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CCL5 promotes breast cancer recurrence through macrophage recruitment in residual tumors

eLife, 2019-04, Vol.8

2019

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Context-dependent effects of whole-genome duplication during mammary tumor recurrence

Scientific reports, 2021-07, Vol.11 (1), p.14932-14932, Article 14932

2021

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Par-4 Downregulation Promotes Breast Cancer Recurrence by Preventing Multinucleation following Targeted Therapy

Cancer cell, 2013-07, Vol.24 (1), p.30-44

2013

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Epigenetic silencing of tumor suppressor Par-4 promotes chemoresistance in recurrent breast cancer

The Journal of clinical investigation, 2018-10, Vol.128 (10), p.4413-4428

2018

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Adaptation and selection shape clonal evolution of tumors during residual disease and recurrence

Nature communications, 2020-10, Vol.11 (1), p.5017-5017, Article 5017

2020

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Oncogene pathway activation in mammary tumors dictates FDG-PET uptake

Cancer research (Chicago, Ill.), 2014-12, Vol.74 (24), p.7583-7598

2014

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Autocrine prolactin induced by the Pten-Akt pathway is required for lactation initiation and provides a direct link between the Akt and Stat5 pathways

Genes & development, 2012-10, Vol.26 (19), p.2154-2168

2012

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G9a Promotes Breast Cancer Recurrence through Repression of a Pro-inflammatory Program

Cell reports (Cambridge), 2020-11, Vol.33 (5), p.108341-108341, Article 108341

2020

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Oncogenic transformation by inhibitor-sensitive and -resistant EGFR mutants

PLoS medicine, 2005-11, Vol.2 (11), p.e313

2005

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Signal transducer and activator of transcription 3 is required for the oncogenic effects of non-small-cell lung cancer-associated mutations of the epidermal growth factor receptor

Cancer research (Chicago, Ill.), 2006-03, Vol.66 (6), p.3162-3168

2006

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Identification of a genetic signature of activated signal transducer and activator of transcription 3 in human tumors

Cancer research (Chicago, Ill.), 2005-06, Vol.65 (12), p.5054-5062

2005

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In vivo metabolic imaging identifies lipid vulnerability in a preclinical model of Her2+/Neu breast cancer residual disease and recurrence

NPJ breast cancer, 2022-09, Vol.8 (1), p.111-111, Article 111

2022

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DDR2 upregulation confers ferroptosis susceptibility of recurrent breast tumors through the Hippo pathway

Oncogene, 2021-03, Vol.40 (11), p.2018-2034

2021

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Genome-wide analysis of STAT target genes: Elucidating the mechanism of STAT-mediated oncogenesis

Cancer biology & therapy, 2004-11, Vol.3 (11), p.1045-1050

2004

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NRF2 activation promotes the recurrence of dormant tumour cells through regulation of redox and nucleotide metabolism

Nature metabolism, 2020-04, Vol.2 (4), p.318-334

2020

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RIPK3 upregulation confers robust proliferation and collateral cystine-dependence on breast cancer recurrence

Cell death and differentiation, 2020-07, Vol.27 (7), p.2234-2247

2020

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Correction: Oncogenic Transformation by Inhibitor-Sensitive and -Resistant EGFR Mutants

PLoS medicine, 2024-09, Vol.21 (9), p.e1004470

2024

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Optical Imaging of Glucose Uptake and Mitochondrial Membrane Potential to Characterize Her2 Breast Tumor Metabolic Phenotypes

Molecular cancer research, 2019-07, Vol.17 (7), p.1545-1555

2019

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Isolation of Unique STAT5 Targets by Chromatin Immunoprecipitation-based Gene Identification

The Journal of biological chemistry, 2004-12, Vol.279 (52), p.54724-54730

2004

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Immune cells in residual disease and recurrence

Trends in Cancer, 2023-07, Vol.9 (7), p.554-565

2023

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APOBEC Mutagenesis Inhibits Breast Cancer Growth through Induction of T cell-Mediated Antitumor Immune Responses

Cancer immunology research, 2022-01, Vol.10 (1), p.70-86

2022

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Foxo-dependent Par-4 Upregulation Prevents Long-term Survival of Residual Cells Following PI3K-Akt Inhibition

Molecular cancer research, 2018-04, Vol.16 (4), p.599-609

2018

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Local-Scale Bat Guild Activity Differs with Rice Growth Stage at Ground Level in the Philippines

Diversity (Basel), 2019-09, Vol.11 (9), p.148

2019

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SPSB1 promotes breast cancer recurrence by potentiating c-MET signaling

Cancer discovery, 2014-07, Vol.4 (7), p.790-803

2014

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Optical imaging reveals chemotherapy-induced metabolic reprogramming of residual disease and recurrence

Science advances, 2024-04, Vol.10 (14), p.eadj7540-eadj7540

2024

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CCL5 promotes breast cancer recurrence through macrophage recruitment in residual tumors

Context-dependent effects of whole-genome duplication during mammary tumor recurrence

Par-4 Downregulation Promotes Breast Cancer Recurrence by Preventing Multinucleation following Targeted Therapy

Epigenetic silencing of tumor suppressor Par-4 promotes chemoresistance in recurrent breast cancer

Adaptation and selection shape clonal evolution of tumors during residual disease and recurrence

Oncogene pathway activation in mammary tumors dictates FDG-PET uptake

Autocrine prolactin induced by the Pten-Akt pathway is required for lactation initiation and provides a direct link between the Akt and Stat5 pathways

G9a Promotes Breast Cancer Recurrence through Repression of a Pro-inflammatory Program

Oncogenic transformation by inhibitor-sensitive and -resistant EGFR mutants

Signal transducer and activator of transcription 3 is required for the oncogenic effects of non-small-cell lung cancer-associated mutations of the epidermal growth factor receptor

Identification of a genetic signature of activated signal transducer and activator of transcription 3 in human tumors

In vivo metabolic imaging identifies lipid vulnerability in a preclinical model of Her2+/Neu breast cancer residual disease and recurrence

DDR2 upregulation confers ferroptosis susceptibility of recurrent breast tumors through the Hippo pathway

Genome-wide analysis of STAT target genes: Elucidating the mechanism of STAT-mediated oncogenesis

NRF2 activation promotes the recurrence of dormant tumour cells through regulation of redox and nucleotide metabolism

RIPK3 upregulation confers robust proliferation and collateral cystine-dependence on breast cancer recurrence

Correction: Oncogenic Transformation by Inhibitor-Sensitive and -Resistant EGFR Mutants

Optical Imaging of Glucose Uptake and Mitochondrial Membrane Potential to Characterize Her2 Breast Tumor Metabolic Phenotypes

Isolation of Unique STAT5 Targets by Chromatin Immunoprecipitation-based Gene Identification

Immune cells in residual disease and recurrence

APOBEC Mutagenesis Inhibits Breast Cancer Growth through Induction of T cell-Mediated Antitumor Immune Responses

Foxo-dependent Par-4 Upregulation Prevents Long-term Survival of Residual Cells Following PI3K-Akt Inhibition

Local-Scale Bat Guild Activity Differs with Rice Growth Stage at Ground Level in the Philippines

SPSB1 promotes breast cancer recurrence by potentiating c-MET signaling

Optical imaging reveals chemotherapy-induced metabolic reprogramming of residual disease and recurrence

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