of nitrite reabsorption in experimental and clinical settings

Amino acids, 2018-07, Vol.50 (7), p.799-821

2018

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Anti-TCR therapy combined with fingolimod for reversal of diabetic hyperglycemia by β cell regeneration in the LEW.1AR1-iddm rat model of type 1 diabetes

Journal of molecular medicine (Berlin, Germany), 2014-07, Vol.92 (7), p.743-755

2014

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Kinetics of CD8+ effector T cell responses and induced CD4+ regulatory T cell responses during Friend retrovirus infection

European journal of immunology, 2006-10, Vol.36 (10), p.2658-2670

2006

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A Variable CD3.sup.+ T-Cell Frequency in Peripheral Blood Lymphocytes Associated with Type 1 Diabetes Mellitus Development in the LEW.1AR1-iddm Rat

PloS one, 2013-05, Vol.8 (5), p.e64305

2013

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Anti-TCR therapy combined with fingolimod for reversal of diabetic hyperglycemia by [beta] cell regeneration in the LEW.1AR1-iddm rat model of type 1 diabetes

Journal of molecular medicine (Berlin, Germany), 2014-07, Vol.92 (7), p.743

2014

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Pattern of genes influenced by conditional expression of the transcription factors HNF6, HNF4alpha and HNF1beta in a pancreatic beta-cell line

Nucleic acids research, 2004-11, Vol.32 (19), p.e150

2004

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Results, meta-analysis and a first evaluation of U NOx R, the urinary nitrate-to-nitrite molar ratio, as a measure of nitrite reabsorption in experimental and clinical settings

Amino acids, 2018-07, Vol.50 (7), p.799

2018

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Tissue-specific transcription factor HNF4α inhibits cell proliferation and induces apoptosis in the pancreatic INS-1 β-cell line

Biological chemistry, 2007-01, Vol.388 (1), p.91-106

2007

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A Variable CD3+ T-Cell Frequency in Peripheral Blood Lymphocytes Associated with Type 1 Diabetes Mellitus Development in the LEW.1AR1-iddm Rat. e64305

PloS one, 2013-05, Vol.8 (5)

2013

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mutation of the LEW.1AR1-iddm rat maps to the telomeric end of rat chromosome 1

Mammalian genome, 2008-04, Vol.19 (4), p.292-297

2008

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Kinetics of CD8 super(+) effector T cell responses and induced CD4 super(+) regulatory T cell responses during Friend retrovirus infection

European Journal of Immunology, 2006-01, Vol.36 (10), p.2658-2670

2006

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Tissue-specific transcription factor HNF4I- inhibits cell proliferation and induces apoptosis in the pancreatic INS-1 I2-cell line

Biological chemistry, 2007-01, Vol.388 (1), p.91-106

2007

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Tissue-specific transcription factor HNF4alpha inhibits cell proliferation and induces apoptosis in the pancreatic INS-1 beta-cell line

Biological chemistry, 2007-01, Vol.388 (1), p.91-106

2007

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Effective treatment of retrovirus-induced suppression of antibody responses with CpG oligodeoxynucleotides

Journal of general virology, 2005-12, Vol.86 (12), p.3365-3368

2005

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Detektion von mRNA und Protein in situ

Gentechnische Methoden, 2012, p.327-350

2012

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Correction to: Translation of curative therapy concepts with T cell and cytokine antibody combinations for type 1 diabetes reversal in the IDDM rat

Translation of curative therapy concepts with T cell and cytokine antibody combinations for type 1 diabetes reversal in the IDDM rat

A variable CD3⁺ T-cell frequency in peripheral blood lymphocytes associated with type 1 diabetes mellitus development in the LEW.1AR1-iddm rat

Diabetes Prevention by Immunomodulatory FTY720 Treatment in the LEW.1AR1-iddm Rat Despite Immune Cell Activation

Rat Models of Human Type 1 Diabetes

Changes in immune cell frequencies in primary and secondary lymphatic organs of LEW.1AR1-iddm rats, a model of human type 1 diabetes compared to other MHC congenic LEW inbred strains

Islet infiltration, cytokine expression and beta cell death in the NOD mouse, BB rat, Komeda rat, LEW.1AR1-iddm rat and humans with type 1 diabetes

TNF-α Antibody Therapy in Combination With the T-Cell-Specific Antibody Anti-TCR Reverses the Diabetic Metabolic State in the LEW.1AR1-iddm Rat

TNF-[Alpha] Antibody Therapy in Combination With the T-Cell-Specific Antibody Anti-TCR Reverses the Diabetic Metabolic State in the LEW.1AR1-iddm Rat

A novel Dock8 gene mutation confers diabetogenic susceptibility in the LEW.1AR1/Ztm-iddm rat, an animal model of human type 1 diabetes

Results, meta-analysis and a first evaluation of UNOxR, the urinary nitrate-to-nitrite molar ratio, as a measure of nitrite reabsorption in experimental and clinical settings

Anti-TCR therapy combined with fingolimod for reversal of diabetic hyperglycemia by β cell regeneration in the LEW.1AR1-iddm rat model of type 1 diabetes

Kinetics of CD8+ effector T cell responses and induced CD4+ regulatory T cell responses during Friend retrovirus infection

A Variable CD3.sup.+ T-Cell Frequency in Peripheral Blood Lymphocytes Associated with Type 1 Diabetes Mellitus Development in the LEW.1AR1-iddm Rat

Anti-TCR therapy combined with fingolimod for reversal of diabetic hyperglycemia by [beta] cell regeneration in the LEW.1AR1-iddm rat model of type 1 diabetes

Pattern of genes influenced by conditional expression of the transcription factors HNF6, HNF4alpha and HNF1beta in a pancreatic beta-cell line

Results, meta-analysis and a first evaluation of U NOx R, the urinary nitrate-to-nitrite molar ratio, as a measure of nitrite reabsorption in experimental and clinical settings

Tissue-specific transcription factor HNF4α inhibits cell proliferation and induces apoptosis in the pancreatic INS-1 β-cell line

A Variable CD3+ T-Cell Frequency in Peripheral Blood Lymphocytes Associated with Type 1 Diabetes Mellitus Development in the LEW.1AR1-iddm Rat. e64305

mutation of the LEW.1AR1-iddm rat maps to the telomeric end of rat chromosome 1

Kinetics of CD8 super(+) effector T cell responses and induced CD4 super(+) regulatory T cell responses during Friend retrovirus infection

Tissue-specific transcription factor HNF4I- inhibits cell proliferation and induces apoptosis in the pancreatic INS-1 I2-cell line

Tissue-specific transcription factor HNF4alpha inhibits cell proliferation and induces apoptosis in the pancreatic INS-1 beta-cell line

Effective treatment of retrovirus-induced suppression of antibody responses with CpG oligodeoxynucleotides

Detektion von mRNA und Protein in situ

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