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Cleavage of DFNA5 by caspase-3 during apoptosis mediates progression to secondary necrotic/pyroptotic cell death

Nature communications, 2017-01, Vol.8 (1), p.14128-14128, Article 14128

2017

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Gasdermin pores permeabilize mitochondria to augment caspase-3 activation during apoptosis and inflammasome activation

Nature communications, 2019-04, Vol.10 (1), p.1689-1689, Article 1689

2019

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Non-transcriptional Priming and Deubiquitination Regulate NLRP3 Inflammasome Activation

The Journal of biological chemistry, 2012-10, Vol.287 (43), p.36617-36622

2012

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AIM2 activates the inflammasome and cell death in response to cytoplasmic DNA

Nature, 2009-03, Vol.458 (7237), p.509-513

2009

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The NLRP6 Inflammasome Recognizes Lipoteichoic Acid and Regulates Gram-Positive Pathogen Infection

Cell, 2018-11, Vol.175 (6), p.1651-1664.e14

2018

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Cutting edge: TLR signaling licenses IRAK1 for rapid activation of the NLRP3 inflammasome

The Journal of immunology (1950), 2013-10, Vol.191 (8), p.3995-3999

2013

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Mutant BRAF and MEK Inhibitors Regulate the Tumor Immune Microenvironment via Pyroptosis

Cancer discovery, 2020-02, Vol.10 (2), p.254-269

2020

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Involvement of the AIM2, NLRC4, and NLRP3 Inflammasomes in Caspase-1 Activation by Listeria monocytogenes

Journal of clinical immunology, 2010-09, Vol.30 (5), p.693-702

2010

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Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3

Nature communications, 2015-06, Vol.6 (1), p.7515-7515, Article 7515

2015

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Anti-inflammatory Compounds Parthenolide and Bay 11-7082 Are Direct Inhibitors of the Inflammasome

The Journal of biological chemistry, 2010-03, Vol.285 (13), p.9792-9802

2010

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Inflammasome priming by lipopolysaccharide is dependent upon ERK signaling and proteasome function

The Journal of immunology (1950), 2014-04, Vol.192 (8), p.3881-3888

2014

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Chloride sensing by WNK1 regulates NLRP3 inflammasome activation and pyroptosis

Nature communications, 2021-07, Vol.12 (1), p.4546-4546, Article 4546

2021

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The AIM2 inflammasome is critical for innate immunity to Francisella tularensis

Nature immunology, 2010-05, Vol.11 (5), p.385-393

2010

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Critical roles of ASC inflammasomes in caspase-1 activation and host innate resistance to Streptococcus pneumoniae infection

The Journal of immunology (1950), 2011-11, Vol.187 (9), p.4890-4899

2011

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Inactivation of Omi/HtrA2 protease leads to the deregulation of mitochondrial Mulan E3 ubiquitin ligase and increased mitophagy

Biochimica et biophysica acta, 2014-07, Vol.1843 (7), p.1295-1307

2014

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Pyrin Activates the ASC Pyroptosome in Response to Engagement by Autoinflammatory PSTPIP1 Mutants

Molecular cell, 2007-10, Vol.28 (2), p.214-227

2007

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Gasdermins: novel mitochondrial pore-forming proteins

Molecular & cellular oncology, 2019-01, Vol.6 (5), p.e1621501-e1621501

2019

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A novel role for the mitochondrial HTRA2/OMI protease in aging

Autophagy, 2013-03, Vol.9 (3), p.420-421

2013

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Identification of Omi/HtrA2 as a Mitochondrial Apoptotic Serine Protease That Disrupts Inhibitor of Apoptosis Protein-Caspase Interaction

The Journal of biological chemistry, 2002-01, Vol.277 (1), p.432-438

2002

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Caspase-2 Induces Apoptosis by Releasing Proapoptotic Proteins from Mitochondria

The Journal of biological chemistry, 2002-04, Vol.277 (16), p.13430-13437

2002

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The PYRIN-CARD Protein ASC Is an Activating Adaptor for Caspase-1

The Journal of biological chemistry, 2002-06, Vol.277 (24), p.21119-21122

2002

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Mechanism of XIAP-Mediated Inhibition of Caspase-9

Molecular cell, 2003-02, Vol.11 (2), p.519-527

2003

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Negative regulation of the Apaf-1 apoptosome by Hsp70

Nature cell biology, 2000-08, Vol.2 (8), p.476-483

2000

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Multiple Autonomous Cell Death Suppression Strategies Ensure Cytomegalovirus Fitness

Viruses, 2021-08, Vol.13 (9), p.1707

2021

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Loss of Omi mitochondrial protease activity causes the neuromuscular disorder of mnd2 mutant mice

Nature, 2003-10, Vol.425 (6959), p.721-727

2003

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Cleavage of DFNA5 by caspase-3 during apoptosis mediates progression to secondary necrotic/pyroptotic cell death

Gasdermin pores permeabilize mitochondria to augment caspase-3 activation during apoptosis and inflammasome activation

Non-transcriptional Priming and Deubiquitination Regulate NLRP3 Inflammasome Activation

AIM2 activates the inflammasome and cell death in response to cytoplasmic DNA

The NLRP6 Inflammasome Recognizes Lipoteichoic Acid and Regulates Gram-Positive Pathogen Infection

Cutting edge: TLR signaling licenses IRAK1 for rapid activation of the NLRP3 inflammasome

Mutant BRAF and MEK Inhibitors Regulate the Tumor Immune Microenvironment via Pyroptosis

Involvement of the AIM2, NLRC4, and NLRP3 Inflammasomes in Caspase-1 Activation by Listeria monocytogenes

Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3

Anti-inflammatory Compounds Parthenolide and Bay 11-7082 Are Direct Inhibitors of the Inflammasome

Inflammasome priming by lipopolysaccharide is dependent upon ERK signaling and proteasome function

Chloride sensing by WNK1 regulates NLRP3 inflammasome activation and pyroptosis

The AIM2 inflammasome is critical for innate immunity to Francisella tularensis

Critical roles of ASC inflammasomes in caspase-1 activation and host innate resistance to Streptococcus pneumoniae infection

Inactivation of Omi/HtrA2 protease leads to the deregulation of mitochondrial Mulan E3 ubiquitin ligase and increased mitophagy

Pyrin Activates the ASC Pyroptosome in Response to Engagement by Autoinflammatory PSTPIP1 Mutants

Gasdermins: novel mitochondrial pore-forming proteins

A novel role for the mitochondrial HTRA2/OMI protease in aging

Identification of Omi/HtrA2 as a Mitochondrial Apoptotic Serine Protease That Disrupts Inhibitor of Apoptosis Protein-Caspase Interaction

Caspase-2 Induces Apoptosis by Releasing Proapoptotic Proteins from Mitochondria

The PYRIN-CARD Protein ASC Is an Activating Adaptor for Caspase-1

Mechanism of XIAP-Mediated Inhibition of Caspase-9

Negative regulation of the Apaf-1 apoptosome by Hsp70

Multiple Autonomous Cell Death Suppression Strategies Ensure Cytomegalovirus Fitness

Loss of Omi mitochondrial protease activity causes the neuromuscular disorder of mnd2 mutant mice

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