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The Inflammatory Chemokine CCL5 and Cancer Progression

Mediators of Inflammation, 2014-01, Vol.2014 (3), p.7-18

2014

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Inhibition of the CCL5/CCR5 Axis against the Progression of Gastric Cancer

International journal of molecular sciences, 2018-05, Vol.19 (5), p.1477

2018

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The CCL5/CCR5 Axis in Cancer Progression

Cancers, 2020-07, Vol.12 (7), p.1765

2020

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Formation of the Immunosuppressive Microenvironment of Classic Hodgkin Lymphoma and Therapeutic Approaches to Counter It

International journal of molecular sciences, 2019-05, Vol.20 (10), p.2416

2019

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CCR5 antagonism by maraviroc inhibits Hodgkin lymphoma microenvironment interactions and xenograft growth

Haematologica (Roma), 2019-03, Vol.104 (3), p.564-575

2019

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In ovarian cancer maraviroc potentiates the antitumoral activity and further inhibits the formation of a tumor-promoting microenvironment by trabectedin

Biomedicine & pharmacotherapy, 2024-03, Vol.172, p.116296-116296, Article 116296

2024

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In classical Hodgkin lymphoma the combination of the CCR5 antagonist maraviroc with trabectedin synergizes, enhances DNA damage and decreases three-dimensional tumor-stroma heterospheroid viability

Haematologica (Roma), 2022-01, Vol.107 (1), p.287-291

2022

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In Ovarian Cancer Multicellular Spheroids, Platelet Releasate Promotes Growth, Expansion of ALDH+ and CD133+ Cancer Stem Cells, and Protection against the Cytotoxic Effects of Cisplatin, Carboplatin and Paclitaxel

International journal of molecular sciences, 2021-03, Vol.22 (6), p.3019

2021

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Analogs of a Natural Peptaibol Exert Anticancer Activity in Both Cisplatin- and Doxorubicin-Resistant Cells and in Multicellular Tumor Spheroids

International journal of molecular sciences, 2021-08, Vol.22 (16), p.8362

2021

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Adipose-Derived Stem Cells Primed with Paclitaxel Inhibit Ovarian Cancer Spheroid Growth and Overcome Paclitaxel Resistance

Pharmaceutics, 2020-04, Vol.12 (5), p.401

2020

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The role of the EGFR signaling in tumor microenvironment

Journal of cellular physiology, 2008-03, Vol.214 (3), p.559-567

2008

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In Doxorubicin-Adapted Hodgkin Lymphoma Cells, Acquiring Multidrug Resistance and Improved Immunosuppressive Abilities, Doxorubicin Activity Was Enhanced by Chloroquine and GW4869

Cells (Basel, Switzerland), 2023-11, Vol.12 (23), p.2732

2023

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Dinuclear gold(I) complexes based on carbene and diphosphane ligands: bis[2-(dicyclohexylphosphano)ethyl]amine complex inhibits the proteasome activity, decreases stem cell markers and spheroid viability in lung cancer cells

Journal of biological inorganic chemistry, 2023-12, Vol.28 (8), p.751-766

2023

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Expression of CCR5 receptors on Reed–Sternberg cells and Hodgkin lymphoma cell lines: Involvement of CCL5/Rantes in tumor cell growth and microenvironmental interactions

International journal of cancer, 2008-02, Vol.122 (4), p.769-776

2008

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Role of the EGFR ligand/receptor system in the secretion of angiogenic factors in mesenchymal stem cells

Journal of cellular physiology, 2011-08, Vol.226 (8), p.2131-2138

2011

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Improved GMP compliant approach to manipulate lipoaspirates, to cryopreserve stromal vascular fraction, and to expand adipose stem cells in xeno-free media

Stem cell research & therapy, 2018-05, Vol.9 (1), p.130-130, Article 130

2018

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Latest insights into the anticancer activity of gold(III)-dithiocarbamato complexes

Anti-cancer agents in medicinal chemistry, 2010-05, Vol.10 (4), p.283-292

2010

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Gefitinib inhibits the cross-talk between mesenchymal stem cells and prostate cancer cells leading to tumor cell proliferation and inhibition of docetaxel activity

Journal of cellular biochemistry, 2013-05, Vol.114 (5), p.1135-1144

2013

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Functional coexpression of Interleukin (IL)‐7 and its receptor (IL‐7R) on Hodgkin and Reed‐Sternberg cells: Involvement of IL‐7 in tumor cell growth and microenvironmental interactions of Hodgkin's lymphoma

International journal of cancer, 2009-09, Vol.125 (5), p.1092-1101

2009

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Microenvironmental interactions in classical Hodgkin lymphoma and their role in promoting tumor growth, immune escape and drug resistance

Cancer letters, 2016-09, Vol.380 (1), p.243-252

2016

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Bortezomib down-modulates the survival factor interferon regulatory factor 4 in Hodgkin lymphoma cell lines and decreases the protective activity of Hodgkin lymphoma-associated fibroblasts

