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MicroRNA Regulation of Intestinal Epithelial Tight Junction Permeability

Gastroenterology (New York, N.Y. 1943), 2011-10, Vol.141 (4), p.1323-1333

2011

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Mechanism of cytokine modulation of epithelial tight junction barrier

Frontiers in bioscience, 2009-01, Vol.14 (7), p.2765-2778

2009

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IL-1β and the Intestinal Epithelial Tight Junction Barrier

Frontiers in immunology, 2021-10, Vol.12, p.767456-767456

2021

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Lipopolysaccharide Causes an Increase in Intestinal Tight Junction Permeability in Vitro and in Vivo by Inducing Enterocyte Membrane Expression and Localization of TLR-4 and CD14

The American journal of pathology, 2013-02, Vol.182 (2), p.375-387

2013

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IL1B Increases Intestinal Tight Junction Permeability by Up-regulation of MIR200C-3p, Which Degrades Occludin mRNA

Gastroenterology (New York, N.Y. 1943), 2020-10, Vol.159 (4), p.1375-1389

2020

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Lipopolysaccharide Regulation of Intestinal Tight Junction Permeability Is Mediated by TLR4 Signal Transduction Pathway Activation of FAK and MyD88

The Journal of immunology (1950), 2015-11, Vol.195 (10), p.4999-5010

2015

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Lipopolysaccharide-Induced Increase in Intestinal Epithelial Tight Permeability Is Mediated by Toll-Like Receptor 4/Myeloid Differentiation Primary Response 88 (MyD88) Activation of Myosin Light Chain Kinase Expression

The American journal of pathology, 2017-12, Vol.187 (12), p.2698-2710

2017

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IL-1β Causes an Increase in Intestinal Epithelial Tight Junction Permeability

Journal of Immunology, 2007-04, Vol.178 (7), p.4641-4649

2007

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Occludin regulates macromolecule flux across the intestinal epithelial tight junction barrier

American journal of physiology: Gastrointestinal and liver physiology, 2011-06, Vol.300 (6), p.G1054-G1064

2011

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Interleukin-6 modulation of intestinal epithelial tight junction permeability is mediated by JNK pathway activation of claudin-2 gene

PloS one, 2014-03, Vol.9 (3), p.e85345

2014

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Lipopolysaccharide-Induced Increase in Intestinal Permeability Is Mediated by TAK-1 Activation of IKK and MLCK/MYLK Gene

The American journal of pathology, 2019-04, Vol.189 (4), p.797-812

2019

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Matrix Metalloproteinase-9 (MMP-9) induced disruption of intestinal epithelial tight junction barrier is mediated by NF-κB activation

PloS one, 2021-04, Vol.16 (4), p.e0249544-e0249544

2021

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Bifidobacterium bifidum Enhances the Intestinal Epithelial Tight Junction Barrier and Protects against Intestinal Inflammation by Targeting the Toll-like Receptor-2 Pathway in an NF-κB-Independent Manner

International journal of molecular sciences, 2021-08, Vol.22 (15), p.8070

2021

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Editorial: Intestinal epithelial barrier disruption by enteric pathogens

Frontiers in cellular and infection microbiology, 2023-01, Vol.13, p.1134753-1134753

2023

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Editorial: Role of matrix metalloproteinases and other inflammatory mediators in the disruption of the intestinal tight junction barrier

Frontiers in immunology, 2023, Vol.14, p.1194827-1194827

2023

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Lactobacillus acidophilus inhibits the TNF-α-induced increase in intestinal epithelial tight junction permeability via a TLR-2 and PI3K-dependent inhibition of NF-κB activation

Frontiers in immunology, 2024-07, Vol.15, p.1348010

2024

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Cellular and molecular mechanism of interleukin‐1β modulation of Caco‐2 intestinal epithelial tight junction barrier

Journal of cellular and molecular medicine, 2011-04, Vol.15 (4), p.970-982

2011

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Talk about micromanaging! Role of microRNAs in intestinal barrier function

American journal of physiology: Gastrointestinal and liver physiology, 2020-08, Vol.319 (2), p.G170-G174

2020

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Bifidobacterium bifidum inhibits the TNF-α induced increase in intestinal epithelial tight junction permeability via a TLR-2/TLR-6 and PPAR-γ dependent inhibition of NF-κB p50/p65

Physiology (Bethesda, Md.), 2024-05, Vol.39 (S1)

2024

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Bifidobacterium bifidum prevents the IL-1B induced increase in intestinal permeability by a novel mechanism: TLR-2 dependent activation of PPAR-gamma and inhibition of NF-kB signaling pathway

Physiology (Bethesda, Md.), 2023-05, Vol.38 (S1)

2023

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Bifidobacterium bifidum causes an enhancement of the intestinal epithelial tight junction barrier is mediated by TLR-2 dependent increases of Toll-interacting protein (TOLLIP) expression in a MYD88 independent manner

