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Soil humic acids degrade CWD prions and reduce infectivity

PLoS pathogens, 2018-11, Vol.14 (11), p.e1007414-e1007414

2018

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Remdesivir does not affect mitochondrial DNA copy number or deletion mutation frequency in aged male rats: A short report

PloS one, 2022-10, Vol.17 (10), p.e0271850

2022

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Apoptosis and necrosis mediate skeletal muscle fiber loss in age‐induced mitochondrial enzymatic abnormalities

Aging cell, 2015-12, Vol.14 (6), p.1085-1093

2015

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Chronic Wasting Disease Prion Strain Emergence and Host Range Expansion

Emerging infectious diseases, 2017-09, Vol.23 (9), p.1598-1600

2017

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Latent mitochondrial DNA deletion mutations drive muscle fiber loss at old age

Aging cell, 2016-12, Vol.15 (6), p.1132-1139

2016

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Detection of Chronic Wasting Disease Prions in Prairie Soils from Endemic Regions

Environmental science & technology, 2024-06, Vol.58 (25), p.10932-10940

2024

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Neural transcriptomic signature of chronic wasting disease in white-tailed deer

BMC genomics, 2022-01, Vol.23 (1), p.69-69, Article 69

2022

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Prions adhere to soil minerals and remain infectious

PLoS pathogens, 2006-04, Vol.2 (4), p.e32

2006

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Mitochondrial DNA–Deletion Mutations Accumulate Intracellularly to Detrimental Levels in Aged Human Skeletal Muscle Fibers

American journal of human genetics, 2006-09, Vol.79 (3), p.469-480

2006

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Oral transmissibility of prion disease is enhanced by binding to soil particles

PLoS pathogens, 2007-07, Vol.3 (7), p.e93-e93

2007

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Sheep scrapie and deer rabies in England prior to 1800

Prion, 2023-12, Vol.17 (1), p.7-15

2023

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Chronic wasting disease: a cervid prion infection looming to spillover

Veterinary Research, 2021-09, Vol.52 (1), p.1-115, Article 115

2021

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Movement of Chronic Wasting Disease Prions in Prairie, Boreal and Alpine Soils

Pathogens (Basel), 2023-02, Vol.12 (2), p.269

2023

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Prion protein lowering is a disease-modifying therapy across prion disease stages, strains and endpoints

Nucleic acids research, 2020-11, Vol.48 (19), p.10615-10631

2020

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Nanopore sequencing identifies a higher frequency and expanded spectrum of mitochondrial DNA deletion mutations in human aging

Aging cell, 2023-06, Vol.22 (6), p.e13842-n/a

2023

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White-tailed deer S96 prion protein does not support stable in vitro propagation of most common CWD strains

Scientific reports, 2021-05, Vol.11 (1), p.11193-11193, Article 11193

2021

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Mitochondrial DNA deletion mutations increase exponentially with age in human skeletal muscle

Aging clinical and experimental research, 2021-07, Vol.33 (7), p.1811-1820

2021

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Deer Prion Proteins Modulate the Emergence and Adaptation of Chronic Wasting Disease Strains

Journal of virology, 2015-12, Vol.89 (24), p.12362-12373

2015

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Chronic wasting disease prions in mule deer interdigital glands

PloS one, 2022-10, Vol.17 (10), p.e0275375-e0275375

2022

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Mitochondrial biogenesis drives a vicious cycle of metabolic insufficiency and mitochondrial DNA deletion mutation accumulation in aged rat skeletal muscle fibers

PloS one, 2013-03, Vol.8 (3), p.e59006

2013

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Highly efficient amplification of chronic wasting disease agent by protein misfolding cyclic amplification with beads (PMCAb)

PloS one, 2012-04, Vol.7 (4), p.e35383-e35383

2012

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Novel Sarcopenia-related Alterations in Sarcomeric Protein Post-translational Modifications (PTMs) in Skeletal Muscles Identified by Top-down Proteomics

Molecular & cellular proteomics, 2018-01, Vol.17 (1), p.134-145

2018

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Long-Term Incubation PrPCWD with Soils Affects Prion Recovery but Not Infectivity

Pathogens (Basel), 2020-04, Vol.9 (4), p.311

2020

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Metformin Treatment in Old Rats and Effects on Mitochondrial Integrity

Rejuvenation research, 2021-12, Vol.24 (6), p.434-440

2021

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Long term rapamycin treatment improves mitochondrial DNA quality in aging mice

Experimental gerontology, 2018-06, Vol.106, p.125-131

2018

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Soil humic acids degrade CWD prions and reduce infectivity

Remdesivir does not affect mitochondrial DNA copy number or deletion mutation frequency in aged male rats: A short report

Apoptosis and necrosis mediate skeletal muscle fiber loss in age‐induced mitochondrial enzymatic abnormalities

Chronic Wasting Disease Prion Strain Emergence and Host Range Expansion

Latent mitochondrial DNA deletion mutations drive muscle fiber loss at old age

Detection of Chronic Wasting Disease Prions in Prairie Soils from Endemic Regions

Neural transcriptomic signature of chronic wasting disease in white-tailed deer

Prions adhere to soil minerals and remain infectious

Mitochondrial DNA–Deletion Mutations Accumulate Intracellularly to Detrimental Levels in Aged Human Skeletal Muscle Fibers

Oral transmissibility of prion disease is enhanced by binding to soil particles

Sheep scrapie and deer rabies in England prior to 1800

Chronic wasting disease: a cervid prion infection looming to spillover

Movement of Chronic Wasting Disease Prions in Prairie, Boreal and Alpine Soils

Prion protein lowering is a disease-modifying therapy across prion disease stages, strains and endpoints

Nanopore sequencing identifies a higher frequency and expanded spectrum of mitochondrial DNA deletion mutations in human aging

White-tailed deer S96 prion protein does not support stable in vitro propagation of most common CWD strains

Mitochondrial DNA deletion mutations increase exponentially with age in human skeletal muscle

Deer Prion Proteins Modulate the Emergence and Adaptation of Chronic Wasting Disease Strains

Chronic wasting disease prions in mule deer interdigital glands

Mitochondrial biogenesis drives a vicious cycle of metabolic insufficiency and mitochondrial DNA deletion mutation accumulation in aged rat skeletal muscle fibers

Highly efficient amplification of chronic wasting disease agent by protein misfolding cyclic amplification with beads (PMCAb)

Novel Sarcopenia-related Alterations in Sarcomeric Protein Post-translational Modifications (PTMs) in Skeletal Muscles Identified by Top-down Proteomics

Long-Term Incubation PrPCWD with Soils Affects Prion Recovery but Not Infectivity

Metformin Treatment in Old Rats and Effects on Mitochondrial Integrity

Long term rapamycin treatment improves mitochondrial DNA quality in aging mice

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