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George Washington Memorial Parkway

Public Roads, 2024-04, Vol.88 (1), p.22-26

Arispe, Eduardo

2024

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The New George Washington Memorial Parkway: United States Department of the Interior National Park Service: Major rehabilitation will help preserve the road's natural scenery and connect metropolitan areas more safely

Public roads, 2024-03, Vol.88 (1), p.22

Arispe, Eduardo

2024

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Alzheimer's β-Amyloid, Human Islet Amylin, and Prion Protein Fragment Evoke Intracellular Free Calcium Elevations by a Common Mechanism in a Hypothalamic GnRH Neuronal Cell Line

The Journal of biological chemistry, 2000-05, Vol.275 (19), p.14077-14083

2000

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AGENT ASSOCIATED WITH EARLY BLIGHT Alternaria alternata (FR.) KEISSLER IN THE Solanum lycopersicum L. TOMATO CROP IN MEXICO

Tropical and subtropical agroecosystems, 2021-01, Vol.24 (1)

2021

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Adolescent idiopathic scoliosis: is there a relationship between Risser staging and the proximal humerus ossification system?

Spine deformity, 2024-05, Vol.12 (3), p.629-633

2024

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Alzheimer Disease Amyloid β Protein Forms Calcium Channels in Bilayer Membranes: Blockade by Tromethamine and Aluminum

Proceedings of the National Academy of Sciences - PNAS, 1993-01, Vol.90 (2), p.567-571

1993

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Giant Multilevel Cation Channels Formed by Alzheimer Disease Amyloid β-Protein [AβP-(1-40)] in Bilayer Membranes

Proceedings of the National Academy of Sciences - PNAS, 1993-11, Vol.90 (22), p.10573-10577

1993

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Zn2+Interaction with Alzheimer Amyloid β Protein Calcium Channels

Proceedings of the National Academy of Sciences - PNAS, 1996-02, Vol.93 (4), p.1710-1715

1996

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beta-Amyloid Ca(2+)-channel hypothesis for neuronal death in Alzheimer disease

Molecular and cellular biochemistry, 1994-11, Vol.140 (2), p.119-125

1994

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Similarity in calcium channel activity of annexin V and matrix vesicles in planar lipid bilayers

Biophysical journal, 1996-10, Vol.71 (4), p.1764-1775

1996

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The Ability of Amyloid β-Protein [AβP (1-40)] to Form Ca2+ Channels Provides a Mechanism for Neuronal Death in Alzheimer's Disease

Annals of the New York Academy of Sciences, 1994-12, Vol.747 (1), p.256-266

1994

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Veinte años de investigación sobre HTLV-1 y sus complicaciones médicas en el Perú: Perspectivas generales

Acta médica peruana, 2010, Vol.27 (3), p.196-203

2010

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Ion channel hypothesis for Alzheimer amyloid peptide neurotoxicity

Cellular and molecular neurobiology, 1995-10, Vol.15 (5), p.513-526

1995

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A New Hypothesis for the Mechanism of Amyloid Toxicity, Based on the Calcium Channel Activity of Amyloid β Protein (AβP) in Phospholipid Bilayer Membranes

Annals of the New York Academy of Sciences, 1993-09, Vol.695 (1), p.165-168

1993

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Alzheimer's Î²-Amyloid, Human Islet Amylin, and Prion Protein Fragment Evoke Intracellular Free Calcium Elevations by a Common Mechanism in a Hypothalamic GnRH Neuronal Cell Line

The Journal of biological chemistry, 2000-05, Vol.275 (19), p.14077

2000

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Intrinsic Anion Channel Activity of the Recombinant First Nucleotide Binding Fold Domain of the Cystic Fibrosis Transmembrane Regulator Protein

Proceedings of the National Academy of Sciences - PNAS, 1992-03, Vol.89 (5), p.1539-1543

1992

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Zn2positive interaction with Alzheimer amyloid beta protein calcium channels

Proceedings of the National Academy of Sciences - PNAS, 1996-02, Vol.93 (4), p.1710

1996

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The Ability of Amyloid β‐Protein [AβP (1–40)] to Form Ca 2+ Channels Provides a Mechanism for Neuronal Death in Alzheimer's Disease

Annals of the New York Academy of Sciences, 1994-12, Vol.747 (1), p.256-266

1994

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β-Amyloid in Alzheimerʼs Disease: Therapeutic Implications

