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Quercetin prevents cardiac hypertrophy induced by pressure overload in rats

Journal of Veterinary Medical Science, 2009, Vol.71(6), pp.737-743

2009

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Mechanisms of the inhibitory effects of epigallocatechin‐3 gallate on platelet‐derived growth factor‐BB‐induced cell signaling and mitogenesis

The FASEB journal, 2004-01, Vol.18 (1), p.128-130

2004

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Angiotensin II suppresses adenosine monophosphate-activated protein kinase of podocytes via angiotensin II type 1 receptor and mitogen-activated protein kinase signaling

Clinical and experimental nephrology, 2013-02, Vol.17 (1), p.16-23

2013

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Epigallocatechin-3 gallate prevents cardiac hypertrophy induced by pressure overload in rats

Journal of veterinary science (Suwŏn-si, Korea), 2007-06, Vol.8 (2), p.121-129

2007

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Activation of Akt/protein kinase B mediates the protective effects of mechanical stretching against myocardial ischemia-reperfusion injury

Journal of Veterinary Science, 2012, 13(3), , pp.235-244

2012

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Angiotensin II Modulates p130Cas of Podocytes by the Suppression of AMP-Activated Protein Kinase

Journal of Korean Medical Science, 2016, 31(4), 214, pp.535-541

2016

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(–)-Epigallocatechin-3-O-gallate (EGCG) attenuates the hemodynamics stimulated by caffeine through decrease of catecholamines release

Archives of Pharmacal Research, 2016, 39(9), , pp.1307-1312

2016

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Mechanical Stretch-Induced Protection against Myocardial Ischemia- Reperfusion Injury Involves AMP-Activated Protein Kinase

The Korean Journal of Physiology & Pharmacology, 2010, 14(1), , pp.1-9

2010

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Epigallocatechin-3-O-Gallate Inhibits the Angiotensin II-Induced Adhesion Molecule Expression in Human Umbilical Vein Endothelial Cell Via Inhibition of MAPK Pathways

Cellular physiology and biochemistry, 2007-01, Vol.20 (6), p.859-866

2007

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Inhibition of the PDGF beta-receptor tyrosine phosphorylation and its downstream intracellular signal transduction pathway in rat and human vascular smooth muscle cells by different catechins

The FASEB journal, 2002-06, Vol.16 (8), p.893-895

2002

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p38 Mitogen-Activated Protein Kinase Regulates Vasoconstriction in Spontaneously Hypertensive Rats

Journal of Pharmacological Sciences, 2004, Vol.95(2), pp.267-272

2004

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Epigallocathechin-3 gallate selectively inhibits the PDGF-BB-induced intracellular signaling transduction pathway in vascular smooth muscle cells and inhibits transformation of sis-transfected NIH 3T3 fibroblasts and human glioblastoma cells (A172)

Molecular biology of the cell, 1999-04, Vol.10 (4), p.1093-1104

1999

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Differential expression of αB-crystallin causes maturation-dependent susceptibility of oligodendrocytes to oxidative stress

BMB reports, 2013-10, Vol.46 (10), p.501-506

2013

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Vasodilating and hypotensive effects of fangchinoline and tetrandrine on the rat aorta and the stroke-prone spontaneously hypertensive rat

Journal of ethnopharmacology, 1997-10, Vol.58 (2), p.117-123

1997

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Green tea compounds inhibit tyrosine phosphorylation of PDGF β-receptor and transformation of A172 human glioblastoma

FEBS letters, 2000-04, Vol.471 (1), p.51-55

2000

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Local delivery of green tea catechins inhibits neointimal formation in the rat carotid artery injury model

Heart and vessels, 2004-09, Vol.19 (5), p.242-247

2004

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Mitogen-activated protein kinases partially regulate endothelin-1-induced contractions through a myosin light chain phosphorylation-independent pathway

Journal of Veterinary Medical Science, 2003, Vol.65(2), pp.225-230

2003

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Ginsenoside Rg2 Inhibits Lipopolysaccharide-Induced Adhesion Molecule Expression in Human Umbilical Vein Endothelial Cell

The Korean journal of physiology & pharmacology, 2013-01, Vol.17 (2), p.133-137

2013

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Regulation of type IV collagen alpha chains of glomerular epithelial cells in diabetic conditions

Journal of Korean Medical Science, 2009, 24(5), 132, pp.837-843

2009

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Inhibitory Effect of Epigallocatechin 3-O-Gallate on Vascular Smooth Muscle Cell Hypertrophy Induced by Angiotensin II