Leukemia & lymphoma, 2014-01, Vol.55 (1), p.149-159

2014

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Trabectedin overcomes doxorubicin-resistance, counteracts tumor-immunosuppressive reprogramming of monocytes and decreases xenograft growth in Hodgkin lymphoma

Cancer letters, 2021-03, Vol.500, p.182-193

2021

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Comment on “Fibroblasts in Nodular Sclerosing Classical Hodgkin Lymphoma Are Defined by a Specific Phenotype and Protect Tumor Cells from Brentuximab-Vedotin Induced Injury” Cancers 2019, 11, 1687

Cancers, 2020-02, Vol.12 (2), p.343

2020

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Comment on "Fibroblasts in Nodular SclerosingClassical Hodgkin Lymphoma Are Defined by a Specific Phenotype and Protect Tumor Cells from Brentuximab-Vedotin Induced Injury" Cancers 2019, 11, 1687

Cancers, 2020-02, Vol.12 (2)

2020

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Current and Emerging Approaches to Study Microenvironmental Interactions and Drug Activity in Classical Hodgkin Lymphoma

Cancers, 2022-05, Vol.14 (10), p.2427

2022

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The Inflammatory Chemokine CCL5 and Cancer Progression

Inhibition of the CCL5/CCR5 Axis against the Progression of Gastric Cancer

The CCL5/CCR5 Axis in Cancer Progression

Formation of the Immunosuppressive Microenvironment of Classic Hodgkin Lymphoma and Therapeutic Approaches to Counter It

CCR5 antagonism by maraviroc inhibits Hodgkin lymphoma microenvironment interactions and xenograft growth

In ovarian cancer maraviroc potentiates the antitumoral activity and further inhibits the formation of a tumor-promoting microenvironment by trabectedin

In classical Hodgkin lymphoma the combination of the CCR5 antagonist maraviroc with trabectedin synergizes, enhances DNA damage and decreases three-dimensional tumor-stroma heterospheroid viability

In Ovarian Cancer Multicellular Spheroids, Platelet Releasate Promotes Growth, Expansion of ALDH+ and CD133+ Cancer Stem Cells, and Protection against the Cytotoxic Effects of Cisplatin, Carboplatin and Paclitaxel

Analogs of a Natural Peptaibol Exert Anticancer Activity in Both Cisplatin- and Doxorubicin-Resistant Cells and in Multicellular Tumor Spheroids

Adipose-Derived Stem Cells Primed with Paclitaxel Inhibit Ovarian Cancer Spheroid Growth and Overcome Paclitaxel Resistance

The role of the EGFR signaling in tumor microenvironment

In Doxorubicin-Adapted Hodgkin Lymphoma Cells, Acquiring Multidrug Resistance and Improved Immunosuppressive Abilities, Doxorubicin Activity Was Enhanced by Chloroquine and GW4869

Dinuclear gold(I) complexes based on carbene and diphosphane ligands: bis[2-(dicyclohexylphosphano)ethyl]amine complex inhibits the proteasome activity, decreases stem cell markers and spheroid viability in lung cancer cells

Expression of CCR5 receptors on Reed–Sternberg cells and Hodgkin lymphoma cell lines: Involvement of CCL5/Rantes in tumor cell growth and microenvironmental interactions

Role of the EGFR ligand/receptor system in the secretion of angiogenic factors in mesenchymal stem cells

Improved GMP compliant approach to manipulate lipoaspirates, to cryopreserve stromal vascular fraction, and to expand adipose stem cells in xeno-free media

Latest insights into the anticancer activity of gold(III)-dithiocarbamato complexes

Gefitinib inhibits the cross-talk between mesenchymal stem cells and prostate cancer cells leading to tumor cell proliferation and inhibition of docetaxel activity

Functional coexpression of Interleukin (IL)‐7 and its receptor (IL‐7R) on Hodgkin and Reed‐Sternberg cells: Involvement of IL‐7 in tumor cell growth and microenvironmental interactions of Hodgkin's lymphoma

Microenvironmental interactions in classical Hodgkin lymphoma and their role in promoting tumor growth, immune escape and drug resistance

Bortezomib down-modulates the survival factor interferon regulatory factor 4 in Hodgkin lymphoma cell lines and decreases the protective activity of Hodgkin lymphoma-associated fibroblasts

Trabectedin overcomes doxorubicin-resistance, counteracts tumor-immunosuppressive reprogramming of monocytes and decreases xenograft growth in Hodgkin lymphoma

Comment on “Fibroblasts in Nodular Sclerosing Classical Hodgkin Lymphoma Are Defined by a Specific Phenotype and Protect Tumor Cells from Brentuximab-Vedotin Induced Injury” Cancers 2019, 11, 1687

Comment on "Fibroblasts in Nodular SclerosingClassical Hodgkin Lymphoma Are Defined by a Specific Phenotype and Protect Tumor Cells from Brentuximab-Vedotin Induced Injury" Cancers 2019, 11, 1687

Current and Emerging Approaches to Study Microenvironmental Interactions and Drug Activity in Classical Hodgkin Lymphoma

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