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N-terminal tau truncation in the pathogenesis of Alzheimer's disease (AD): Developing a novel diagnostic and therapeutic approach

Biochimica et biophysica acta. Molecular basis of disease, 2020-03, Vol.1866 (3), p.165584, Article 165584

2020

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NH2-truncated human tau induces deregulated mitophagy in neurons by aberrant recruitment of Parkin and UCHL-1: implications in Alzheimer's disease

Human molecular genetics, 2015-06, Vol.24 (11), p.3058-3081

2015

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Nerve growth factor as a paradigm of neurotrophins related to Alzheimer's disease

Developmental neurobiology (Hoboken, N.J.), 2010-04, Vol.70 (5), p.372-383

2010

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Mitochondrial respiratory chain Complexes I and IV are impaired by β-amyloid via direct interaction and through Complex I-dependent ROS production, respectively

Mitochondrion, 2013-07, Vol.13 (4), p.298-311

2013

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AD-linked, toxic NH2 human tau affects the quality control of mitochondria in neurons

Neurobiology of disease, 2014-02, Vol.62, p.489-507

2014

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Glycolytic enzyme upregulation and numbness of mitochondrial activity characterize the early phase of apoptosis in cerebellar granule cells

Apoptosis (London), 2015-01, Vol.20 (1), p.10-28

2015

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A peptide containing residues 26–44 of tau protein impairs mitochondrial oxidative phosphorylation acting at the level of the adenine nucleotide translocator

Biochimica et biophysica acta, 2008-10, Vol.1777 (10), p.1289-1300

2008

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AMPK is activated early in cerebellar granule cells undergoing apoptosis and influences VADC1 phosphorylation status and activity

Apoptosis (London), 2017-09, Vol.22 (9), p.1069-1078

2017

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AD-Related N-Terminal Truncated Tau Is Sufficient to Recapitulate In Vivo the Early Perturbations of Human Neuropathology: Implications for Immunotherapy

Molecular neurobiology, 2018-10, Vol.55 (10), p.8124-8153

2018

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Endogenous Aβ causes cell death via early tau hyperphosphorylation

Neurobiology of aging, 2011-06, Vol.32 (6), p.969-990

2011

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Apoptosis and in vitro Alzheimer disease neuronal models

Communicative & integrative biology, 2009, Vol.2 (2), p.163-169

2009

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Extracellular ADP prevents neuronal apoptosis via activation of cell antioxidant enzymes and protection of mitochondrial ANT-1

Biochimica et biophysica acta, 2014-08, Vol.1837 (8), p.1338-1349

2014

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Glucose-6-phosphate tips the balance in modulating apoptosis in cerebellar granule cells

FEBS letters, 2015-02, Vol.589 (5), p.651-658

2015

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Dissecting the molecular mechanism by which NH2htau and Aβ1-42 peptides impair mitochondrial ANT-1 in Alzheimer disease

Biochimica et biophysica acta, 2013-07, Vol.1827 (7), p.848-860

2013

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Corrigendum to “AD-linked, toxic NH2 human tau affects the quality control of mitochondria in neurons” Neurobioly of Disease (2014) 62:489–507

Neurobiology of disease, 2015-02, Vol.74, p.102-103

2015

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Identification of a caspase-derived N-terminal tau fragment in cellular and animal Alzheimer's disease models

Molecular and cellular neuroscience, 2008-07, Vol.38 (3), p.381-392

2008

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SP protects cerebellar granule cells against β-amyloid-induced apoptosis by down-regulation and reduced activity of Kv4 potassium channels

Neuropharmacology, 2010, Vol.58 (1), p.268-276

2010

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Does the term 'trophic' actually mean anti-amyloidogenic? The case of NGF

Cell death and differentiation, 2010-07, Vol.17 (7), p.1126-1133

2010

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Role of N-terminal tau domain integrity on the survival of cerebellar granule neurons

Cell death and differentiation, 2004-02, Vol.11 (2), p.217-230

2004

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Isolation and characterization of VGF peptides in rat brain. Role of PC1/3 and PC2 in the maturation of VGF precursor