Physiology (Bethesda, Md.), 2023-05, Vol.38 (S1)

2023

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TNF-α Modulation of Intestinal Tight Junction Permeability Is Mediated by NIK/IKK-α Axis Activation of the Canonical NF-κB Pathway

The American journal of pathology, 2016-05, Vol.186 (5), p.1151-1165

2016

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MATRIX METALLOPROTEINASE (MMP-9) INDUCED INCREASE IN INTESTINAL EPITHELIAL TIGHT JUNCTION BARRIER IS MEDIATED BY EGFR ACTIVATION

Gastroenterology (New York, N.Y. 1943), 2022-02, Vol.162 (3), p.S50-S50

2022

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Lactobacillus acidophilus causes enhancement of the intestinal tight junction barrier by a toll-like receptor-2-dependent increase in occludin

Journal of allergy and clinical immunology, 2022-02, Vol.149 (2), p.AB99-AB99

2022

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MATRIX METALLOPROTEINASE (MMP-9) INDUCED INCREASE IN INTESTINAL EPITHELIAL TIGHT JUNCTION BARRIER IS MEDIATED BY EGFR ACTIVATION

Inflammatory bowel diseases, 2022-01, Vol.28 (Supplement_1), p.S50-S50

2022

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MicroRNA Regulation of Intestinal Epithelial Tight Junction Permeability

Mechanism of cytokine modulation of epithelial tight junction barrier

IL-1β and the Intestinal Epithelial Tight Junction Barrier

Lipopolysaccharide Causes an Increase in Intestinal Tight Junction Permeability in Vitro and in Vivo by Inducing Enterocyte Membrane Expression and Localization of TLR-4 and CD14

IL1B Increases Intestinal Tight Junction Permeability by Up-regulation of MIR200C-3p, Which Degrades Occludin mRNA

Lipopolysaccharide Regulation of Intestinal Tight Junction Permeability Is Mediated by TLR4 Signal Transduction Pathway Activation of FAK and MyD88

Lipopolysaccharide-Induced Increase in Intestinal Epithelial Tight Permeability Is Mediated by Toll-Like Receptor 4/Myeloid Differentiation Primary Response 88 (MyD88) Activation of Myosin Light Chain Kinase Expression

IL-1β Causes an Increase in Intestinal Epithelial Tight Junction Permeability

Occludin regulates macromolecule flux across the intestinal epithelial tight junction barrier

Interleukin-6 modulation of intestinal epithelial tight junction permeability is mediated by JNK pathway activation of claudin-2 gene

Lipopolysaccharide-Induced Increase in Intestinal Permeability Is Mediated by TAK-1 Activation of IKK and MLCK/MYLK Gene

Matrix Metalloproteinase-9 (MMP-9) induced disruption of intestinal epithelial tight junction barrier is mediated by NF-κB activation

Bifidobacterium bifidum Enhances the Intestinal Epithelial Tight Junction Barrier and Protects against Intestinal Inflammation by Targeting the Toll-like Receptor-2 Pathway in an NF-κB-Independent Manner

Editorial: Intestinal epithelial barrier disruption by enteric pathogens

Editorial: Role of matrix metalloproteinases and other inflammatory mediators in the disruption of the intestinal tight junction barrier

Lactobacillus acidophilus inhibits the TNF-α-induced increase in intestinal epithelial tight junction permeability via a TLR-2 and PI3K-dependent inhibition of NF-κB activation

Cellular and molecular mechanism of interleukin‐1β modulation of Caco‐2 intestinal epithelial tight junction barrier

Talk about micromanaging! Role of microRNAs in intestinal barrier function

Bifidobacterium bifidum inhibits the TNF-α induced increase in intestinal epithelial tight junction permeability via a TLR-2/TLR-6 and PPAR-γ dependent inhibition of NF-κB p50/p65

Bifidobacterium bifidum prevents the IL-1B induced increase in intestinal permeability by a novel mechanism: TLR-2 dependent activation of PPAR-gamma and inhibition of NF-kB signaling pathway

Bifidobacterium bifidum causes an enhancement of the intestinal epithelial tight junction barrier is mediated by TLR-2 dependent increases of Toll-interacting protein (TOLLIP) expression in a MYD88 independent manner

TNF-α Modulation of Intestinal Tight Junction Permeability Is Mediated by NIK/IKK-α Axis Activation of the Canonical NF-κB Pathway

MATRIX METALLOPROTEINASE (MMP-9) INDUCED INCREASE IN INTESTINAL EPITHELIAL TIGHT JUNCTION BARRIER IS MEDIATED BY EGFR ACTIVATION

Lactobacillus acidophilus causes enhancement of the intestinal tight junction barrier by a toll-like receptor-2-dependent increase in occludin

MATRIX METALLOPROTEINASE (MMP-9) INDUCED INCREASE IN INTESTINAL EPITHELIAL TIGHT JUNCTION BARRIER IS MEDIATED BY EGFR ACTIVATION

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