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Repressive Control of Keratinocyte Cytoplasmic Inflammatory Signaling

International journal of molecular sciences, 2023-08, Vol.24 (15), p.11943

2023

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TNIP1 in Autoimmune Diseases: Regulation of Toll-like Receptor Signaling

Journal of immunology research, 2018-01, Vol.2018, p.3491269-13

2018

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Thermostable Proteins from HaCaT Keratinocytes Identify a Wide Breadth of Intrinsically Disordered Proteins and Candidates for Liquid-Liquid Phase Separation

International journal of molecular sciences, 2022-11, Vol.23 (22), p.14323

2022

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Seeing Keratinocyte Proteins through the Looking Glass of Intrinsic Disorder

International journal of molecular sciences, 2021-08, Vol.22 (15), p.7912

2021

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Enhanced Wound Healing- and Inflammasome-Associated Gene Expression in TNFAIP3-Interacting Protein 1- (TNIP1-) Deficient HaCaT Keratinocytes Parallels Reduced Reepithelialization

Mediators of inflammation, 2020, Vol.2020, p.5919150-14

2020

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Basal and stress-inducible expression of HSPA6 in human keratinocytes is regulated by negative and positive promoter regions

Cell stress & chaperones, 2015-01, Vol.20 (1), p.95-107

2015

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The Anti-Inflammatory Protein TNIP1 Is Intrinsically Disordered with Structural Flexibility Contributed by Its AHD1-UBAN Domain

Biomolecules (Basel, Switzerland), 2020-11, Vol.10 (11), p.1531

2020

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Sp sites contribute to basal and inducible expression of the human TNIP1 (TNFα-inducible protein 3-interacting protein 1) promoter

Biochemical journal, 2013-06, Vol.452 (3), p.519-529

2013

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TNIP1 reduction sensitizes keratinocytes to post-receptor signalling following exposure to TLR agonists

Cellular signalling, 2018-05, Vol.45, p.81-92

2018

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Corepressors of agonist-bound nuclear receptors

Toxicology and applied pharmacology, 2007-09, Vol.223 (3), p.288-298

2007

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Organotypic Modeling of Human Keratinocyte Response to Peroxisome Proliferators

Cells, tissues, organs, 2012-01, Vol.196 (5), p.431-441

2012

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In Silico and In Vitro Considerations of Keratinocyte Nuclear Receptor Protein Structural Order for Improving Experimental Analysis

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2109, p.93

2020

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Emerging roles for TNIP1 in regulating post-receptor signaling

Cytokine & growth factor reviews, 2012-06, Vol.23 (3), p.109-118

2012

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TNIP1 reduction of HSPA6 gene expression occurs in promoter regions lacking binding sites for known TNIP1-repressed transcription factors

Gene, 2015-01, Vol.555 (2), p.430-437

2015

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Intrinsic Disorder in Nuclear Receptor Amino Termini: From Investigational Challenge to Therapeutic Opportunity

Nuclear receptor research, 2019-06, Vol.6, p.1-16

2019

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TNIP1, a Retinoic Acid Receptor Corepressor and A20-binding Inhibitor of NF-κB, Distributes to Both Nuclear and Cytoplasmic Locations

The journal of histochemistry and cytochemistry, 2011-12, Vol.59 (12), p.1101-1112

2011

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Intrinsic disorder in integral membrane proteins

Progress in molecular biology and translational science, 2021, Vol.183, p.101-134

2021

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TNIP1 is a corepressor of agonist-bound PPARs

Archives of biochemistry and biophysics, 2011-12, Vol.516 (1), p.58-66

2011

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Human TNFα-induced protein 3-interacting protein 1 (TNIP1) promoter activation is regulated by retinoic acid receptors

Gene, 2013-02, Vol.515 (1), p.42-48

2013

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PPARγ and NF-κB regulate the gene promoter activity of their shared repressor, TNIP1

Biochimica et biophysica acta, 2012-01, Vol.1819 (1), p.1-15

2012

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Scanning for transcription factor binding by a variant EMSA

Methods in molecular biology (Clifton, N.J.), 2010-01, Vol.585, p.147

2010

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Transgene delivery to cultured keratinocytes via replication-deficient adenovirus vectors

Methods in molecular biology (Clifton, N.J.), 2014-01, Vol.1195, p.43

2014

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FHL2 expression in keratinocytes and transcriptional effect on PPARα/RXRα

Journal of dermatological science, 2004-06, Vol.35 (1), p.61-63

2004

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Interleukin-17 signaling triggers degradation of the constitutive NF-κB inhibitor ABIN-1

ImmunoHorizons, 2017-09, Vol.1 (7), p.133-141

2017

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The Anti-Inflammatory Protein TNIP1 Is Intrinsically Disordered with Structural Flexibility Contributed by Its AHD1-UBAN Domain

Biomolecules (Basel, Switzerland), 2020-11, Vol.10 (11)

2020

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Repressive Control of Keratinocyte Cytoplasmic Inflammatory Signaling

TNIP1 in Autoimmune Diseases: Regulation of Toll-like Receptor Signaling

Thermostable Proteins from HaCaT Keratinocytes Identify a Wide Breadth of Intrinsically Disordered Proteins and Candidates for Liquid-Liquid Phase Separation

Seeing Keratinocyte Proteins through the Looking Glass of Intrinsic Disorder

Enhanced Wound Healing- and Inflammasome-Associated Gene Expression in TNFAIP3-Interacting Protein 1- (TNIP1-) Deficient HaCaT Keratinocytes Parallels Reduced Reepithelialization

Basal and stress-inducible expression of HSPA6 in human keratinocytes is regulated by negative and positive promoter regions

The Anti-Inflammatory Protein TNIP1 Is Intrinsically Disordered with Structural Flexibility Contributed by Its AHD1-UBAN Domain

Sp sites contribute to basal and inducible expression of the human TNIP1 (TNFα-inducible protein 3-interacting protein 1) promoter

TNIP1 reduction sensitizes keratinocytes to post-receptor signalling following exposure to TLR agonists

Corepressors of agonist-bound nuclear receptors

Organotypic Modeling of Human Keratinocyte Response to Peroxisome Proliferators

In Silico and In Vitro Considerations of Keratinocyte Nuclear Receptor Protein Structural Order for Improving Experimental Analysis

Emerging roles for TNIP1 in regulating post-receptor signaling

TNIP1 reduction of HSPA6 gene expression occurs in promoter regions lacking binding sites for known TNIP1-repressed transcription factors

Intrinsic Disorder in Nuclear Receptor Amino Termini: From Investigational Challenge to Therapeutic Opportunity

TNIP1, a Retinoic Acid Receptor Corepressor and A20-binding Inhibitor of NF-κB, Distributes to Both Nuclear and Cytoplasmic Locations

Intrinsic disorder in integral membrane proteins

TNIP1 is a corepressor of agonist-bound PPARs

Human TNFα-induced protein 3-interacting protein 1 (TNIP1) promoter activation is regulated by retinoic acid receptors

PPARγ and NF-κB regulate the gene promoter activity of their shared repressor, TNIP1

Scanning for transcription factor binding by a variant EMSA

Transgene delivery to cultured keratinocytes via replication-deficient adenovirus vectors

FHL2 expression in keratinocytes and transcriptional effect on PPARα/RXRα

Interleukin-17 signaling triggers degradation of the constitutive NF-κB inhibitor ABIN-1

The Anti-Inflammatory Protein TNIP1 Is Intrinsically Disordered with Structural Flexibility Contributed by Its AHD1-UBAN Domain

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