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Rotavirus Infection Accelerates Type 1 Diabetes in Mice with Established Insulitis

Journal of Virology, 2008-07, Vol.82 (13), p.6139-6149

2008

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Proapoptotic BH3-Only Protein Bid Is Essential For Death Receptor–Induced Apoptosis of Pancreatic β-Cells

Diabetes (New York, N.Y.), 2008-05, Vol.57 (5), p.1284-1292

2008

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Islet β-Cells Deficient in Bcl-xL Develop but Are Abnormally Sensitive to Apoptotic Stimuli

Diabetes (New York, N.Y.), 2009-10, Vol.58 (10), p.2316-2323

2009

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IL-1 Receptor Deficiency Slows Progression to Diabetes in the NOD Mouse

Diabetes (New York, N.Y.), 2004-01, Vol.53 (1), p.113-121

2004

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Role of SLAM in NKT cell development revealed by transgenic complementation in NOD mice

The Journal of immunology (1950), 2011-04, Vol.186 (7), p.3953-3965

2011

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Transgenic Expression of CD95 Ligand on Islet β Cells Induces a Granulocytic Infiltration but does not Confer Immune Privilege upon Islet Allografts

Proceedings of the National Academy of Sciences - PNAS, 1997-04, Vol.94 (8), p.3943-3947

1997

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Rotavirus Infection of Infant and Young Adult Nonobese Diabetic Mice Involves Extraintestinal Spread and Delays Diabetes Onset

Journal of Virology, 2007-06, Vol.81 (12), p.6446-6458

2007

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Mechanisms of β Cell Death in Diabetes: A Minor Role for CD95

Proceedings of the National Academy of Sciences - PNAS, 1998-11, Vol.95 (23), p.13818-13822

1998

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Cytotoxic T-Cells From T-Cell Receptor Transgenic NOD8.3 Mice Destroy β-Cells via the Perforin and Fas Pathways

Diabetes (New York, N.Y.), 2006-09, Vol.55 (9), p.2412-2418

2006

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Increased Susceptibility of Diabetic Mice to Influenza Virus Infection: Compromise of Collectin-Mediated Host Defense of the Lung by Glucose?

Journal of Virology, 1998-08, Vol.72 (8), p.6884-6887

1998

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The Beta Cell in Autoimmune Diabetes: Many Mechanisms and Pathways of Loss

Trends in Endocrinology & Metabolism, 2000-01, Vol.11 (1), p.11-15

2000

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Tolerance of class I histocompatibility antigens expressed extrathymically

Nature (London), 1989-06, Vol.339 (6226), p.622-624

1989

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Glucose Induces Pancreatic Islet Cell Apoptosis That Requires the BH3-Only Proteins Bim and Puma and Multi-BH Domain Protein Bax

Diabetes (New York, N.Y.), 2010-03, Vol.59 (3), p.644-652

2010

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Resistance to Celiac Disease in Humanized HLA-DR3-DQ2-Transgenic Mice Expressing Specific Anti-Gliadin CD4+ T Cells

The Journal of immunology (1950), 2009-06, Vol.182 (12), p.7440-7450

2009

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BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells

Diabetes (New York, N.Y.), 2015-09, Vol.64 (9), p.3229-3238

2015

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TNF receptor 1 deficiency increases regulatory T cell function in nonobese diabetic mice

The Journal of immunology (1950), 2011-08, Vol.187 (4), p.1702-1712

2011

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Reducing or increasing β‐cell apoptosis without inflammation does not affect diabetes initiation in neonatal NOD mice

European journal of immunology, 2011-08, Vol.41 (8), p.2238-2247

2011

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Constitutive class I-restricted exogenous presentation of self antigens in vivo

The Journal of experimental medicine, 1996-09, Vol.184 (3), p.923-930

1996

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Reducing or increasing [beta]-cell apoptosis without inflammation does not affect diabetes initiation in neonatal NOD mice

European journal of immunology, 2011-08, Vol.41 (8), p.2238-2247

2011

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Suppressor of cytokine signaling-1 overexpression protects pancreatic beta cells from CD8+ T cell-mediated autoimmune destruction