CNS drugs, 1994-07, Vol.2 (1), p.1-6

1994

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Giant multilevel cation channels formed by Alzheimer disease amyloid (beta)-protein {A-beta-P-(1-40)} in bilayer membranes

Proceedings of the National Academy of Sciences - PNAS, 1993-11, Vol.90 (22), p.10573

1993

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A New Hypothesis for the Mechanism of Amyloid Toxicity, Based on the Calcium Channel Activity of Amyloid I2 Protein (AI2P) in Phospholipid Bilayer Membranes

Annals of the New York Academy of Sciences, 1993-09, Vol.695 (1), p.165-168

1993

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Alzheimer disease amyloid (beta) protein forms calcium channels in bilayer membranes: Blockade by tromethamine and aluminum

Proceedings of the National Academy of Sciences - PNAS, 1993-01, Vol.90 (2), p.567

1993

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George Washington Memorial Parkway

The New George Washington Memorial Parkway: United States Department of the Interior National Park Service: Major rehabilitation will help preserve the road's natural scenery and connect metropolitan areas more safely

Alzheimer's β-Amyloid, Human Islet Amylin, and Prion Protein Fragment Evoke Intracellular Free Calcium Elevations by a Common Mechanism in a Hypothalamic GnRH Neuronal Cell Line

AGENT ASSOCIATED WITH EARLY BLIGHT Alternaria alternata (FR.) KEISSLER IN THE Solanum lycopersicum L. TOMATO CROP IN MEXICO

Adolescent idiopathic scoliosis: is there a relationship between Risser staging and the proximal humerus ossification system?

Alzheimer Disease Amyloid β Protein Forms Calcium Channels in Bilayer Membranes: Blockade by Tromethamine and Aluminum

Giant Multilevel Cation Channels Formed by Alzheimer Disease Amyloid β-Protein [AβP-(1-40)] in Bilayer Membranes

Zn2+Interaction with Alzheimer Amyloid β Protein Calcium Channels

Recomendaciones para la atención de pacientes con diabetes mellitus con factores de riesgo o enfermedad cardiovascular establecida y SARS-CoV-2

beta-Amyloid Ca(2+)-channel hypothesis for neuronal death in Alzheimer disease

Similarity in calcium channel activity of annexin V and matrix vesicles in planar lipid bilayers

The Ability of Amyloid β-Protein [AβP (1-40)] to Form Ca2+ Channels Provides a Mechanism for Neuronal Death in Alzheimer's Disease

Veinte años de investigación sobre HTLV-1 y sus complicaciones médicas en el Perú: Perspectivas generales

Ion channel hypothesis for Alzheimer amyloid peptide neurotoxicity

A New Hypothesis for the Mechanism of Amyloid Toxicity, Based on the Calcium Channel Activity of Amyloid β Protein (AβP) in Phospholipid Bilayer Membranes

Alzheimer's Î²-Amyloid, Human Islet Amylin, and Prion Protein Fragment Evoke Intracellular Free Calcium Elevations by a Common Mechanism in a Hypothalamic GnRH Neuronal Cell Line

Intrinsic Anion Channel Activity of the Recombinant First Nucleotide Binding Fold Domain of the Cystic Fibrosis Transmembrane Regulator Protein

Zn2positive interaction with Alzheimer amyloid beta protein calcium channels

Recomendaciones para la atención de pacientes con diabetes mellitus con factores de riesgo o enfermedad cardiovascular establecida y SARS-CoV-2

Recommendations for the care of patients with diabetes mellitus with risk factors or established cardiovascular disease and SARS-CoV-2mellitus con factores de riesgo o enfermedad cardiovascular establecida y SARS-CoV-2

The Ability of Amyloid β‐Protein [AβP (1–40)] to Form Ca 2+ Channels Provides a Mechanism for Neuronal Death in Alzheimer's Disease

β-Amyloid in Alzheimerʼs Disease: Therapeutic Implications

Giant multilevel cation channels formed by Alzheimer disease amyloid (beta)-protein {A-beta-P-(1-40)} in bilayer membranes

A New Hypothesis for the Mechanism of Amyloid Toxicity, Based on the Calcium Channel Activity of Amyloid I2 Protein (AI2P) in Phospholipid Bilayer Membranes

Alzheimer disease amyloid (beta) protein forms calcium channels in bilayer membranes: Blockade by tromethamine and aluminum

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