Journal of cardiovascular pharmacology, 2004-02, Vol.43 (2), p.200-208

2004

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Epigallocatechin-3-gallate Regulates NADPH Oxidase Expression in Human Umbilical Vein Endothelial Cells

The Korean journal of physiology & pharmacology, 2010-01, Vol.14 (5), p.325-329

2010

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Inhibition of STAT3 phosphorylation by curcumin partly reduces adhesion molecule expression in vascular endothelial cell

The FASEB journal, 2018-04, Vol.32 (S1), p.837.6-837.6

2018

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Epigallocatechin-3-gallate Regulates Inducible Nitric Oxide Synthase Expression in Human Umbilical Vein Endothelial Cells

Laboratory Animal Research, 2011, 27(2), , pp.85-90

2011

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Tyrosine kinase participates in vasoconstriction through a Ca2+‐ and myosin light chain phosphorylation‐independent pathway

FEBS letters, 2002-02, Vol.512 (1-3), p.282-286

2002

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Differential expression of αB-crystallin causes maturationdependent susceptibility of oligodendrocytes to oxidative stress

BMB reports, 2013-10, Vol.46 (10), p.501

2013

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Quercetin prevents cardiac hypertrophy induced by pressure overload in rats

Mechanisms of the inhibitory effects of epigallocatechin‐3 gallate on platelet‐derived growth factor‐BB‐induced cell signaling and mitogenesis

Angiotensin II suppresses adenosine monophosphate-activated protein kinase of podocytes via angiotensin II type 1 receptor and mitogen-activated protein kinase signaling

Epigallocatechin-3 gallate prevents cardiac hypertrophy induced by pressure overload in rats

Activation of Akt/protein kinase B mediates the protective effects of mechanical stretching against myocardial ischemia-reperfusion injury

Angiotensin II Modulates p130Cas of Podocytes by the Suppression of AMP-Activated Protein Kinase

(–)-Epigallocatechin-3-O-gallate (EGCG) attenuates the hemodynamics stimulated by caffeine through decrease of catecholamines release

Mechanical Stretch-Induced Protection against Myocardial Ischemia- Reperfusion Injury Involves AMP-Activated Protein Kinase

Epigallocatechin-3-O-Gallate Inhibits the Angiotensin II-Induced Adhesion Molecule Expression in Human Umbilical Vein Endothelial Cell Via Inhibition of MAPK Pathways

Inhibition of the PDGF beta-receptor tyrosine phosphorylation and its downstream intracellular signal transduction pathway in rat and human vascular smooth muscle cells by different catechins

p38 Mitogen-Activated Protein Kinase Regulates Vasoconstriction in Spontaneously Hypertensive Rats

Epigallocathechin-3 gallate selectively inhibits the PDGF-BB-induced intracellular signaling transduction pathway in vascular smooth muscle cells and inhibits transformation of sis-transfected NIH 3T3 fibroblasts and human glioblastoma cells (A172)

Differential expression of αB-crystallin causes maturation-dependent susceptibility of oligodendrocytes to oxidative stress

Vasodilating and hypotensive effects of fangchinoline and tetrandrine on the rat aorta and the stroke-prone spontaneously hypertensive rat

Green tea compounds inhibit tyrosine phosphorylation of PDGF β-receptor and transformation of A172 human glioblastoma

Local delivery of green tea catechins inhibits neointimal formation in the rat carotid artery injury model

Mitogen-activated protein kinases partially regulate endothelin-1-induced contractions through a myosin light chain phosphorylation-independent pathway

Ginsenoside Rg2 Inhibits Lipopolysaccharide-Induced Adhesion Molecule Expression in Human Umbilical Vein Endothelial Cell

Regulation of type IV collagen alpha chains of glomerular epithelial cells in diabetic conditions

Inhibitory Effect of Epigallocatechin 3-O-Gallate on Vascular Smooth Muscle Cell Hypertrophy Induced by Angiotensin II

Epigallocatechin-3-gallate Regulates NADPH Oxidase Expression in Human Umbilical Vein Endothelial Cells

Inhibition of STAT3 phosphorylation by curcumin partly reduces adhesion molecule expression in vascular endothelial cell

Epigallocatechin-3-gallate Regulates Inducible Nitric Oxide Synthase Expression in Human Umbilical Vein Endothelial Cells

Tyrosine kinase participates in vasoconstriction through a Ca2+‐ and myosin light chain phosphorylation‐independent pathway

Differential expression of αB-crystallin causes maturationdependent susceptibility of oligodendrocytes to oxidative stress

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