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Physiological relevance of cell-specific distribution patterns of CFTR, NKCC1, NBCe1, and NHE3 along the crypt-villus axis in the intestine

American journal of physiology: Gastrointestinal and liver physiology, 2011-01, Vol.300 (1), p.G82-G98

2011

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Zinc ameliorates intestinal barrier dysfunctions in shigellosis by reinstating claudin-2 and -4 on the membranes

American journal of physiology: Gastrointestinal and liver physiology, 2019-02, Vol.316 (2), p.G229-G246

2019

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CFTR High Expresser Cells in cystic fibrosis and intestinal diseases

Heliyon, 2023-03, Vol.9 (3), p.e14568-e14568, Article e14568

2023

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Loss of Serum Glucocorticoid-Inducible Kinase 1 SGK1 Worsens Malabsorption and Diarrhea in Microvillus Inclusion Disease (MVID)

Journal of clinical medicine, 2022-07, Vol.11 (14), p.4179

2022

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Myosin Ia is Required for CFTR Brush Border Membrane Trafficking and Ion Transport in the Mouse Small Intestine

Traffic (Copenhagen, Denmark), 2012-08, Vol.13 (8), p.1072-1082

2012

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Linaclotide activates guanylate cyclase‐C/cGMP/protein kinase‐II‐dependent trafficking of CFTR in the intestine

Physiological reports, 2017-06, Vol.5 (11), p.e13299-n/a

2017

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Localization and function of humanized F508del-CFTR in mouse intestine following activation of serum glucocorticoid kinase 1 and Trikafta

European journal of pharmacology, 2024-09, Vol.978, p.176771, Article 176771

2024

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Molecular motors and apical CFTR traffic in epithelia

International Journal of Molecular Sciences, 2013-05, Vol.14 (5), p.9628-9642

2013

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Serum and Glucocorticoid‐Inducible Kinase 1 (SGK1): An Important Contributor to Diarrhea and Malabsorption in Microvillus Inclusion Disease (MVID)

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Glucocorticoids and myosin5b loss of function induce heightened PKA signaling in addition to membrane traffic defects

Molecular biology of the cell, 2019-12, Vol.30 (26), p.3076-3089

2019

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AP2 α modulates cystic fibrosis transmembrane conductance regulator function in the human intestine

Journal of cystic fibrosis, 2017-05, Vol.16 (3), p.327-334

2017

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Heterogeneity in mouse spasmolytic polypeptide-expressing metaplasia lineages identifies markers of metaplastic progression

Gut, 2013-09, Vol.62 (9), p.1270-1279

2013

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Glucocorticoids and serum- and glucocorticoid-inducible kinase 1 are potent regulators of CFTR in the native intestine: implications for stress-induced diarrhea

American journal of physiology: Gastrointestinal and liver physiology, 2020-08, Vol.319 (2), p.G121-G132

2020

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Myosin 5b loss of function leads to defects in polarized signaling: implication for microvillus inclusion disease pathogenesis and treatment

American journal of physiology: Gastrointestinal and liver physiology, 2014-11, Vol.307 (10), p.G992-G1001

2014

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Characterization of CFTR High Expresser cells in the intestine

American journal of physiology: Gastrointestinal and liver physiology, 2013-09, Vol.305 (6), p.G453-G465

2013

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Lubiprostone Targets Prostanoid Signaling and Promotes Ion Transporter Trafficking, Mucus Exocytosis, and Contractility

Digestive diseases and sciences, 2012-11, Vol.57 (11), p.2826-2845

2012

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Identification of intestinal ion transport defects in microvillus inclusion disease

American journal of physiology: Gastrointestinal and liver physiology, 2016-07, Vol.311 (1), p.G142-G155

2016

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Regulated traffic of anion transporters in mammalian Brunner's glands: a role for water and fluid transport

American journal of physiology: Gastrointestinal and liver physiology, 2013-08, Vol.305 (3), p.G258-G275

2013

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Microvillus Inclusion Disease: A Genetic Defect Affecting Apical Membrane Protein Traffic in Intestinal Epithelium

Traffic (Copenhagen, Denmark), 2000-01, Vol.1 (1), p.76-83

2000

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Cell-specific effects of luminal acid, bicarbonate, cAMP, and carbachol on transporter trafficking in the intestine

