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Cdc48/p97 segregase is modulated by cyclin‐dependent kinase to determine cyclin fate during G1 progression

The EMBO journal, 2018-08, Vol.37 (16), p.n/a

2018

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The critical size is set at a single-cell level by growth rate to attain homeostasis and adaptation

Nature communications, 2012, Vol.3 (1), p.1012-1012, Article 1012

2012

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Proteostasis collapse, a hallmark of aging, hinders the chaperone-Start network and arrests cells in G1

eLife, 2019-09, Vol.8

2019

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Centromeric signaling proteins boost G1 cyclin degradation and modulate cell size in budding yeast

PLoS biology, 2018-08, Vol.16 (8), p.e2005388-e2005388

2018

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Cyclin Cln3 Is Retained at the ER and Released by the J Chaperone Ydj1 in Late G1 to Trigger Cell Cycle Entry

Molecular cell, 2007-06, Vol.26 (5), p.649-662

2007

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Growth Rate as a Direct Regulator of the Start Network to Set Cell Size

Frontiers in cell and developmental biology, 2017-05, Vol.5, p.57-57

2017

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Whi3, a Developmental Regulator of Budding Yeast, Binds a Large Set of mRNAs Functionally Related to the Endoplasmic Reticulum

The Journal of biological chemistry, 2008-10, Vol.283 (42), p.28670-28679

2008

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Protein Kinase KIS Localizes to RNA Granules and Enhances Local Translation

Molecular and Cellular Biology, 2009-02, Vol.29 (3), p.726-735

2009

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Bck2 is a phase-independent activator of cell cycle-regulated genes in yeast

Cell cycle (Georgetown, Tex.), 2009-01, Vol.8 (2), p.239-252

2009

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Proteostatic stress as a nodal hallmark of replicative aging

Experimental cell research, 2020-09, Vol.394 (2), p.112163-112163, Article 112163

2020

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Chaperone-Dependent Degradation of Cdc42 Promotes Cell Polarity and Shields the Protein from Aggregation

Molecular and cellular biology, 2023-05, Vol.43 (5), p.200-222

2023

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Phosphorylation of Hsl1 by Hog1 leads to a G2 arrest essential for cell survival at high osmolarity

The EMBO journal, 2006-06, Vol.25 (11), p.2338-2346

2006

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Control of Cell Cycle and Cell Growth by Molecular Chaperones

Cell cycle (Georgetown, Tex.), 2007-11, Vol.6 (21), p.2599-2603

2007

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Coincidence Analysis of Molecular Dynamics by Raster Image Correlation Spectroscopy

Methods in molecular biology (Clifton, N.J.), 2019, Vol.2040, p.375

2019

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Compartmentalization of ER-Bound Chaperone Confines Protein Deposit Formation to the Aging Yeast Cell

Current biology, 2017-03, Vol.27 (6), p.773-783

2017

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Whi3 regulates morphogenesis in budding yeast by enhancing Cdk functions in apical growth

Cell cycle (Georgetown, Tex.), 2009-06, Vol.8 (12), p.1912-1920

2009

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A Whi7-Anchored Loop Controls the G1 Cdk-Cyclin Complex at Start

Molecular cell, 2014-01, Vol.53 (1), p.115-126

2014

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The transcriptional network activated by Cln3 cyclin at the G1-to-S transition of the yeast cell cycle

Genome biology, 2010-06, Vol.11 (6), p.R67-R67

2010

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Biogenesis of Yeast Telomerase Depends on the Importin Mtr10

Molecular and Cellular Biology, 2002-09, Vol.22 (17), p.6046-6055

2002

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1,25-Dihydroxyvitamin D 3 regulates VEGF production through a vitamin D response element in the VEGF promoter

Atherosclerosis, 2009-05, Vol.204 (1), p.85-89

2009

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Recruitment of Cdc28 by Whi3 restricts nuclear accumulation of the G1 cyclin-Cdk complex to late G1

The EMBO journal, 2004-01, Vol.23 (1), p.180-190

2004

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KIS, a kinase associated with microtubule regulators, enhances translation of AMPA receptors and stimulates dendritic spine remodeling

The Journal of neuroscience, 2014-10, Vol.34 (42), p.13988-13997

2014

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Transcriptomic balance and optimal growth are determined by cell size

Molecular cell, 2024-09, Vol.84 (17), p.3288-3301.e3

2024

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Stress granules display bistable dynamics modulated by Cdk

The Journal of cell biology, 2021-03, Vol.220 (3), p.1

2021

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1,25-Dihydroxyvitamin D3 regulates VEGF production through a vitamin D response element in the VEGF promoter

Atherosclerosis, 2009-05, Vol.204 (1), p.85-89

2009

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Cdc48/p97 segregase is modulated by cyclin‐dependent kinase to determine cyclin fate during G1 progression

The critical size is set at a single-cell level by growth rate to attain homeostasis and adaptation

Proteostasis collapse, a hallmark of aging, hinders the chaperone-Start network and arrests cells in G1

Centromeric signaling proteins boost G1 cyclin degradation and modulate cell size in budding yeast

Cyclin Cln3 Is Retained at the ER and Released by the J Chaperone Ydj1 in Late G1 to Trigger Cell Cycle Entry

Growth Rate as a Direct Regulator of the Start Network to Set Cell Size

Whi3, a Developmental Regulator of Budding Yeast, Binds a Large Set of mRNAs Functionally Related to the Endoplasmic Reticulum

Protein Kinase KIS Localizes to RNA Granules and Enhances Local Translation

Bck2 is a phase-independent activator of cell cycle-regulated genes in yeast

Proteostatic stress as a nodal hallmark of replicative aging

Chaperone-Dependent Degradation of Cdc42 Promotes Cell Polarity and Shields the Protein from Aggregation

Phosphorylation of Hsl1 by Hog1 leads to a G2 arrest essential for cell survival at high osmolarity

Control of Cell Cycle and Cell Growth by Molecular Chaperones

Coincidence Analysis of Molecular Dynamics by Raster Image Correlation Spectroscopy

Compartmentalization of ER-Bound Chaperone Confines Protein Deposit Formation to the Aging Yeast Cell

Whi3 regulates morphogenesis in budding yeast by enhancing Cdk functions in apical growth

A Whi7-Anchored Loop Controls the G1 Cdk-Cyclin Complex at Start

The transcriptional network activated by Cln3 cyclin at the G1-to-S transition of the yeast cell cycle

Biogenesis of Yeast Telomerase Depends on the Importin Mtr10

1,25-Dihydroxyvitamin D 3 regulates VEGF production through a vitamin D response element in the VEGF promoter

Recruitment of Cdc28 by Whi3 restricts nuclear accumulation of the G1 cyclin-Cdk complex to late G1

KIS, a kinase associated with microtubule regulators, enhances translation of AMPA receptors and stimulates dendritic spine remodeling

Transcriptomic balance and optimal growth are determined by cell size

Stress granules display bistable dynamics modulated by Cdk

1,25-Dihydroxyvitamin D3 regulates VEGF production through a vitamin D response element in the VEGF promoter

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