Journal of neurochemistry, 2002-05, Vol.81 (3), p.565-574

2002

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Substance P provides neuroprotection in cerebellar granule cells through Akt and MAPK/Erk activation: Evidence for the involvement of the delayed rectifier potassium current

Neuropharmacology, 2007-05, Vol.52 (6), p.1366-1377

2007

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Involvement of cannabinoid CB1- and CB2-receptors in the modulation of exocrine pancreatic secretion

Pharmacological research, 2009-03, Vol.59 (3), p.207-214

2009

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NMDA Receptor Mediates Tau-Induced Neurotoxicity by Calpain and ERK/MAPK Activation

Proceedings of the National Academy of Sciences - PNAS, 2006-02, Vol.103 (8), p.2892-2897

2006

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Apoptosis and in vitro Alzheimer's disease neuronal models

Communicative & integrative biology, 2009-03, Vol.2 (2), p.163-169

2009

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Role of the autophagic‐lysosomal system on low potassium‐induced apoptosis in cultured cerebellar granule cells

Journal of neurochemistry, 2005-03, Vol.92 (5), p.1228-1242

2005

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N-terminal tau truncation in the pathogenesis of Alzheimer's disease (AD): Developing a novel diagnostic and therapeutic approach

NH2-truncated human tau induces deregulated mitophagy in neurons by aberrant recruitment of Parkin and UCHL-1: implications in Alzheimer's disease

Nerve growth factor as a paradigm of neurotrophins related to Alzheimer's disease

Mitochondrial respiratory chain Complexes I and IV are impaired by β-amyloid via direct interaction and through Complex I-dependent ROS production, respectively

AD-linked, toxic NH2 human tau affects the quality control of mitochondria in neurons

Glycolytic enzyme upregulation and numbness of mitochondrial activity characterize the early phase of apoptosis in cerebellar granule cells

A peptide containing residues 26–44 of tau protein impairs mitochondrial oxidative phosphorylation acting at the level of the adenine nucleotide translocator

AMPK is activated early in cerebellar granule cells undergoing apoptosis and influences VADC1 phosphorylation status and activity

AD-Related N-Terminal Truncated Tau Is Sufficient to Recapitulate In Vivo the Early Perturbations of Human Neuropathology: Implications for Immunotherapy

Endogenous Aβ causes cell death via early tau hyperphosphorylation

Apoptosis and in vitro Alzheimer disease neuronal models

Extracellular ADP prevents neuronal apoptosis via activation of cell antioxidant enzymes and protection of mitochondrial ANT-1

Glucose-6-phosphate tips the balance in modulating apoptosis in cerebellar granule cells

Dissecting the molecular mechanism by which NH2htau and Aβ1-42 peptides impair mitochondrial ANT-1 in Alzheimer disease

Corrigendum to “AD-linked, toxic NH2 human tau affects the quality control of mitochondria in neurons” Neurobioly of Disease (2014) 62:489–507

Identification of a caspase-derived N-terminal tau fragment in cellular and animal Alzheimer's disease models

SP protects cerebellar granule cells against β-amyloid-induced apoptosis by down-regulation and reduced activity of Kv4 potassium channels

Does the term 'trophic' actually mean anti-amyloidogenic? The case of NGF

Role of N-terminal tau domain integrity on the survival of cerebellar granule neurons

Isolation and characterization of VGF peptides in rat brain. Role of PC1/3 and PC2 in the maturation of VGF precursor

Substance P provides neuroprotection in cerebellar granule cells through Akt and MAPK/Erk activation: Evidence for the involvement of the delayed rectifier potassium current

Involvement of cannabinoid CB1- and CB2-receptors in the modulation of exocrine pancreatic secretion

NMDA Receptor Mediates Tau-Induced Neurotoxicity by Calpain and ERK/MAPK Activation

Apoptosis and in vitro Alzheimer's disease neuronal models

Role of the autophagic‐lysosomal system on low potassium‐induced apoptosis in cultured cerebellar granule cells

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