American journal of physiology: Gastrointestinal and liver physiology, 2012-10, Vol.303 (8), p.G937-G950

2012

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Functional vacuolar ATPase (V-ATPase) proton pumps traffic to the enterocyte brush border membrane and require CFTR

American Journal of Physiology: Cell Physiology, 2013-11, Vol.305 (9), p.C981-C996

2013

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Deficits in Enterocyte Apical Transporters Associated with Loss of Myosin VB

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S187-S187

2017

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Restoration of cytoskeletal and membrane tethering defects but not defects in membrane trafficking in the intestinal brush border of mice lacking both myosin Ia and myosin VI

Cytoskeleton (Hoboken, N.J.), 2015-09, Vol.72 (9), p.455-476

2015

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Anomalous apical plasma membrane phenotype in CK8-deficient mice indicates a novel role for intermediate filaments in the polarization of simple epithelia

Journal of cell science, 2001-02, Vol.114 (Pt 3), p.563-575

2001

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978 CFTR HIGH EXPRESSER CELLS EXPRESS NEUROPOD GENES AND RESPOND TO ENVIRONMENTAL CUES IN THE INTESTINE

Gastroenterology (New York, N.Y. 1943), 2024-05, Vol.166 (5), p.S-236-S-236

2024

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Physiological relevance of cell-specific distribution patterns of CFTR, NKCC1, NBCe1, and NHE3 along the crypt-villus axis in the intestine

Zinc ameliorates intestinal barrier dysfunctions in shigellosis by reinstating claudin-2 and -4 on the membranes

CFTR High Expresser Cells in cystic fibrosis and intestinal diseases

Loss of Serum Glucocorticoid-Inducible Kinase 1 SGK1 Worsens Malabsorption and Diarrhea in Microvillus Inclusion Disease (MVID)

Myosin Ia is Required for CFTR Brush Border Membrane Trafficking and Ion Transport in the Mouse Small Intestine

Linaclotide activates guanylate cyclase‐C/cGMP/protein kinase‐II‐dependent trafficking of CFTR in the intestine

Localization and function of humanized F508del-CFTR in mouse intestine following activation of serum glucocorticoid kinase 1 and Trikafta

Molecular motors and apical CFTR traffic in epithelia

Serum and Glucocorticoid‐Inducible Kinase 1 (SGK1): An Important Contributor to Diarrhea and Malabsorption in Microvillus Inclusion Disease (MVID)

Glucocorticoids and myosin5b loss of function induce heightened PKA signaling in addition to membrane traffic defects

AP2 α modulates cystic fibrosis transmembrane conductance regulator function in the human intestine

Heterogeneity in mouse spasmolytic polypeptide-expressing metaplasia lineages identifies markers of metaplastic progression

Glucocorticoids and serum- and glucocorticoid-inducible kinase 1 are potent regulators of CFTR in the native intestine: implications for stress-induced diarrhea

Myosin 5b loss of function leads to defects in polarized signaling: implication for microvillus inclusion disease pathogenesis and treatment

Characterization of CFTR High Expresser cells in the intestine

Lubiprostone Targets Prostanoid Signaling and Promotes Ion Transporter Trafficking, Mucus Exocytosis, and Contractility

Identification of intestinal ion transport defects in microvillus inclusion disease

Regulated traffic of anion transporters in mammalian Brunner's glands: a role for water and fluid transport

Microvillus Inclusion Disease: A Genetic Defect Affecting Apical Membrane Protein Traffic in Intestinal Epithelium

Cell-specific effects of luminal acid, bicarbonate, cAMP, and carbachol on transporter trafficking in the intestine

Functional vacuolar ATPase (V-ATPase) proton pumps traffic to the enterocyte brush border membrane and require CFTR

Deficits in Enterocyte Apical Transporters Associated with Loss of Myosin VB

Restoration of cytoskeletal and membrane tethering defects but not defects in membrane trafficking in the intestinal brush border of mice lacking both myosin Ia and myosin VI

Anomalous apical plasma membrane phenotype in CK8-deficient mice indicates a novel role for intermediate filaments in the polarization of simple epithelia

978 CFTR HIGH EXPRESSER CELLS EXPRESS NEUROPOD GENES AND RESPOND TO ENVIRONMENTAL CUES IN THE INTESTINE

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