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Controlling Visually Guided Behavior by Holographic Recalling of Cortical Ensembles

Cell, 2019-07, Vol.178 (2), p.447-457.e5

2019

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Stimulus encoding by specific inactivation of cortical neurons

Nature communications, 2024-04, Vol.15 (1), p.3192-3192, Article 3192

2024

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Intersecting Circuits Generate Precisely Patterned Retinal Waves

Neuron (Cambridge, Mass.), 2013-07, Vol.79 (2), p.322-334

2013

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Mapping neuronal ensembles and pattern-completion neurons through graphical models

STAR protocols, 2023-09, Vol.4 (3), p.102543, Article 102543

2023

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Molecular biology of KATP channels and implications for health and disease

IUBMB life, 2009-10, Vol.61 (10), p.971-978

2009

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Molecular biology of K ATP channels and implications for health and disease

IUBMB life, 2009-10, Vol.61 (10), p.971-978

2009

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Kir6.2 Variant E23K Increases ATP-Sensitive K+ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Diabetes (New York, N.Y.), 2009-08, Vol.58 (8), p.1869-1878

2009

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Morphology and function of three VIP-expressing amacrine cell types in the mouse retina

Journal of neurophysiology, 2015-10, Vol.114 (4), p.2431-2438

2015

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Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic KATP channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits

Channels (Austin, Tex.), 2012-03, Vol.6 (2), p.133-138

2012

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Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 ( KCNJ11 ) Mutation V290M Mutation

Diabetes (New York, N.Y.), 2010-10, Vol.60 (1), p.209-217

2010

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Differential Effects of Axon Initial Segment and Somatodendritic GABA A Receptors on Excitability Measures in Rat Dentate Granule Neurons

Journal of neurophysiology, 2011-01, Vol.105 (1), p.366-379

2011

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Scanning the Topography of Polyamine Blocker Binding in an Inwardly Rectifying Potassium Channel

The Journal of biological chemistry, 2013-03, Vol.288 (9), p.6591-6601

2013

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Circuit Mechanisms of Spontaneous and Light Evoked Activity in the Retina

Akrouh, Alejandro

2016

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HMR 1098 is not an SUR isotype specific inhibitor of heterologous or sarcolemmal KATP channels

Journal of molecular and cellular cardiology, 2011-03, Vol.50 (3), p.552-560

2011

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HMR 1098 is not an SUR isotype specific inhibitor of heterologous or sarcolemmal K ATP channels

Journal of molecular and cellular cardiology, 2011-03, Vol.50 (3), p.552-560

2011

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Differential effects of axon initial segment and somatodendritic GABAA receptors on excitability measures in rat dentate granule neurons

Journal of neurophysiology, 2011-01, Vol.105 (1), p.366-379

2011

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HMR 1098 is not an Sur Isotype Specific Inhibitor of Sarcolemmal or Heterologous KATP Channels

Biophysical journal, 2010-01, Vol.98 (3), p.137a-138a

2010

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Electrostatic Interactions Between Polyamines And Charged Adducts In The Kir Inner Cavity

Biophysical journal, 2009-02, Vol.96 (3), p.465a-465a

2009

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Compound heterozygous mutations in the SUR1 ( ABCC 8 ) subunit of pancreatic K ATP channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits

Channels (Austin, Tex.), 2012-03, Vol.6 (2), p.133-138

2012

Volltextzugriff (PDF)

Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits

Channels (Austin, Tex.), 2012-03, Vol.6 (2), p.133-138

2012

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Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: KATP Channel Inactivation Mechanism and Clinical Management

Diabetes (New York, N.Y.), 2011, Vol.60 (1), p.209-217

2011

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Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 mutation V290M mutation: [K.sub.ATP] channel inactivation mechanism and clinical management

Diabetes (New York, N.Y.), 2011-01, Vol.60 (1), p.209

2011

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Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: K^sub ATP^ Channel Inactivation Mechanism and Clinical Management

Diabetes (New York, N.Y.), 2011-01, Vol.60 (1), p.209

2011

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Kir6.2 variant E23K increases ATP-sensitive [K.sup.+] channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance

Diabetes (New York, N.Y.), 2009-08, Vol.58 (8), p.1869

2009

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Molecular biology of K(ATP) channels and implications for health and disease

IUBMB life, 2009-10, Vol.61 (10), p.971-978

2009

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Controlling Visually Guided Behavior by Holographic Recalling of Cortical Ensembles

Stimulus encoding by specific inactivation of cortical neurons

Intersecting Circuits Generate Precisely Patterned Retinal Waves

Mapping neuronal ensembles and pattern-completion neurons through graphical models

Molecular biology of KATP channels and implications for health and disease

Molecular biology of K ATP channels and implications for health and disease

Kir6.2 Variant E23K Increases ATP-Sensitive K+ Channel Activity and Is Associated With Impaired Insulin Release and Enhanced Insulin Sensitivity in Adults With Normal Glucose Tolerance

Morphology and function of three VIP-expressing amacrine cell types in the mouse retina

Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic KATP channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 ( KCNJ11 ) Mutation V290M Mutation

Differential Effects of Axon Initial Segment and Somatodendritic GABA A Receptors on Excitability Measures in Rat Dentate Granule Neurons

Scanning the Topography of Polyamine Blocker Binding in an Inwardly Rectifying Potassium Channel

Circuit Mechanisms of Spontaneous and Light Evoked Activity in the Retina

HMR 1098 is not an SUR isotype specific inhibitor of heterologous or sarcolemmal KATP channels

HMR 1098 is not an SUR isotype specific inhibitor of heterologous or sarcolemmal K ATP channels

Differential effects of axon initial segment and somatodendritic GABAA receptors on excitability measures in rat dentate granule neurons

HMR 1098 is not an Sur Isotype Specific Inhibitor of Sarcolemmal or Heterologous KATP Channels

Electrostatic Interactions Between Polyamines And Charged Adducts In The Kir Inner Cavity

Compound heterozygous mutations in the SUR1 ( ABCC 8 ) subunit of pancreatic K ATP channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits

Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: KATP Channel Inactivation Mechanism and Clinical Management

Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 mutation V290M mutation: [K.sub.ATP] channel inactivation mechanism and clinical management

Congenital Hyperinsulinism and Glucose Hypersensitivity in Homozygous and Heterozygous Carriers of Kir6.2 (KCNJ11) Mutation V290M Mutation: K^sub ATP^ Channel Inactivation Mechanism and Clinical Management

Kir6.2 variant E23K increases ATP-sensitive [K.sup.+] channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance

Molecular biology of K(ATP) channels and implications for health and disease

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