UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 196

Sortiert nach

Increased glycolytic substrate protection improves ischemic cardiac dysfunction and reduces injury

The American heart journal, 2000-02, Vol.139 (2), p.s107-s114

Apstein, Carl S

2000

Volltextzugriff (PDF)

Glucose-insulin-potassium for acute myocardial infarction: remarkable results from a new prospective, randomized trial

Circulation (New York, N.Y.), 1998-11, Vol.98 (21), p.2223-2226

Apstein, C S

1998

Volltextzugriff (PDF)

ATP synthesis during low-flow ischemia influence of increased glycolytic substrate

Circulation (New York, N.Y.), 2000-05, Vol.101 (17), p.2090-2096

2000

Volltextzugriff (PDF)

Increased rat cardiac angiotensin converting enzyme activity and mRNA expression in pressure overload left ventricular hypertrophy : effects on coronary resistance, contractility, and relaxation

The Journal of clinical investigation, 1990-12, Vol.86 (6), p.1913-1920

1990

Volltextzugriff (PDF)

Exercise and heart failure: A statement from the American Heart Association Committee on Exercise, Rehabilitation, and Prevention

Circulation (New York, N.Y.), 2003-03, Vol.107 (8), p.1210-1225

2003

Volltextzugriff (PDF)

Rapid expression of heat shock protein in the rabbit after brief cardiac ischemia

The Journal of clinical investigation, 1991, Vol.87 (1), p.139-147

1991

Volltextzugriff (PDF)

Glucose-insulin-potassium in acute myocardial infarction: the time has come for a large, prospective trial

Circulation (New York, N.Y.), 1997-08, Vol.96 (4), p.1074-1077

1997

Volltextzugriff (PDF)

Distribution and functional significance of cardiac angiotensin converting enzyme in hypertrophied rat hearts

Circulation (New York, N.Y.), 1993-04, Vol.87 (4), p.1328-1339

1993

Volltextzugriff (PDF)

Protective Effect of Increased Glycolytic Substrate Against Systolic and Diastolic Dysfunction and Increased Coronary Resistance From Prolonged Global: Underperfusion and Reperfusion in Isolated Rabbit Hearts Perfused With Erythrocyte Suspensions

Circulation research, 1991-02, Vol.68 (2), p.466-481

1991

Volltextzugriff (PDF)

Infusion of light chains from patients with cardiac amyloidosis causes diastolic dysfunction in isolated mouse hearts

Circulation (New York, N.Y.), 2001-10, Vol.104 (14), p.1594-1597

2001

Volltextzugriff (PDF)

Stress-shortening relations and myocardial blood flow in compensated and failing canine hearts with pressure-overload hypertrophy

Circulation (New York, N.Y.), 1989-04, Vol.79 (4), p.872-883

1989

Volltextzugriff (PDF)

Cell therapy attenuates deleterious ventricular remodeling and improves cardiac performance after myocardial infarction

Circulation (New York, N.Y.), 2001-04, Vol.103 (14), p.1920-1927

2001

Volltextzugriff (PDF)

Exaggerated Left Ventricular Dilation and Reduced Collagen Deposition After Myocardial Infarction in Mice Lacking Osteopontin

Circulation research, 2001-05, Vol.88 (10), p.1080-1087

2001

Volltextzugriff (PDF)

Angiotensin II receptor blockade attenuates the deleterious effects of exercise training on post-MI ventricular remodelling in rats

Cardiovascular research, 2000-04, Vol.46 (1), p.66-72

2000

Volltextzugriff (PDF)

Validation of different methods to compare isovolumic cardiac function in isolated hearts of varying sizes

American journal of physiology. Heart and circulatory physiology, 1997-01, Vol.272 (1), p.H501-H510

1997

Volltextzugriff (PDF)

Glucose-6-Phosphate Dehydrogenase Modulates Cytosolic Redox Status and Contractile Phenotype in Adult Cardiomyocytes

Circulation research, 2003-07, Vol.93 (2), p.1-8

2003

Volltextzugriff (PDF)

Increased myocardial dysfunction after ischemia-reperfusion in mice lacking glucose-6-phosphate dehydrogenase

Circulation (New York, N.Y.), 2004-02, Vol.109 (7), p.898-903

2004

Volltextzugriff (PDF)

Effects of angiotensin II on intracellular Ca2+ and pH in isolated beating rabbit hearts and myocytes loaded with the indicator indo-1

The Journal of physiology, 1994-10, Vol.480 (Pt 2), p.203-215

1994

Volltextzugriff (PDF)

Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart

American journal of physiology. Heart and circulatory physiology, 2000-07, Vol.279 (1), p.H422-H428

2000

Volltextzugriff (PDF)

Exacerbation of Left Ventricular Ischemic Diastolic Dysfunction by Pressure-Overload Hypertrophy: Modification by Specific Inhibition of Cardiac Angiotensin Converting Enzyme

Circulation research, 1992-05, Vol.70 (5), p.931-943

1992

Volltextzugriff (PDF)