The Journal of immunology (1950), 2004-05, Vol.172 (9), p.5714-5721

2004

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CD4+ T cell help impairs CD8+ T cell deletion induced by cross-presentation of self-antigens and favors autoimmunity

The Journal of experimental medicine, 1997-12, Vol.186 (12), p.2057-2062

1997

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Transgenic expression of dominant-negative Fas-associated death domain protein in beta cells protects against Fas ligand-induced apoptosis and reduces spontaneous diabetes in nonobese diabetic mice

The Journal of immunology (1950), 2005-07, Vol.175 (1), p.293-301

2005

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Complete Diabetes Protection Despite Delayed Thymic Tolerance in NOD8.3 TCR Transgenic Mice Due to Antigen-Induced Extrathymic Deletion of T Cells

Diabetes (New York, N.Y.), 2012-02, Vol.61 (2), p.425-435

2012

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Helper requirements for generation of effector CTL to islet beta cell antigens

The Journal of immunology (1950), 2004-05, Vol.172 (9), p.5420-5426

2004

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Islet [beta]-Cells Deficient in Bcl-xL Develop but Are Abnormally Sensitive to Apoptotic Stimuli

Diabetes (New York, N.Y.), 2009-10, Vol.58 (10), p.2316

2009

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Rotavirus Infection Accelerates Type 1 Diabetes in Mice with Established Insulitis

Proapoptotic BH3-Only Protein Bid Is Essential For Death Receptor–Induced Apoptosis of Pancreatic β-Cells

Islet β-Cells Deficient in Bcl-xL Develop but Are Abnormally Sensitive to Apoptotic Stimuli

IL-1 Receptor Deficiency Slows Progression to Diabetes in the NOD Mouse

Role of SLAM in NKT cell development revealed by transgenic complementation in NOD mice

Transgenic Expression of CD95 Ligand on Islet β Cells Induces a Granulocytic Infiltration but does not Confer Immune Privilege upon Islet Allografts

Rotavirus Infection of Infant and Young Adult Nonobese Diabetic Mice Involves Extraintestinal Spread and Delays Diabetes Onset

Mechanisms of β Cell Death in Diabetes: A Minor Role for CD95

Cytotoxic T-Cells From T-Cell Receptor Transgenic NOD8.3 Mice Destroy β-Cells via the Perforin and Fas Pathways

Increased Susceptibility of Diabetic Mice to Influenza Virus Infection: Compromise of Collectin-Mediated Host Defense of the Lung by Glucose?

The Beta Cell in Autoimmune Diabetes: Many Mechanisms and Pathways of Loss

Tolerance of class I histocompatibility antigens expressed extrathymically

Glucose Induces Pancreatic Islet Cell Apoptosis That Requires the BH3-Only Proteins Bim and Puma and Multi-BH Domain Protein Bax

Resistance to Celiac Disease in Humanized HLA-DR3-DQ2-Transgenic Mice Expressing Specific Anti-Gliadin CD4+ T Cells

BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells

TNF receptor 1 deficiency increases regulatory T cell function in nonobese diabetic mice

Reducing or increasing β‐cell apoptosis without inflammation does not affect diabetes initiation in neonatal NOD mice

Constitutive class I-restricted exogenous presentation of self antigens in vivo

Reducing or increasing [beta]-cell apoptosis without inflammation does not affect diabetes initiation in neonatal NOD mice

Suppressor of cytokine signaling-1 overexpression protects pancreatic beta cells from CD8+ T cell-mediated autoimmune destruction

CD4+ T cell help impairs CD8+ T cell deletion induced by cross-presentation of self-antigens and favors autoimmunity

Transgenic expression of dominant-negative Fas-associated death domain protein in beta cells protects against Fas ligand-induced apoptosis and reduces spontaneous diabetes in nonobese diabetic mice

Complete Diabetes Protection Despite Delayed Thymic Tolerance in NOD8.3 TCR Transgenic Mice Due to Antigen-Induced Extrathymic Deletion of T Cells

Helper requirements for generation of effector CTL to islet beta cell antigens

Islet [beta]-Cells Deficient in Bcl-xL Develop but Are Abnormally Sensitive to Apoptotic Stimuli

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