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Body-first Parkinson’s disease and variant Creutzfeldt–Jakob disease – similar or different?

Neurobiology of disease, 2022-03, Vol.164, p.105625-105625, Article 105625

2022

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Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism

Proceedings of the National Academy of Sciences - PNAS, 2015-09, Vol.112 (38), p.E5308-E5317

2015

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The role of α-synuclein prion strains in Parkinson's disease and multiple system atrophy

PLoS pathogens, 2024-01, Vol.20 (1), p.e1011920

2024

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Tau prions from Alzheimer’s disease and chronic traumatic encephalopathy patients propagate in cultured cells

Proceedings of the National Academy of Sciences - PNAS, 2016-12, Vol.113 (50), p.E8187-E8196

2016

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The E46K mutation modulates α-synuclein prion replication in transgenic mice

PLoS pathogens, 2022-12, Vol.18 (12), p.e1010956-e1010956

2022

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Propagation of prions causing synucleinopathies in cultured cells

Proceedings of the National Academy of Sciences - PNAS, 2015-09, Vol.112 (35), p.E4949-E4958

2015

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Kinetics of α-synuclein prions preceding neuropathological inclusions in multiple system atrophy

PLoS pathogens, 2020-02, Vol.16 (2), p.e1008222-e1008222

2020

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Multiple system atrophy prions retain strain specificity after serial propagation in two different Tg(SNCAA53T) mouse lines

Acta neuropathologica, 2019-03, Vol.137 (3), p.437-454

2019

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Familial Parkinson’s point mutation abolishes multiple system atrophy prion replication

Proceedings of the National Academy of Sciences - PNAS, 2018-01, Vol.115 (2), p.409-414

2018

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Mouse closed head traumatic brain injury replicates the histological tau pathology pattern of human disease: characterization of a novel model and systematic review of the literature

Acta neuropathologica communications, 2021-06, Vol.9 (1), p.118-118, Article 118

2021

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Strain diversity in neurodegenerative disease: an argument for a personalized medicine approach to diagnosis and treatment

Acta neuropathologica, 2021-07, Vol.142 (1), p.1-3

Woerman, Amanda L.

2021

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Tau aggregates are RNA-protein assemblies that mislocalize multiple nuclear speckle components

Neuron (Cambridge, Mass.), 2021-05, Vol.109 (10), p.1675-1691.e9

2021

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Effect of host and strain factors on α-synuclein prion pathogenesis

Trends in neurosciences (Regular ed.), 2024-07, Vol.47 (7), p.538-550

2024

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Evidence of distinct α-synuclein strains underlying disease heterogeneity

Acta neuropathologica, 2021-07, Vol.142 (1), p.73-86

2021

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Different α-synuclein prion strains cause dementia with Lewy bodies and multiple system atrophy

Proceedings of the National Academy of Sciences - PNAS, 2022-02, Vol.119 (6), p.1

2022

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The importance of developing strain-specific models of neurodegenerative disease

Acta neuropathologica, 2017-11, Vol.134 (5), p.809-812

Woerman, Amanda L.

2017

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Consequences of variability in α-synuclein fibril structure on strain biology

Acta neuropathologica, 2022-03, Vol.143 (3), p.311-330

2022

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Aβ and tau prion-like activities decline with longevity in the Alzheimer’s disease human brain

Science translational medicine, 2019-05, Vol.11 (490)

2019

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α-Synuclein: Multiple System Atrophy Prions

Cold Spring Harbor perspectives in medicine, 2018-07, Vol.8 (7), p.a024588

2018

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MSA prions exhibit remarkable stability and resistance to inactivation

Acta neuropathologica, 2018-01, Vol.135 (1), p.49-63

2018

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Are Preformed Fibrils a Model of Parkinson’s Disease?

Journal of Parkinson's disease, 2024-01, Vol.14 (6), p.1095-1103

2024

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Bioassays and Inactivation of Prions

Cold Spring Harbor perspectives in biology, 2017-08, Vol.9 (8), p.a023499

2017

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Tau interactome analyses in CRISPR-Cas9 engineered neuronal cells reveal ATPase-dependent binding of wild-type but not P301L Tau to non-muscle myosins

Scientific reports, 2019-11, Vol.9 (1), p.16238-21, Article 16238

2019

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Kinetics of Human Mutant Tau Prion Formation in the Brains of 2 Transgenic Mouse Lines

JAMA neurology, 2017-12, Vol.74 (12), p.1464

2017

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Multiple system atrophy prions transmit neurological disease to mice expressing wild-type human α-synuclein

Acta neuropathologica, 2022-10, Vol.144 (4), p.677-690

2022

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Body-first Parkinson’s disease and variant Creutzfeldt–Jakob disease – similar or different?

Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism

The role of α-synuclein prion strains in Parkinson's disease and multiple system atrophy

Tau prions from Alzheimer’s disease and chronic traumatic encephalopathy patients propagate in cultured cells

The E46K mutation modulates α-synuclein prion replication in transgenic mice

Propagation of prions causing synucleinopathies in cultured cells

Kinetics of α-synuclein prions preceding neuropathological inclusions in multiple system atrophy

Multiple system atrophy prions retain strain specificity after serial propagation in two different Tg(SNCAA53T) mouse lines

Familial Parkinson’s point mutation abolishes multiple system atrophy prion replication

Mouse closed head traumatic brain injury replicates the histological tau pathology pattern of human disease: characterization of a novel model and systematic review of the literature

Strain diversity in neurodegenerative disease: an argument for a personalized medicine approach to diagnosis and treatment

Tau aggregates are RNA-protein assemblies that mislocalize multiple nuclear speckle components

Effect of host and strain factors on α-synuclein prion pathogenesis

Evidence of distinct α-synuclein strains underlying disease heterogeneity

Different α-synuclein prion strains cause dementia with Lewy bodies and multiple system atrophy

The importance of developing strain-specific models of neurodegenerative disease

Consequences of variability in α-synuclein fibril structure on strain biology

Aβ and tau prion-like activities decline with longevity in the Alzheimer’s disease human brain

α-Synuclein: Multiple System Atrophy Prions

MSA prions exhibit remarkable stability and resistance to inactivation

Are Preformed Fibrils a Model of Parkinson’s Disease?

Bioassays and Inactivation of Prions

Tau interactome analyses in CRISPR-Cas9 engineered neuronal cells reveal ATPase-dependent binding of wild-type but not P301L Tau to non-muscle myosins

Kinetics of Human Mutant Tau Prion Formation in the Brains of 2 Transgenic Mouse Lines

Multiple system atrophy prions transmit neurological disease to mice expressing wild-type human α-synuclein

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