Mice Lacking Inducible Nitric Oxide Synthase Have Improved Left Ventricular Contractile Function and Reduced Apoptotic Cell Death Late After Myocardial Infarction

Circulation research, 2001-08, Vol.89 (4), p.351-356

2001

Volltextzugriff (PDF)

Rapid expression of fibronectin in the rabbit heart after myocardial infarction with and without reperfusion

The Journal of clinical investigation, 1992-04, Vol.89 (4), p.1060-1068

1992

Volltextzugriff (PDF)

Comparison of Hearts With 2 Types of Pressure-Overload Left Ventricular Hypertrophy

Hypertension (Dallas, Tex. 1979), 2000-05, Vol.35 (5), p.1167-1172

2000

Volltextzugriff (PDF)

Exercise training attenuates age-associated diastolic dysfunction in rats

Circulation (New York, N.Y.), 2001-07, Vol.104 (2), p.221-226

2001

Volltextzugriff (PDF)

Localization and regulation of c-fos and c-jun protooncogene induction by systolic wall stress in normal and hypertrophied rat hearts

Proceedings of the National Academy of Sciences - PNAS, 1991-12, Vol.88 (24), p.11480-11484

1991

Volltextzugriff (PDF)

1 – 25 von 196

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(186)
Mit Volltextzugriff
(183)

Publikationsform

Sprache

Englisch
(194)
Japanisch
(32)
Deutsch
(2)

Erscheinungsjahr

n.n

Thema

Science & Technology
(134)
Life Sciences & Biomedicine
(131)
Animals
(120)
Cardiovascular System & Cardiology
(102)
Cardiac & Cardiovascular Systems
(95)
Biological And Medical Sciences
(92)
Male
(83)
Medical Sciences
(79)
Peripheral Vascular Disease
(61)
Rats
(55)
Heart
(54)
Cardiology. Vascular System
(53)
Rabbits
(48)
In Vitro Techniques
(45)
Myocardium - Metabolism
(40)
Humans
(38)
Myocardial Contraction
(36)
Heart - Physiopathology
(31)
Coronary Heart Disease
(28)
Diastole
(28)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Increased glycolytic substrate protection improves ischemic cardiac dysfunction and reduces injury

Glucose-insulin-potassium for acute myocardial infarction: remarkable results from a new prospective, randomized trial

ATP synthesis during low-flow ischemia influence of increased glycolytic substrate

Increased rat cardiac angiotensin converting enzyme activity and mRNA expression in pressure overload left ventricular hypertrophy : effects on coronary resistance, contractility, and relaxation

Exercise and heart failure: A statement from the American Heart Association Committee on Exercise, Rehabilitation, and Prevention

Rapid expression of heat shock protein in the rabbit after brief cardiac ischemia

Glucose-insulin-potassium in acute myocardial infarction: the time has come for a large, prospective trial

Distribution and functional significance of cardiac angiotensin converting enzyme in hypertrophied rat hearts

Protective Effect of Increased Glycolytic Substrate Against Systolic and Diastolic Dysfunction and Increased Coronary Resistance From Prolonged Global: Underperfusion and Reperfusion in Isolated Rabbit Hearts Perfused With Erythrocyte Suspensions

Infusion of light chains from patients with cardiac amyloidosis causes diastolic dysfunction in isolated mouse hearts

Stress-shortening relations and myocardial blood flow in compensated and failing canine hearts with pressure-overload hypertrophy

Cell therapy attenuates deleterious ventricular remodeling and improves cardiac performance after myocardial infarction

Exaggerated Left Ventricular Dilation and Reduced Collagen Deposition After Myocardial Infarction in Mice Lacking Osteopontin

Angiotensin II receptor blockade attenuates the deleterious effects of exercise training on post-MI ventricular remodelling in rats

Validation of different methods to compare isovolumic cardiac function in isolated hearts of varying sizes

Glucose-6-Phosphate Dehydrogenase Modulates Cytosolic Redox Status and Contractile Phenotype in Adult Cardiomyocytes

Increased myocardial dysfunction after ischemia-reperfusion in mice lacking glucose-6-phosphate dehydrogenase

Effects of angiotensin II on intracellular Ca2+ and pH in isolated beating rabbit hearts and myocytes loaded with the indicator indo-1

Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart

Exacerbation of Left Ventricular Ischemic Diastolic Dysfunction by Pressure-Overload Hypertrophy: Modification by Specific Inhibition of Cardiac Angiotensin Converting Enzyme

Mice Lacking Inducible Nitric Oxide Synthase Have Improved Left Ventricular Contractile Function and Reduced Apoptotic Cell Death Late After Myocardial Infarction

Rapid expression of fibronectin in the rabbit heart after myocardial infarction with and without reperfusion

Comparison of Hearts With 2 Types of Pressure-Overload Left Ventricular Hypertrophy

Exercise training attenuates age-associated diastolic dysfunction in rats

Localization and regulation of c-fos and c-jun protooncogene induction by systolic wall stress in normal and hypertrophied rat hearts

Suchergebnisse filtern